Antibacterial mechanism of ε -Poly-lysine against Listeria monocytogenes and its application on cheese

Food Control ◽  
2018 ◽  
Vol 91 ◽  
pp. 76-84 ◽  
Author(s):  
Lin Lin ◽  
Yulei Gu ◽  
Changzhu Li ◽  
Saritporn Vittayapadung ◽  
Haiying Cui
Keyword(s):  
Listeria Monocytogenes ◽  
Antibacterial Mechanism

A novel subtilin-like lantibiotics subtilin JS-4 produced by Bacillus subtilis JS-4, and its antibacterial mechanism against Listeria monocytogenes

LWT ◽  
2021 ◽  
Vol 142 ◽  
pp. 110993
Author(s):  
Zhaohui Wei ◽  
Chengjun Shan ◽  
Lixia Zhang ◽  
Da'e Ge ◽  
Ying Wang ◽  
...  
Keyword(s):  
Bacillus Subtilis ◽  
Listeria Monocytogenes ◽  
Antibacterial Mechanism

scholarly journals Antimicrobial Susceptibility and Antibacterial Mechanism of Limonene against Listeria monocytogenes

Molecules ◽  
2019 ◽  
Vol 25 (1) ◽  
pp. 33 ◽  
Author(s):  
Yingjie Han ◽  
Zhichang Sun ◽  
Wenxue Chen
Keyword(s):  
Listeria Monocytogenes ◽  
Cell Membrane ◽  
Respiratory Chain ◽  
Antimicrobial Susceptibility ◽  
Chain Complex ◽  
Cell Membrane Permeability ◽  
Wall Structure ◽  
Antibacterial Mechanism ◽  
Respiratory Chain Complex ◽  
Complex Activity

Limonene is a monoterpenoid compound, which is founded in a lot of plants’ essential oils with good antibacterial activity against food-borne pathogens, but it has an ambiguous antimicrobial susceptibility and mechanism against Listeria monocytogenes (L. monocytogenes). In this study, the antimicrobial susceptibility of Limonene to L. monocytogenes was studied, and some new sights regarding its antibacterial mechanism were further explored. Scanning electron microscopy (SEM) verified that limonene caused the destruction of the cell integrity and wall structure of L. monocytogenes. The increase in conductivity and the leakage of intracellular biomacromolecules (nucleic acids and proteins) confirmed that limonene had an obvious effect on cell membrane permeability. The results of Propidium Iodide (PI) fluorescence staining were consistent with the results of the conductivity measurements. This indicated that limonene treatment caused damage to the L. monocytogenes cell membrane. Furthermore, the decrease in ATP content, ATPase (Na+K+-ATPase, Ca2+-ATPase) activity and respiratory chain complex activity indicated that limonene could hinder ATP synthesis by inhibiting the activity of the respiratory complex and ATPase. Finally, differential expression of proteins in the respiratory chain confirmed that limonene affected respiration and energy metabolism by inhibiting the function of the respiratory chain complex.

Antibacterial mechanism of Myagropsis myagroides extract on Listeria monocytogenes

Food Control ◽  
2014 ◽  
Vol 42 ◽  
pp. 23-28 ◽  
Author(s):  
So-Young Lee ◽  
Koth-Bong-Woo-Ri Kim ◽  
Seong-Il Lim ◽  
Dong-Hyun Ahn
Keyword(s):  
Listeria Monocytogenes ◽  
Antibacterial Mechanism ◽  
Myagropsis Myagroides

A novel bacteriocin BMP11 and its antibacterial mechanism on cell envelope of Listeria monocytogenes and Cronobacter sakazakii

Food Control ◽  
2018 ◽  
Vol 91 ◽  
pp. 160-169 ◽  
Author(s):  
Lanhua Yi ◽  
Xin Li ◽  
Lingli Luo ◽  
Yingying Lu ◽  
Hong Yan ◽  
...  
Keyword(s):  
Listeria Monocytogenes ◽  
Cell Envelope ◽  
Antibacterial Mechanism ◽  
Cronobacter Sakazakii

Antibacterial mechanism of brevilaterin B: an amphiphilic lipopeptide targeting the membrane of Listeria monocytogenes

2020 ◽  
Vol 104 (24) ◽  
pp. 10531-10539
Author(s):  
Yangliu Liu ◽  
Aijin Ma ◽  
Panpan Han ◽  
Zhou Chen ◽  
Yingmin Jia
Keyword(s):  
Listeria Monocytogenes ◽  
Antibacterial Mechanism

Antibacterial mechanism of plantaricin LPL-1, a novel class IIa bacteriocin against Listeria monocytogenes

Food Control ◽  
2019 ◽  
Vol 97 ◽  
pp. 87-93 ◽  
Author(s):  
Yao Wang ◽  
Yuxuan Qin ◽  
Ying Zhang ◽  
Ruiyun Wu ◽  
Pinglan Li
Keyword(s):  
Listeria Monocytogenes ◽  
Antibacterial Mechanism

scholarly journals CELLULAR RESISTANCE TO INFECTION

1962 ◽  
Vol 116 (3) ◽  
pp. 381-406 ◽  
Author(s):  
G. B. Mackaness
Keyword(s):  
Listeria Monocytogenes ◽  
Primary Infection ◽  
Protective Factor ◽  
Acquired Resistance ◽  
Mononuclear Phagocytes ◽  
Antibacterial Mechanism ◽  
Bacterial Populations ◽  
Constant Rate ◽  
Resistant Cells

The mouse was found to be natively susceptible to Listeria monocytogenes. Its susceptibility was attributed to the capacity of the organism to survive and multiplying in host macrophages. During the first 3 days of a primary infection the bacterial populations of spleen and liver were found to increase at a constant rate. On the 4th day of infection the host became hypersensitive to Listeria antigens and at the same time bacterial growth ceased. A rapid inactivation of the organism ensued. Convalescent mice were resistant to challenge, but no protective factor could be found in their serum. Histological evidence suggested that acquired resistance was the result of a change occurring in the host's mononuclear phagocytes. When challenged in vitro, the macrophages of convalescent mice were found to resist infection with Listeria monocytogenes. Listeria-resistant cells appeared during the course of infection at a time which corresponded with the development of the antibacterial mechanism in the spleen. They persisted for as long as the antibacterial mechanism remained intact in this organ. This period of absolute resistance to Listeria lasted about 3 weeks. Thereafter, the host remained hypersensitive but unable to inactivate a challenge inoculum of Listeria. However, it remained capable of producing an accelerated response to reinfection. This was thought to depend upon an ability to generate a new population of resistant cells from a residuum of specifically sensitized macrophages or macrophage precursors still surviving in the tissues as a result of the immunological activation which occurred during the primary infection.

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