Hyperoxia-induced preconditioning against renal ischemic injury is mediated by reactive oxygen species but not related to heat shock proteins 70 and 32

Surgery ◽  
2015 ◽  
Vol 157 (6) ◽  
pp. 1014-1022 ◽  
Author(s):  
Hannaneh Wahhabaghai ◽  
Reza Heidari ◽  
Atefeh Zeinoddini ◽  
Saeed Soleyman-Jahi ◽  
Leila Golmanesh ◽  
...  
Keyword(s):  
Reactive Oxygen Species ◽  
Heat Shock ◽  
Heat Shock Proteins ◽  
Reactive Oxygen ◽  
Ischemic Injury ◽  
Oxygen Species ◽  
Shock Proteins

scholarly journals Small Heat-Shock Proteins, IbpAB, Protect Non-Pathogenic Escherichia coli from Killing by Macrophage-Derived Reactive Oxygen Species

PLoS ONE ◽  
2015 ◽  
Vol 10 (3) ◽  
pp. e0120249 ◽  
Author(s):  
Laura Goeser ◽  
Ting-Jia Fan ◽  
Sandrine Tchaptchet ◽  
Nikolas Stasulli ◽  
William E. Goldman ◽  
...  
Keyword(s):  
Escherichia Coli ◽  
Reactive Oxygen Species ◽  
Heat Shock ◽  
Heat Shock Proteins ◽  
Reactive Oxygen ◽  
Small Heat Shock Proteins ◽  
Oxygen Species ◽  
Pathogenic Escherichia Coli ◽  
Shock Proteins

scholarly journals The Influence of Hyperoxia On Heat Shock Proteins Expression and Nitric Oxide Synthase Activity – the Review

2017 ◽  
Vol 58 (1) ◽  
pp. 23-28
Author(s):  
Jakub Szyller ◽  
Mariusz Kozakiewicz ◽  
Piotr Siermontowski
Keyword(s):  
Oxidative Stress ◽  
Reactive Oxygen Species ◽  
Heat Shock ◽  
Heat Shock Proteins ◽  
Nitrogen Oxide ◽  
Nitrosative Stress ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Shock Proteins ◽  
Oxide Synthase

Abstract Any stay in an environment with an increased oxygen content (a higher oxygen partial pressure, pO2) and an increased pressure (hyperbaric conditions) leads to an intensification of oxidative stress. Reactive oxygen species (ROS) damage the molecules of proteins, nucleic acids, cause lipid oxidation and are engaged in the development of numerous diseases, including diseases of the circulatory system, neurodegenerative diseases, etc. There are certain mechanisms of protection against unfavourable effects of oxidative stress. Enzymatic and non-enzymatic systems belong to them. The latter include, among others, heat shock proteins (HSP). Their precise role and mechanism of action have been a subject of intensive research conducted in recent years. Hyperoxia and hyperbaria also have an effect on the expression and activity of nitrogen oxide synthase (NOS). Its product - nitrogen oxide (NO) can react with reactive oxygen species and contribute to the development of nitrosative stress. NOS occurs as isoforms in various tissues and exhibit different reactions to the discussed factors. The authors have prepared a brief review of research determining the effect of hyperoxia and hyperbaria on HSP expression and NOS activity.

Cardioprotective effects of iroxanadine: Role of nitric oxide, reactive oxygen species and heat-shock proteins

2002 ◽  
Vol 34 (6) ◽  
pp. A3
Author(s):  
Károly Acsai ◽  
Csaba Csonka ◽  
Andrea Jednákovits ◽  
Krisztián Stadler ◽  
Zoltán Pénzes ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Reactive Oxygen Species ◽  
Heat Shock ◽  
Heat Shock Proteins ◽  
Reactive Oxygen ◽  
Oxygen Species ◽  
Cardioprotective Effects ◽  
Shock Proteins

Small Heat Shock Proteins and Protection Against Ischemic Injury in Cardiac Myocytes

Circulation ◽  
1997 ◽  
Vol 96 (12) ◽  
pp. 4343-4348 ◽  
Author(s):  
Jody L. Martin ◽  
Ruben Mestril ◽  
Randa Hilal-Dandan ◽  
Laurence L. Brunton ◽  
Wolfgang H. Dillmann
Keyword(s):  
Heat Shock ◽  
Heat Shock Proteins ◽  
Cardiac Myocytes ◽  
Ischemic Injury ◽  
Small Heat Shock Proteins ◽  
Shock Proteins
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