Inhibitory effect of troglitazone on tumor necrosis factor alpha—induced expression of monocyte chemoattractant protein-1 in human mesangial cells

Metabolism ◽  
2000 ◽  
Vol 49 (2) ◽  
pp. 163-166 ◽  
Author(s):  
Masayoshi Y. Ohta ◽  
Yukihiro Nagai ◽  
Toshinari Takamura ◽  
Erika Nohara ◽  
Ken-ichi Kobayashi
Keyword(s):  
Tumor Necrosis ◽  
Mesangial Cells ◽  
Inhibitory Effect ◽  
Necrosis Factor Alpha ◽  
Human Mesangial Cells ◽  
Monocyte Chemoattractant Protein 1 ◽  
Monocyte Chemoattractant ◽  
Monocyte Chemoattractant Protein ◽  
Factor Alpha ◽  
Necrosis Factor

scholarly journals Mycobacterium avium subsp. paratuberculosis Infection Causes Suppression of RANTES, Monocyte Chemoattractant Protein 1, and Tumor Necrosis Factor Alpha Expression in Peripheral Blood of Experimentally Infected Cattle

2003 ◽  
Vol 71 (12) ◽  
pp. 7223-7227 ◽  
Author(s):  
Joram J. Buza ◽  
Yasuyuki Mori ◽  
Abusaleh M. Bari ◽  
Hirokazu Hikono Aodon-geril ◽  
Sachiyo Hirayama ◽  
...  
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis Factor Alpha ◽  
Tumor Necrosis ◽  
Necrosis Factor Alpha ◽  
Monocyte Chemoattractant Protein 1 ◽  
Monocyte Chemoattractant ◽  
Monocyte Chemoattractant Protein ◽  
Tnf Α ◽  
Factor Alpha ◽  
Necrosis Factor

ABSTRACT Blood from cattle with subclinical Mycobacterium avium subsp. paratuberculosis infection was stimulated with M. avium subsp. paratuberculosis antigens, and expression of interleukin-1β (IL-1β), tumor necrosis factor alpha (TNF-α), RANTES, monocyte chemoattractant protein 1 (MCP-1), and IL-8 was measured. Expression of TNF-α, RANTES, and MCP-1 was lower in infected than in uninfected cattle. The reduced response may weaken protective immunity and perpetuate infection.

scholarly journals Divergent Roles of Tumor Necrosis Factor and Platelet-Activating Factor in Endotoxin-Induced Release of Monocyte Chemoattractant Protein 1 and Macrophage Inflammatory Protein 1β in Chimpanzees

1999 ◽  
Vol 67 (10) ◽  
pp. 5480-5482 ◽  
Author(s):  
Pascale E. P. Dekkers ◽  
Marcel Levi ◽  
Sander J. H. van Deventer ◽  
Tom van der Poll
Keyword(s):  
Tumor Necrosis Factor ◽  
Tumor Necrosis ◽  
Platelet Activating Factor ◽  
Necrosis Factor Alpha ◽  
Monocyte Chemoattractant Protein 1 ◽  
Monocyte Chemoattractant ◽  
Monocyte Chemoattractant Protein ◽  
Inflammatory Protein ◽  
Macrophage Inflammatory Protein ◽  
Necrosis Factor

ABSTRACT A platelet-activating factor receptor antagonist reduced the release of macrophage inflammatory protein 1β (MIP-1β) during endotoxemia in chimpanzees but did not influence the secretion of monocyte chemoattractant protein 1 (MCP-1). Anti-tumor necrosis factor alpha monoclonal antibody completely prevented MCP-1 release and simultaneously enhanced the secretion of MIP-1β. Levels of MIP-1β and MCP-1 release were differentially regulated during endotoxemia.

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