P.1.4 Does left atrial wall strain influence on SA ECG and SA IEGM time domain parameters?

EP Europace ◽  
2003 ◽  
Vol 4 ◽  
pp. A34
Author(s):  
A GOWNIAK
EP Europace ◽  
2003 ◽  
Vol 4 (Supplement_1) ◽  
pp. A34-A34
Author(s):  
A. Głowniak ◽  
A. Kutarski ◽  
D. Szczęśniak ◽  
P. Ruciński ◽  
T. Widomska-Czekajska

Author(s):  
Satoshi Hayashida ◽  
Koichi Nagashima ◽  
Sayaka Kurokawa ◽  
Masaru Arai ◽  
Ryuta Watanabe ◽  
...  

2012 ◽  
Vol 93 (3) ◽  
pp. e57-e59 ◽  
Author(s):  
Giovanni Domenico Cresce ◽  
Diletta Peluso ◽  
Marco Panfili ◽  
Alessandro Favaro ◽  
Antonio Cannarella ◽  
...  

1990 ◽  
Vol 49 (3) ◽  
pp. 476-478 ◽  
Author(s):  
Kenneth S. Stone ◽  
John W. Brown ◽  
David Canal ◽  
Randy Caldwell ◽  
Roger Hurwitz ◽  
...  

1980 ◽  
Vol 59 (s6) ◽  
pp. 361s-363s
Author(s):  
S.-E. Ricksten ◽  
T. Yao ◽  
B. Ljung ◽  
P. Thorean

1. The cardiac mechanoreceptors, which in rats are mainly located in the left atrial wall, are reset in spontaneously hypertensive rats. The atrial pressure has to be almost twice as high in spontaneously hypertensive rats as in normotensive controls to produce similar receptor activations, as is apparent from previous studies. 2. The present study was performed to investigate whether this resetting is due to decreased distensibility of left atrial walls in the spontaneously hypertensive rats. 3. Static load-length relationships were investigated in vitro on left atrial strips, and pressure-volume relationships were studied on isolated left atria from spontaneously hypertensive and normotensive Wistar-Kyoto rats. 4. The force per cross-sectional area exerted during a relative length increase of 80% was significantly greater in spontaneously hypertensive rats. The dynamic but not the static distensibility was significantly lower in these animals. 5. The decreased dynamic distensibility of left atrial walls in spontaneously hypertensive rats can at least partly explain the resetting of atrial receptor function.


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