scholarly journals Identification of genetic locus with resistance to take-all in the wheat-Psathyrostachys huashanica Keng introgression line H148

2021 ◽  
Vol 20 (12) ◽  
pp. 3101-3113
Author(s):  
Sheng-sheng BAI ◽  
Han-bing ZHANG ◽  
Jing HAN ◽  
Jian-hui WU ◽  
Jia-chuang LI ◽  
...  
Keyword(s):  
Genetic Locus ◽  
Introgression Line ◽  
Psathyrostachys Huashanica ◽  
Take All

321-OR: A Genetic Locus on Chromosome 1p36 Associated with Cardiovascular Autonomic Neuropathy in Type 2 Diabetes

Diabetes ◽  
2019 ◽  
Vol 68 (Supplement 1) ◽  
pp. 321-OR
Author(s):  
YALING TANG ◽  
HETAL SHAH ◽  
XIUQIN SUN ◽  
BRUCE A. PERKINS ◽  
MICHAEL J. WAGNER ◽  
...  
Keyword(s):  
Type 2 Diabetes ◽  
Autonomic Neuropathy ◽  
Genetic Locus ◽  
Cardiovascular Autonomic Neuropathy ◽  
Chromosome 1P36

A proposed method for estimating loss in yield of wheat caused by take-all (Ophiobolus graminis SACC.)

1954 ◽  
Vol 19 (1-2) ◽  
pp. 29-32 ◽  
Author(s):  
Heiji TASUGI ◽  
Kan NAKAYA ◽  
Naoji SUZUKI
Keyword(s):  
Take All

Generation and Characterization ofPgPGIP1Transgenic Wheat Plants with Enhanced Resistance to Take-all and Common Root Rot

2013 ◽  
Vol 39 (9) ◽  
pp. 1576 ◽  
Author(s):  
Li-Hua YANG ◽  
Jin-Feng WANG ◽  
Li-Pu DU ◽  
Hui-Jun XU ◽  
Xue-Ning WEI ◽  
...  
Keyword(s):  
Root Rot ◽  
Common Root ◽  
Common Root Rot ◽  
Enhanced Resistance ◽  
Take All

THE CASE VARIANT TRANSLOCATION T(3;9;22)(P24;Q34;Q11) IN CHRONIC MYELOID LEUKEMIA

2018 ◽  
Vol 64 (6) ◽  
pp. 810-814
Author(s):  
Kodirzhon Boboev ◽  
Yuliana Assesorova ◽  
Kh. Karimov ◽  
B. Allanazarova
Keyword(s):  
Adverse Effect ◽  
Chronic Myeloid Leukemia ◽  
Myeloid Leukemia ◽  
Fusion Gene ◽  
Genetic Locus ◽  
Philadelphia Chromosome ◽  
Chromosome 9 ◽  
Banding Technique ◽  
Variant Translocation

This paper presents a case of chronic myeloid leukemia with an earlier unknown variant translocation t (3; 9; 22) (p24; q34; q11) detected by cytogenetic research using the GTG-banding technique. Despite the absence of the classical Philadelphia chromosome, the presence of chromosome 9 and 22 derivatives, as well as the BCR-ABL fusion gene, allow this translocation to be considered pathogenetic for CML. A good response of the patient to the treatment with glivec is that there is no adverse effect on the pathogenesis of the disease of an additional genetic locus (3p24) involved in complex restructuring.

Sign in / Sign up

Export Citation Format

Share Document