Tunable KIT-6 Mesoporous Sulfonic Acid Catalysts for Fatty Acid Esterification

ACS Catalysis ◽  
2012 ◽  
Vol 2 (8) ◽  
pp. 1607-1614 ◽  
Author(s):  
Cyril Pirez ◽  
Jean-Michel Caderon ◽  
Jean-Philippe Dacquin ◽  
Adam F. Lee ◽  
Karen Wilson
Keyword(s):  
Fatty Acid ◽  
Sulfonic Acid ◽  
Acid Catalysts ◽  
Acid Esterification ◽  
Fatty Acid Esterification

scholarly journals Preparation of sulfonated carbon-based catalysts from murumuru kernel shell and their performance in the esterification reaction

RSC Advances ◽  
2020 ◽  
Vol 10 (34) ◽  
pp. 20245-20256 ◽  
Author(s):  
Ana Paula da Luz Corrêa ◽  
Rafael Roberto Cardoso Bastos ◽  
Geraldo Narciso da Rocha Filho ◽  
José Roberto Zamian ◽  
Leyvison Rafael Vieira da Conceição
Keyword(s):  
Fatty Acid ◽  
Industrial Waste ◽  
Esterification Reaction ◽  
Acid Catalysts ◽  
Esterification Reactions ◽  
Acid Esterification ◽  
Fatty Acid Esterification ◽  
Carbon Based

In the present study, heterogeneous acid catalysts for fatty acid esterification reactions were synthesized using agro-industrial waste from murumuru kernel shells.

Relation of Fatty Acid Esterification by Intestinal Mucosa of Several Species to Variations in Mucosal Anatomy

1964 ◽  
Vol 46 (6) ◽  
pp. 691-699 ◽  
Author(s):  
C.F. McCcarthy ◽  
Malcolm P. Tyor
Keyword(s):  
Fatty Acid ◽  
Intestinal Mucosa ◽  
Acid Esterification ◽  
Fatty Acid Esterification

scholarly journals Insulin-independent regulation of hepatic triglyceride synthesis by fatty acids

2015 ◽  
Vol 112 (4) ◽  
pp. 1143-1148 ◽  
Author(s):  
Daniel F. Vatner ◽  
Sachin K. Majumdar ◽  
Naoki Kumashiro ◽  
Max C. Petersen ◽  
Yasmeen Rahimi ◽  
...  
Keyword(s):  
Insulin Resistance ◽  
Fatty Acids ◽  
Fatty Acid ◽  
Insulin Signaling ◽  
Plasma Fatty Acid ◽  
Hepatic Triglyceride ◽  
Triglyceride Synthesis ◽  
Hepatic Glucose ◽  
Acid Esterification ◽  
Fatty Acid Esterification

A central paradox in type 2 diabetes is the apparent selective nature of hepatic insulin resistance—wherein insulin fails to suppress hepatic glucose production yet continues to stimulate lipogenesis, resulting in hyperglycemia, hyperlipidemia, and hepatic steatosis. Although efforts to explain this have focused on finding a branch point in insulin signaling where hepatic glucose and lipid metabolism diverge, we hypothesized that hepatic triglyceride synthesis could be driven by substrate, independent of changes in hepatic insulin signaling. We tested this hypothesis in rats by infusing [U-13C] palmitate to measure rates of fatty acid esterification into hepatic triglyceride while varying plasma fatty acid and insulin concentrations independently. These experiments were performed in normal rats, high fat-fed insulin-resistant rats, and insulin receptor 2′-O-methoxyethyl chimeric antisense oligonucleotide-treated rats. Rates of fatty acid esterification into hepatic triglyceride were found to be dependent on plasma fatty acid infusion rates, independent of changes in plasma insulin concentrations and independent of hepatocellular insulin signaling. Taken together, these results obviate a paradox of selective insulin resistance, because the major source of hepatic lipid synthesis, esterification of preformed fatty acids, is primarily dependent on substrate delivery and largely independent of hepatic insulin action.

scholarly journals Metabolic regulation of fatty acid esterification and effects of conjugated linoleic acid on glucose homeostasis in pig hepatocytes

animal ◽  
2012 ◽  
Vol 6 (2) ◽  
pp. 254-261 ◽  
Author(s):  
J.A. Conde-Aguilera ◽  
M. Lachica ◽  
R. Nieto ◽  
I. Fernández-Fígares
Keyword(s):  
Fatty Acid ◽  
Linoleic Acid ◽  
Conjugated Linoleic Acid ◽  
Glucose Homeostasis ◽  
Metabolic Regulation ◽  
Acid Esterification ◽  
Fatty Acid Esterification

Fatty acid esterification in the yolk sac membrane of the avian embryo

2004 ◽  
Vol 174 (2) ◽  
pp. 163-168 ◽  
Author(s):  
K. A. Powell ◽  
E. A. Deans ◽  
B. K. Speake
Keyword(s):  
Fatty Acid ◽  
Yolk Sac ◽  
Avian Embryo ◽  
Acid Esterification ◽  
Fatty Acid Esterification

Development of fatty acid esterification mechanisms in rat small intestine.

1979 ◽  
Vol 237 (5) ◽  
pp. E399 ◽  
Author(s):  
Y F Shiau ◽  
C Umstetter ◽  
K Kendall ◽  
O Koldovsky
Keyword(s):  
Small Intestine ◽  
Fatty Acid ◽  
Total Fatty Acid ◽  
Fetal Programming ◽  
Rat Small Intestine ◽  
Adult Rats ◽  
Major Pathway ◽  
Acid Esterification ◽  
Fatty Acid Esterification

Fatty acid esterification was measured in fetal jejunal and ileal isografts implanted under the kidney capsules of adult host rats and compared to the age-controlled intestine grown in situ. Studies were conducted on the 21st, 35th, 49th, and 63rd postconceptional days, corresponding to prenatal, suckling, weaning, and weaned rats. Substantial fatty acid esterification activity was found in prenatal jejunum but not in ileum. A proximal-distal gradient of fatty acid esterification was observed in all groups grown in situ, but not in isografts. The monoglyceride pathway (MG-P) accounted for about one-third of total fatty acid esterification (TFAE) in jejunum grown in situ and remained constant through the study. In the ileum, MG-P was the major esterification pathway during the first 4 postnatal weeks, but decreased progressively after weaning to become insignificant in adult rats. TFAE fell in the jejunal isografts, whereas it increased in the ileum. MG-P remained as the major pathway in the implanted jejunum and ileum. Our studies suggest that luminal contents are probably the most important modulator for the development and maintenance of intestinal fatty acid esterification, and "fetal programming" manifested by changes in fatty acid esterification mechanisms in the isografts is less important.

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