scholarly journals Loss of miR-144 signaling interrupts extracellular matrix remodeling after myocardial infarction leading to worsened cardiac function

2018 ◽  
Vol 8 (1) ◽  
Author(s):  
Quan He ◽  
Fangfei Wang ◽  
Takashi Honda ◽  
Jeanne James ◽  
Jing Li ◽  
...  
2019 ◽  
Vol 75-76 ◽  
pp. 43-57 ◽  
Author(s):  
Signe Holm Nielsen ◽  
Alan J. Mouton ◽  
Kristine Y. DeLeon-Pennell ◽  
Federica Genovese ◽  
Morten Karsdal ◽  
...  

2009 ◽  
Vol 23 (S1) ◽  
Author(s):  
Sarah M. McCurdy ◽  
Qiuxia Dai ◽  
Amy D. Bradshaw ◽  
Merry L. Lindsey

2019 ◽  
Vol 47 (6) ◽  
pp. 1679-1687
Author(s):  
Mavis A.A. Tenkorang ◽  
Upendra Chalise ◽  
Michael J. Daseke, II ◽  
Shelby R. Konfrst ◽  
Merry L. Lindsey

Myocardial Infarction (MI) initiates a series of wound healing events that begins with up-regulation of an inflammatory response and culminates in scar formation. The extracellular matrix (ECM) is intricately involved in all stages from initial break down of existing ECM to synthesis of new ECM to form the scar. This review will summarize our current knowledge on the processes involved in ECM remodeling after MI and identify the gaps that still need to be filled.


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