scholarly journals Osteolysis and Cervical Cord Compression Secondary to Silicone Granuloma Formation around a Dorsal Spinal Cord Stimulator: A Case Report

2016 ◽  
Vol 77 (02) ◽  
pp. e67-e72 ◽  
Author(s):  
John Dimar ◽  
David Endriga ◽  
Leah Carreon
2020 ◽  
Vol 11 ◽  
pp. 20
Author(s):  
Ryan G. Eaton ◽  
Stephanus V. Viljoen

Background: Atlantoaxial pseudoarticulation rarely involves the cervical spine, and its etiology is unclear. In theory, pseudoarticulation is comparable to Bertolotti’s syndrome in the lumbar spine or may be attributed to an aberrant focal fusion between C0-C1-C2 that occurs during the gastrulation of embryologic development. Case Description: A 39-year-old female presented with neck pain and upper extremity weakness. Magnetic resonance/computed tomography studies documented a left-sided unilateral pseudoarticulation between the lamina of C1 and C2 causing compression of the dorsal spinal cord. Following resection of the accessory C1/C2 joint utilizing a C1 hemilaminectomy and partial C2 laminectomy, the patient’s neck pain and weakness resolved. Histologically, the tissue showed benign osteocartilaginous tissue with no synovial capsule. Conclusion: Here, the authors present a case of occiput-C1-C2 pseudojoint formation, leading to clinical and radiographic findings of cord compression due to boney outgrowth.


2020 ◽  
Vol 2020 (12) ◽  
Author(s):  
Diogo Belo ◽  
Joaquim Cruz Teixeira ◽  
José Pedro Lavrador

Abstract Brown-Séquard syndrome (BSS) is a rare neurological condition caused by a hemi-lesion of the spinal cord and was first described in the 1800s. BSS is characterized by an ipsilateral absence of motor control and discriminatory/proprioceptive/vibratory sensation at and below the spinal level involved, associated with loss of contralateral temperature and pain sensation a couple of vertebral segments below the lesion. BSS is commonly associated with trauma, but can also be iatrogenic. The authors report a case of a patient who presented with neoplastic dorsal spinal cord compression and developed a BSS after surgical decompression and review of the literature of postoperative BSS cases.


Author(s):  
Haruki Funao ◽  
Satoshi Nakamura ◽  
Kenshi Daimon ◽  
Norihiro Isogai ◽  
Yutaka Sasao ◽  
...  

PM&R ◽  
2014 ◽  
Vol 6 (9) ◽  
pp. S312-S313
Author(s):  
Maria Margarita Lopez ◽  
Manish Mammen ◽  
Joseph David ◽  
Sanjeev Agarwal ◽  
Hana Ilan

2019 ◽  
Vol 24 ◽  
pp. 100863
Author(s):  
Mehdi Borni ◽  
Brahim Kammoun ◽  
Fatma Kolsi ◽  
Mohamed Zaher Boudawara

2020 ◽  
Vol 21 (1) ◽  
Author(s):  
Takuhei Kozaki ◽  
Akihito Minamide ◽  
Hiroshi Iwasaki ◽  
Yasutsugu Yuakawa ◽  
Muneharu Ando ◽  
...  

1993 ◽  
Vol 40 (1) ◽  
pp. 43-46 ◽  
Author(s):  
Koh Yano ◽  
Satoru Murase ◽  
Tatsuya Kuroda ◽  
Kouji Noguchi ◽  
Yusuke Tanabe ◽  
...  

PLoS ONE ◽  
2021 ◽  
Vol 16 (8) ◽  
pp. e0256732
Author(s):  
Toru Hirai ◽  
Koji Otani ◽  
Miho Sekiguchi ◽  
Shin-ichi Kikuchi ◽  
Shin-ichi Konno

Background Degenerative compressive myelopathy (DCM) is caused by cervical cord compression. The relationship between the magnitude and clinical findings of cervical cord compression has been described in the literature, but the details remain unclear. This study aimed to clarify the relationship between the magnitude and clinical symptoms of cervical cord compression in community-dwelling residents. Methods The present study included 532 subjects. The subjective symptoms and the objective findings of one board-certified spine surgeon were assessed. The subjective symptoms were upper extremity pain and numbness, clumsy hand, fall in the past 1 year, and subjective gait disturbance. The objective findings were: Hoffmann, Trömner, and Wartenberg signs; Babinski’s and Chaddock’s signs; hyperreflexia of the patellar tendon and Achilles tendon reflexes; ankle clonus; Romberg and modified Romberg tests; grip and release test; finger escape sign; and grip strength. Using midsagittal T2-weighted magnetic resonance imaging, the anterior–posterior (AP) diameters (mm) of the spinal cord at the C2 midvertebral body level (DC2) and at each intervertebral disc level from C2/3 to C7/T1 (DC2/3-C7/T1) were measured. The spinal cord compression ratio (R) for each intervertebral disc level was defined and calculated as DC2/3-C7/T1 divided by DC2. The lowest R (LR) along C2/3 to C7/T1 of each individual was divided into 3 grades by the tertile method. The relationship between LR and clinical symptoms was investigated by trend analysis. Results The prevalence of subjective gait disturbance increased significantly with the severity of spinal cord compression (p = 0.002812), whereas the other clinical symptoms were not significantly related with the severity of spinal cord compression. Conclusions The magnitude of cervical cord compression had no relationship with any of the neurologic findings. However, subjective gait disturbance might be a better indicator of the possibility of early stage cervical cord compression.


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