Fetal macrosomia as a risk factor for shoulder dystocia during vacuum extraction

2016 ◽  
Vol 30 (15) ◽  
pp. 1870-1873 ◽  
Author(s):  
Shmuel Herzberg ◽  
Doron Kabiri ◽  
Tzlil Mordechai ◽  
Rani Haj Yahya ◽  
Henry Chill ◽  
...  
Author(s):  
Rakesh Appasaheb Hasabe ◽  
Dnyaneshwar S. Diwane ◽  
Sushant S. Chandawar

Background: Brachial plexus injury (BPI) has prevalence of between 0.5 and 4.4/1000 live births. It is commonly believed to be attributed to extensive lateral traction during difficult neck delivery. This paper aims to look at one aspect of birth injuries, Erb-Duchenne palsy, its incidence and contributing factors.Methods: All infants sustaining Erb-Duchenne palsy during birth were identified at Department of OBGY, NIMS Hospital, Jaipur. The notes of the infants and mothers were reviewed. Special attention was given to the known risk factors for birth trauma such as ethnicity, parity, antenatal care, history of diabetes etc.Results: During the two-year period 2013 to 2015, there were 6 infants diagnosed with Erb-Duchenne palsy, giving an incidence of 0.79 in 1000 live births. Out of six, four were primigravidae. All pregnancies were singleton, cephalic/vertex undergoing vaginal deliveries; none required forceps or ventouse deliveries. The mean infant birthweight was 4.378 kg with a median value of 4.48 kg (range 3.51-4.78). Four infants were classified as macrosomic i.e. birthweights greater than 4 kg, four infants had birthweights of 4 kg to 5 kg; none weighed over 5 kg. Three of the deliveries were documented as difficult shoulder delivery/shoulder dystocia. Five of the infants had APGAR scores that were less than seven.Conclusions: This study suggested that Erb-Duchenne palsy is strongly associated with fetal macrosomia and shoulder dystocia. Diabetes was not significant in causing macrosomia as only single mother was found diabetic. Fetal macrosomia contributed to shoulder dystocia in the majority of cases.


1999 ◽  
Vol 78 (8) ◽  
pp. 735-736 ◽  
Author(s):  
Ofer Gemer ◽  
Marina Bergman ◽  
Shmuel Segal

2015 ◽  
Vol 2015 ◽  
pp. 1-5 ◽  
Author(s):  
Marta Simó González ◽  
Oriol Porta Roda ◽  
Josep Perelló Capó ◽  
Ignasi Gich Saladich ◽  
Joaquim Calaf Alsina

The aim of this study was to analyze the comparative risks of this anal sphincter injury in relation to the type of intervention in vaginal delivery. We performed an observational, retrospective study of all vaginal deliveries attended at a tertiary university hospital between January 2006 and December 2009. We analyzed the incidence of obstetric anal sphincter injury for each mode of vaginal delivery: spontaneous delivery, vacuum, Thierry spatulas, and forceps. We determined the proportional incidence between methods taking spontaneous delivery as the reference. Ninety-seven of 4526 (2.14%) women included in the study presented obstetric anal sphincter injury. Instrumental deliveries showed a significantly higher risk of anal sphincter injury (2.7 to 4.9%) than spontaneous deliveries (1.1%). The highest incidence was for Thierry spatulas (OR 4.804), followed by forceps (OR 4.089) and vacuum extraction (OR 2.509). The type of intervention in a vaginal delivery is a modifiable intrapartum risk factor for obstetric anal sphincter injury. Tearing can occur in any type of delivery but proportions vary significantly. All healthcare professionals attending childbirth should be aware of the risk for each type of intervention and consider these together with the obstetric factors in each case.


2019 ◽  
Vol 12 (1) ◽  
Author(s):  
Tess Cruickshank ◽  
Tu’uhevaha J. Kaitu’u-Lino ◽  
Ping Cannon ◽  
Alesia Harper ◽  
Tuong-Vi Nguyen ◽  
...  

Abstract Objective Fetal macrosomia is a major risk factor for shoulder dystocia, which can lead to birth asphyxia, maternal and neonatal traumatic injuries, and perinatal death. If macrosomia is diagnosed in the antenatal period, labour can be induced to decrease shoulder dystocia. But current clinical methods to diagnose fetal macrosomia antenatally perform with poor accuracy. Therefore, improved methods to accurately diagnose fetal macrosomia are required. Blood biomarkers that predict fetal macrosomia could be one such novel diagnostic strategy. We undertook a nested case–control study from a prospective collection of 1000 blood samples collected at 36 weeks’ gestation. We analysed plasma samples from 52 women who subsequently delivered a macrosomic (> 95th centile for gestational age) infant and 106 controls. Circulating concentrations of the proteins COBLL1, CSH1, HSD3B1, EGFL6, XAGE3, S100P, PAPPA-1, ERBB2 were assessed for their ability to predict macrosomic infants. Results We did not identify any significant changes in the plasma concentrations of COBLL1, CSH1, HSD3B1, EGFL6, XAGE3, S100P, PAPPA-1, ERBB2 from women who subsequently delivered macrosomic neonates relative to control samples. Although we have not identified any potential biomarkers of fetal macrosomia, we have ruled out these particular eight protein candidates.


1988 ◽  
Vol 26 (1) ◽  
pp. 168-168
Author(s):  
EL Leikin ◽  
JH Jenkins ◽  
GA Pomerantz ◽  
L Klein

2016 ◽  
Vol 3 (2) ◽  
pp. 84
Author(s):  
Kiswa Anggreany ◽  
Mohammmad Luthfi ◽  
Risanto Siswosudarmo

Background: Misoprostol is a synthetic prostaglandin E1 analogue which is now commonly used for induction of labor. Hyperstimulation is a complication of misoprostol that may lead to uterine rupture.Objective: To find the association between misoprostol exposure for induction of labor with uterine rupture.Methods: Case were all women who delivered in Sardjito and affiliate Hospitals from January 2007 to November 2012 with the diagnosis of uterine rupture. Controls were taken randomly from the same hospital. Chi square test and logistic regression model were used for statistical analysis.Result and Discussion: There were 53 cases of uterine rupture and 199 controls. The incidence of uterine rupture was 53 over 64,244 deliveries or 0,08%. Risk of of uterine rupture associated with misoprostol exposure was 1, 09 (CI 95% 0,52-2,2), while that of oxytocin exposure was 0,80 (CI 95% 0,35-1,85). Logistic regression analysis showed that the highest risk factor associated with uterine rupture was fetal weight > 3500 (OR 3,46; 95% CI 1,48-8,56) followed by parity (OR 2,56;95% CI 1,019-6,465) and vacuum extraction(OR 2,45;95% CI 0,94-6,39).Conclusion: There was no association between misoprostol exposure with uterine rupture. Fetal weight> 3500 gram, Parity more than 3, and vacuum extraction increased the risk of uterine rupture associated with misoprostol use.Keywords: Misoprostol, induction of labor, uterine rupture, fetal weight, parity.


2019 ◽  
Author(s):  
Caitlin Clifford ◽  
Andrea G. Edlow

Excessive fetal growth and increased birth weight are associated with significant maternal and neonatal morbidity and have become increasingly common given the global obesity epidemic.  Fetal macrosomia is traditionally defined in developed countries as fetal weight greater than 4,000 grams or 4,500 grams regardless of gestational age.  Large-for-gestational-age is traditionally defined as birth weight equal to or greater than the ninetieth percentile for a given gestational age.  Both are associated with a continuum of risk for complications, including shoulder dystocia, birth trauma, stillbirth, and infant mortality.  Diabetes is strongly associated with macrosomia, and control of maternal hyperglycemia has been proven to decrease rates of macrosomia and associated adverse pregnancy outcomes. Pregnancy-based interventions to minimize gestational weight gain have failed to consistently demonstrate a significant impact on macrosomia. This review contains 5 tables, and 77 references. Keywords: pregnancy, macrosomia, large for gestational age, estimated fetal weight, diabetes, obesity, shoulder dystocia, cesarean delivery, stillbirth


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