Association of Income and Adversity in Childhood with Adult Health and Well-Being

2018 ◽  
Vol 92 (1) ◽  
pp. 69-92 ◽  
Author(s):  
H. Luke Shaefer ◽  
Adrienne Lapidos ◽  
Reid Wilson ◽  
Sandra Danziger
2010 ◽  
Vol 31 (8) ◽  
pp. 1065-1080 ◽  
Author(s):  
Thomas A. LaVeist ◽  
Tia L. Zeno ◽  
Ruth G. Fesahazion

This article explores the effects of being raised by married parents during childhood on health and well-being in adolescence and young adulthood in a longitudinal sample of African Americans. This study aims to address the following three questions: Does childhood with married patients lead to better health and well-being during adolescence? Does childhood with married patients lead to better health and well-being in young adulthood? Do the health effects of childhood with married patients differ for male and female? The authors found modest direct effects of childhood exposure to marriage on health for females. Having at least some childhood marriage exposure was also associated with several positive health behaviors. There is modest evidence that marriage bestows health benefits for children and that these benefits endure into young adulthood.


2015 ◽  
Vol 9s1 ◽  
pp. SART.S31437 ◽  
Author(s):  
John E. Schulenberg ◽  
Megan E. Patrick ◽  
Deborah D. Kloska ◽  
Julie Maslowsky ◽  
Jennifer L. Maggs ◽  
...  

This study used national multicohort panel data from the Monitoring the Future study ( N = 25,536 from senior year classes 1977–1997 followed up to the age of 35 years in 1994–2014) to examine how early midlife (age 35 years) alcohol use disorder (AUD) and cannabis use disorder (CUD) are associated with adolescent and adult sociodemographics and health and well-being risk factors. Survey items adapted from DSM-5 diagnostic criteria were used to identify individuals who (a) showed symptoms consistent with criteria for AUD or CUD at age 35 years, (b) used the substance without qualifying for a disorder (nondisordered users), and (c) abstained from using alcohol or marijuana during the past five years. At age 35 years, the estimated prevalence of past five-year AUD was 28.0%, and that of CUD was 6.1%. Multinomial logistic regressions were used to identify variations in the relative risk of disorder symptoms as a function of sociodemographic characteristics, age 18 educational and social indices and substance use, and age 35 health and satisfaction indices and substance use. In the full models, age 18 binge drinking and marijuana use were found to be among the strongest predictors of age 35 AUD and CUD, respectively. Among age 35 health and well-being indicators, lower overall health, more frequent cognitive difficulties, and lower satisfaction with spouse/partner were consistently associated with greater risks of AUD and CUD. Some evidence was found for a J-shaped association between age 35 AUD or CUD status and health and well-being indices, such that nondisordered users were sometimes better off than both abstainers and those experiencing disorder. Finally, nondisordered cannabis use, but not CUD, was found to be more common in more recent cohorts. Implications are discussed regarding the importance of placing early midlife substance use disorder within the context of both adolescent substance use and adult health and well-being.


2020 ◽  
Vol Publish Ahead of Print ◽  
Author(s):  
Megan Wolfe ◽  
Anne De Biasi ◽  
Jane Carmody ◽  
Terry Fulmer ◽  
John Auerbach

2009 ◽  
Vol 364 (1534) ◽  
pp. 3419-3427 ◽  
Author(s):  
D. S. Gardner ◽  
S. E. Ozanne ◽  
K. D. Sinclair

The early-life developmental environment is instrumental in shaping our overall adult health and well-being. Early-life diet and endocrine exposure may independently, or in concert with our genetic constitution, induce a pathophysiological process that amplifies with age and leads to premature morbidity and mortality. Recently, this has become known as ‘programming’ but is akin to ‘maternal effects’ described for many years in the biological sciences and is defined as any influence that acts during critical developmental windows to induce long-term changes in the organisms' phenotype. To date, such delayed maternal effects have largely been characterized in terms of susceptibility to cardiovascular or metabolic disease. Here, we review evidence from experimental animal species, non-human primates and man for an effect of the early-life nutritional environment on adult fecundity and fertility. In addition, using a database of pedigree sheep, we also specifically test the hypothesis that being born small for gestational age with or without post-natal growth acceleration directly programmes fertility. We conclude that there is a lack of compelling evidence to suggest pre-natal undernutrition may directly reduce adult fecundity and fertility, but may exert some effects secondarily via an increased incidence of ‘metabolic syndrome’. Possible effects of being born relatively large on subsequent fecundity and fertility warrant further investigation.


2007 ◽  
Vol 161 (8) ◽  
pp. 730 ◽  
Author(s):  
Arthur J. Reynolds ◽  
Judy A. Temple ◽  
Suh-Ruu Ou ◽  
Dylan L. Robertson ◽  
Joshua P. Mersky ◽  
...  

Author(s):  
Alan Emond

The first United Nations Sustainable Development Goal is to ‘ensure that all human beings can fulfil their potential in dignity and equality’. Protecting, promoting, and supporting early childhood development is essential to achieve this goal. The foundations of child health and well-being, and of adult health in later life, are laid down in pregnancy and the early years. So this review of evidence takes a life course approach, starting in pregnancy and extending to the age of 7 years, to include transition into school and to cover the ‘early years’.


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