The CD4/CD8 Ratio is Inversely Associated with Carotid Intima-Media Thickness Progression in Human Immunodeficiency Virus-Infected Patients on Antiretroviral Treatment

2016 ◽  
Vol 32 (7) ◽  
pp. 648-653 ◽  
Author(s):  
Enrique Bernal Morell ◽  
José Serrano Cabeza ◽  
Ángeles Muñoz ◽  
Irene Marín ◽  
Mar Masiá ◽  
...  
2007 ◽  
Vol 99 (10) ◽  
pp. 1470-1472 ◽  
Author(s):  
Gianluca Iacobellis ◽  
Adriano M. Pellicelli ◽  
Arya M. Sharma ◽  
Benvenuto Grisorio ◽  
Giorgio Barbarini ◽  
...  

2012 ◽  
Vol 109 (5) ◽  
pp. 742-747 ◽  
Author(s):  
Priscilla Y. Hsue ◽  
Karen Ordovas ◽  
Theodore Lee ◽  
Gautham Reddy ◽  
Michael Gotway ◽  
...  

2011 ◽  
Vol 53 (8) ◽  
pp. 826-835 ◽  
Author(s):  
J. V. Baker ◽  
W. K. Henry ◽  
P. Patel ◽  
T. J. Bush ◽  
L. J. Conley ◽  
...  

Author(s):  
Alexander W. Caulk ◽  
Rudolph L. Gleason

Since the advent of highly active antiretroviral therapy (HAART), patients infected with human immunodeficiency virus-1 (HIV-1) are living longer lives. However, HIV-1-positive (HIV-1+) patients are now experiencing many non-AIDS related comorbidities including myocardial infarction, atherosclerotic lesions, and other preclinical markers of atherosclerosis including increased carotid intima-media thickness (cIMT), arterial stiffness, and impaired flow-mediated dilation (FMD). Studies have implicated the virus, the treatment, or both in the progression of these co-morbidities, causing the exact mechanisms of cardiovascular disease progression to remain unclear.


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