scholarly journals Chronic gastroesophageal reflux disease shares genetic background with esophageal adenocarcinoma and Barrett's esophagus

2015 ◽  
Vol 25 (4) ◽  
pp. 828-835 ◽  
Author(s):  
Puya Gharahkhani ◽  
Joyce Tung ◽  
David Hinds ◽  
Aniket Mishra ◽  
Thomas L. Vaughan ◽  
...  
Surgery ◽  
2013 ◽  
Vol 154 (4) ◽  
pp. 856-866 ◽  
Author(s):  
Michal J. Lada ◽  
Dylan R. Nieman ◽  
Michelle Han ◽  
Poochong Timratana ◽  
Omran Alsalahi ◽  
...  

2012 ◽  
Vol 142 (5) ◽  
pp. S-1086
Author(s):  
Guilherme Pretto ◽  
Richard R. Gurski ◽  
Daniel Navarini ◽  
Marcelo Binato ◽  
Luise Meurer ◽  
...  

Helicobacter ◽  
2012 ◽  
Vol 17 (5) ◽  
pp. 402-403 ◽  
Author(s):  
Jannis Kountouras ◽  
Dimitrios Chatzopoulos ◽  
Christos Zavos ◽  
Stergios A. Polyzos ◽  
Evaggelia Giartza-Taxidou ◽  
...  

Author(s):  
Trent Walradt ◽  
Mohammad Bilal ◽  
Douglas K. Pleskow

AbstractBarrett's esophagus (BE) is the condition in which a metaplastic columnar epithelium that is predisposed to malignancy replaces the stratified squamous epithelium that normally lines the distal esophagus. BE develops as a consequence of chronic gastroesophageal reflux disease and predisposes to esophageal adenocarcinoma (EAC). Several societal guidelines recommend screening and surveillance for BE to reduce the risk of EAC and its related morbidity and mortality. Even among persons undergoing screening and surveillance, a substantial proportion of cases of EAC can be missed. Consequently, the armamentarium for the evaluation of BE has expanded rapidly over the past decade. In this article, we summarize the pathophysiology and diagnosis of BE. We also discuss the latest advancements in the evaluation of BE.


2001 ◽  
Vol 120 (5) ◽  
pp. A410-A410
Author(s):  
F BANKI ◽  
S DEMEESTER ◽  
R MASON ◽  
G CAMPOS ◽  
C STREETS ◽  
...  

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