Comparison of three U-Net family architectures for left ventricular myocardial wall automatic segmentation

Author(s):  
Grigoris I Grigoriadis ◽  
Maria Roumpi ◽  
Dimitrios Zaridis ◽  
Vasilis C Pezoulas ◽  
Aidonis Rammos ◽  
...  
2017 ◽  
Vol 32 (6) ◽  
pp. 405-408 ◽  
Author(s):  
Seok In Lee ◽  
So Young Lee ◽  
Chang Hyu Choi ◽  
Kook Yang Park ◽  
Chul-Hyun Park

Acute myocardial infarction (AMI) can progress to cardiogenic shock and mechanical complications. When extracorporeal membrane oxygenation (ECMO) is applied to a patient with AMI with cardiogenic shock and mechanical complications, left ventricular (LV) decompression is an important recovery factor because LV dilation increases myocardial wall stress and oxygen consumption. The authors present the case of a 72-year-old man with AMI and LV dilation who developed cardiogenic shock and papillary muscle rupture and who was treated successfully by ECMO with a left atrial venting.


2018 ◽  
Vol 03 (01) ◽  
pp. 034-038
Author(s):  
Bharat Goud C ◽  
Johann Christopher

AbstractLeft ventricular free wall rupture (LVFWR) is a near-fatal mechanical complication of acute myocardial infarction in which an early diagnosis and emergency surgery should be of utmost priority for successful treatment. LVFWR is generally perceived to be universally fatal. Majority of LVFWR patients developing cardiac tamponade die rapidly, while in minority of cases the development of tamponade may be sufficiently slow to allow for diagnosis and successful intervention. In this article, the authors report a case of a 63-year-old male patient diagnosed with an inferoposterior wall myocardial infarction treated with early reperfusion thrombolytic therapy presenting 3 days later with diagnosis of subacute LVFWR. Patient had a history of relapse of chest pain which was severe and prolonged with 2 to 3 mm saddle-shaped ST-segment elevation in lateral leads, detected on a routine electrocardiogram, which led to an urgent bedside transthoracic echocardiogram (TTE). TTE showed regional wall motion abnormality in form of akinetic basal inferior-wall, a small echodense pericardial effusion, and a canalicular tract from endocardium to pericardium, through which color-Doppler examination suggested blood crossing the myocardial wall. A cardiac magnetic resonance imaging further reinforced the possibility of contained LVFWR.


1983 ◽  
Vol 61 (3) ◽  
pp. 207-212
Author(s):  
D. Leigh Carson ◽  
Peter E. Dresel

Transmural conduction time, measured as the difference in arrival time of impulses from a distant stimulating site at endocardial and epicardial electrodes near the left ventricular apex, has been reported to decrease when closely coupled extrasystoles are interpolated, indicating that muscle conduction could be supernormal. We have now determined that reduction in transmural conduction time is accounted for completely by the relatively late arrival time of the extrasystolic wave front at the endocardial recording site. The endocardial recording site was activated later than an immediately adjacent site within the wall in two out of eight animals, which could be interpreted as retrograde conduction. No evidence for supernormal conduction within the ventricular wall could be obtained by multiple electrode recordings. Supernormal conduction throughout the myocardial wall could not be demonstrated on stimulation of the endocardial site. We conclude that supernormal conduction in myocardial muscle cannot be demonstrated and that changes in transmural conduction time do not always measure myocardial conduction velocity.


Cardiology ◽  
1994 ◽  
Vol 85 (3-4) ◽  
pp. 175-183 ◽  
Author(s):  
André E. Aubert ◽  
Bart G. Denys ◽  
Hilaire De Geest

1961 ◽  
Vol 201 (1) ◽  
pp. 97-101 ◽  
Author(s):  
Lloyd L. Hefner ◽  
H. Cecil Coghlan ◽  
William B. Jones ◽  
T. Joseph Reeves

Left ventricular diastolic pressure-circumference curves were determined in 13 dogs, during stepwise hemorrhage and transfusion of blood. The linearity and small amount of scatter in the pressure-circumference curves obtained during control circumstances is evidence that elasticity rather than viscosity or inertia is the dominant determinant of the curve. Coincident with the slow infusion of epinephrine the distensibility increased. The importance of viscosity and inertial effects in the myocardial wall may also have increased, as evidenced by the increased scatter of the pressure-circumference graph. The magnitude of the change in distensibility observed was great enough to produce large changes in ventricular performance at a given filling pressure.


1995 ◽  
Vol 269 (1) ◽  
pp. H262-H270 ◽  
Author(s):  
J. W. Holmes ◽  
Y. Takayama ◽  
I. LeGrice ◽  
J. W. Covell

The role of the papillary muscle in left ventricular function has received new attention. We hypothesized that regional mechanics of the left ventricular wall near the anterior papillary muscle are influenced by the papillary muscle insertion. We therefore studied three-dimensional regional mechanics in and near the anterior papillary muscle in anesthetized, open-chest dogs, using implanted radiopaque markers and biplane cineradiography. In seven dogs, deformation differed little between an anterior papillary muscle insertion site (PMA) and a more basal site (PMB) overlying the anterior papillary muscle. However, local shortening and wall thickening were depressed in both locations relative to anterior free wall sites (FWA, FWB) studied in five additional dogs. A distinct structural border was observed at the junction between the myocardial wall and anterior papillary muscle, which may preclude the use of homogeneous strain in that region. Data from within the anterior papillary muscle indicated that uniaxial measurements in the papillary muscle are extremely sensitive to the orientation of the measurement axis, possibly explaining the variety of papillary muscle shortening patterns reported by previous investigators.


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