Estimating Precision in Heat Rate Testing

1974 ◽  
Vol 96 (3) ◽  
pp. 223-227
Author(s):  
W. H. Rousseau ◽  
E. L. Milgram

Obtaining an estimate of the precision realized in heat rate testing is discussed in this paper. Heat rate precision depends largely on the scatter of the most important measurements, so we examine their scatter. All solutions proposed are based on the assumption that the data arise from a normal population, although answers are also useful for near-normal distributions. We propose a three-part procedure designed to ensure the gathering of data of a given precision with a given confidence. The procedure should fit easily into the test engineer’s routine.

2019 ◽  
Vol 27 (2) ◽  
pp. 75-87
Author(s):  
Xiaonan Zhu ◽  
Baokun Li ◽  
Tonghui Wang ◽  
Arjun K. Gupta

Abstract The sample mean and sample variance are commonly used statistics in our study. In this paper, distributions of the sample mean and sample variance from a skew normal population are derived under closed skew normal (CSN) settings. The noncentral closed skew chi-square distribution is defined, and the distribution of quadratic forms is discussed. Our results generalize the corresponding results given under skew normal settings. Several examples are given for illustration of our results.


2013 ◽  
Vol 63 (2) ◽  
Author(s):  
Pooi Ah Hin ◽  
Soo Huei Ching

The Jarque-Bera test is a test based on the coefficients of skewness (S) and kurtosis (K) for testing whether the given random sample is from a normal population. When the random sample of size n contains m outliers, we use the remaining n-m observations to compute two statistics S* and K* which mimic the statistics S and K. The statistics S* and  K* are next  transformed to z1 and z2 which are uncorrelated and having standard normal distributions when the original population is normal. We show that the acceptance region given by a circle in the (z1, z2) plane is suitable for testing the normality assumption.


Author(s):  
W. Liebrich

HeLa cells were grown for 2-3 days in EAGLE'S minimum essential medium with 10% calf serum (S-MEM; Seromed, München) and then incubated for 24 hours in serum free medium (MEM). After detaching the cells with a solution of 0. 14 % EDTA and 0. 07 % trypsin (Difco, 1 : 250) they were suspended in various solutions (S-MEM = control, MEM, buffered salt solutions with or without Me++ions, 0. 9 % NaCl solution) and allowed to settle on glass tube slips (Leighton-tubes). After 5, 10, 15, 20, 25, 30, 1 45, 60 minutes 2, 3, 4, 5 hours cells were prepared for scanning electron microscopy as described by Paweletz and Schroeter. The preparations were examined in a Jeol SEM (JSM-U3) at 25 KV without tilting.The suspended spherical HeLa cells are able to adhere to the glass support in all solutions. The rate of attachment, however, is faster in solutions without serum than in the control. The latter is in agreement with the findings of other authors.


2016 ◽  
Vol 37 (1) ◽  
pp. 49-55 ◽  
Author(s):  
Alberto Dionigi

Abstract. In recent years, both professional and volunteer clowns have become familiar in health settings. The clown represents a peculiar humorist’s character, strictly associated with the performer’s own personality. In this study, the Big Five personality traits (BFI) of 155 Italian clown doctors (130 volunteers and 25 professionals) were compared to published data for the normal population. This study highlighted specific differences between clown doctors and the general population: Clown doctors showed higher agreeableness, conscientiousness, openness, and extraversion, as well as lower neuroticism compared to other people. Moreover, specific differences emerged comparing volunteers and professionals: Professional clowns showed significantly lower in agreeableness compared to their unpaid colleagues. The results are also discussed with reference to previous studies conducted on groups of humorists. Clowns’ personalities showed some peculiarities that can help to explain the facility for their performances in the health setting and that are different than those of other groups of humorists.


1989 ◽  
Author(s):  
Teresa M. Amabile
Keyword(s):  

1998 ◽  
Vol 79 (03) ◽  
pp. 684-685 ◽  
Author(s):  
Birga Zwinge ◽  
Inge Scharrer ◽  
Silke Ehrenforth

1990 ◽  
Vol 64 (02) ◽  
pp. 239-244 ◽  
Author(s):  
P H Reitsma ◽  
W te Lintel Hekkert ◽  
E Koenhen ◽  
P A van der Velden ◽  
C F Allaart ◽  
...  

SummaryScreening of restriction erzyme digested DNA from normal and protein C deficient individuals with a variety of probes derived from the protein C locus has revealed the existence of two neutral MspI polymorphism. One polymorphism (MI), which is located ≈7 kb upstream of the protein C gene, has allelic frequencies of 69 and 31%, and was used to exclude extensive gene deletions as a likely cause of type I protein C deficiency in 50% of cases in a panel of 22 families. Furtherrnore, the same polymorphism has been used in 5 doubly affected individuals establishing compound heterozygosity in 3 of these.The second, intragenic, polymorphism (MII) has allelic frequencies of 99 and 1% in the normal population. The frequency of the rare allele of this RFLP was with 7% much higher in a panel of 22 Dutch families with protein C deficiency. Interestingly, in all three probands that were heterozygous for MII the rare allele of MII coincided with a point mutation that leads to a stop codon in amino acid position 306 of the protein C coding sequence. This mutation may account for 14% of the protein C deficient individuals in The Netherlands.


1995 ◽  
Vol 74 (02) ◽  
pp. 580-583 ◽  
Author(s):  
B P C Koeleman ◽  
D van Rumpt ◽  
K Hamulyák ◽  
P H Reitsma ◽  
R M Bertina

SummaryWe recently reported a high prevalence of the FV Leiden mutation (R506Q, responsible for Activated Protein C resistance) among symptomatic protein C deficient probands (19%), and the involvement of the FV Leiden mutation in the expression of thrombophilia in six protein C deficient families. Here, we report the results of a similar study in protein S deficient probands and families. Among 16 symptomatic protein S deficient probands the prevalence of the FV Leiden mutation was high (38%). This high prevalence is significantly different from that in the normal population, and is probably caused by the selection of probands for familial thrombosis and protein S deficiency. In 4 families, the segregation of the FV Leiden mutation and the protein S deficiency could be studied. In sibships where both abnormalities were segregating, the percentage of symptomatic individuals with both abnormalities was 80%. Three of the seven subjects with only the FV Leiden mutation, and two out of the three subjects with only protein S deficiency had developed thrombosis. These results indicate that in the families presented here the combination of the FV Leiden mutation and the protein S deficiency is associated with a high risk for thrombosis. A reliable estimate of the penetrance of the single defects is not possible, because the number of individuals with a single defect is too low.


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