scholarly journals Takayasu’s arteritis: a unique ophthalmic presentation with CRAO and BRVO

2019 ◽  
Vol 12 (8) ◽  
pp. e228909
Author(s):  
Vinita Gupta ◽  
Saurabh Luthra ◽  
N Shrinkhal ◽  
Sony Sinha
Keyword(s):  
Retinal Vein Occlusion ◽  
Branch Retinal Vein Occlusion ◽  
Takayasu’S Arteritis ◽  
Central Retinal Artery Occlusion ◽  
Retinal Vessel ◽  
Initial Presentation ◽  
Takayasu's Arteritis ◽  
Vein Occlusion ◽  
The Right ◽  
Cardiovascular Evaluation

A unique case of sequential occurrence of central retinal artery occlusion (CRAO) and superotemporal branch retinal vein occlusion (ST-BRVO) in a patient of Takayasu’s arteritis is described. An 18-year-old man was diagnosed as left eye CRAO on his initial presentation and was subjected to a complete cardiovascular evaluation revealing findings diagnostic of Takayasu’s arteritis. Patient was however lost to follow-up and presented 16 months later with ST-BRVO in the right eye. Multidisciplinary intervention and an appropriate ocular intervention led to complete recovery of vision in the right eye that was maintained until his last ophthalmic evaluation (2.5 years after the initial presentation). Though uncommon, small retinal vessel involvement can occur in Takayasu’s arteritis as the inaugural feature. Hence, CRAO or branch retinal vein occlusion in a young patient, especially a male, mandates a thorough systemic evaluation and a high index of suspicion of Takayasu’s arteritis to prevent vision threatening complications.

Retinal Vessel Caliber and Risk Factors for Branch Retinal Vein Occlusion

2012 ◽  
Vol 37 (4) ◽  
pp. 334-338 ◽  
Author(s):  
Dong Ju Youm ◽  
Man Mook Ha ◽  
Yoosoo Chang ◽  
Su Jeong Song
Keyword(s):  
Risk Factors ◽  
Retinal Vein Occlusion ◽  
Branch Retinal Vein Occlusion ◽  
Retinal Vessel ◽  
Vein Occlusion ◽  
Retinal Vessel Caliber

Branch Retinal Vein Occlusion Associated with the 20210 G-to-A Prothrombin Variant

2000 ◽  
Vol 10 (2) ◽  
pp. 177-179 ◽  
Author(s):  
C. Peris Martínez ◽  
J.A. Aviñó Martínez ◽  
M. Díaz-Llopis ◽  
E. España Gregori ◽  
J.L. Menezo ◽  
...  
Keyword(s):  
Risk Factors ◽  
Fluorescein Angiography ◽  
Medical History ◽  
Retinal Vein Occlusion ◽  
Branch Retinal Vein Occlusion ◽  
Laboratory Tests ◽  
Visual Loss ◽  
Vein Occlusion ◽  
Common Risk Factors ◽  
The Right

Purpose To describe a case of branch retinal vein occlusion (BRVO) in a patient who tested positive for the 20210 A allele of the prothrombin (PT) gene. Methods A 48-year-old man had visual loss in the right eye secondary to BRVO confirmed by ophthalmoscopy and fluorescein angiography. His medical history was not remarkable for common risk factors for retinal occlusive diseases. Results Laboratory tests for hypercoagulability were positive for PT 20210 A variant. The patient's family tested negative for the PT variant. Conclusions Laboratory tests for coagulopathy, including the PT 20210 A variant, should be added to the examination of patients with central or BRVO, especially if most common risk factors for thrombosis have been excluded.

scholarly journals Ischemia-induced spreading depolarization in the retina

2016 ◽  
Vol 36 (9) ◽  
pp. 1579-1591 ◽  
Author(s):  
Anja I Srienc ◽  
Kyle R Biesecker ◽  
Angela M Shimoda ◽  
Joanna Kur ◽  
Eric A Newman
Keyword(s):  
Retinal Vein Occlusion ◽  
Branch Retinal Vein Occlusion ◽  
Vision Loss ◽  
Nmda Antagonist ◽  
Retinal Vessel ◽  
Vessel Occlusion ◽  
Spreading Depolarization ◽  
Vein Occlusion ◽  
Retinal Vessel Occlusion

Cortical spreading depolarization is a metabolically costly phenomenon that affects the brain in both health and disease. Following severe stroke, subarachnoid hemorrhage, or traumatic brain injury, cortical spreading depolarization exacerbates tissue damage and enlarges infarct volumes. It is not known, however, whether spreading depolarization also occurs in the retina in vivo. We report now that spreading depolarization episodes are generated in the in vivo rat retina following retinal vessel occlusion produced by photothrombosis. The properties of retinal spreading depolarization are similar to those of cortical spreading depolarization. Retinal spreading depolarization waves propagate at a velocity of 3.0 ± 0.1 mm/min and are associated with a negative shift in direct current potential, a transient cessation of neuronal spiking, arteriole constriction, and a decrease in tissue O2 tension. The frequency of retinal spreading depolarization generation in vivo is reduced by administration of the NMDA antagonist MK-801 and the 5-HT(1D) agonist sumatriptan. Branch retinal vein occlusion is a leading cause of vision loss from vascular disease. Our results suggest that retinal spreading depolarization could contribute to retinal damage in acute retinal ischemia and demonstrate that pharmacological agents can reduce retinal spreading depolarization frequency after retinal vessel occlusion. Blocking retinal spreading depolarization generation may represent a therapeutic strategy for preserving vision in branch retinal vein occlusion patients.

scholarly journals Branch Retinal Vein Occlusion with Vitreous Hemorrhage Identified During Intraoperative Vitrectomy

2018 ◽  
Vol 1 (1) ◽  
Author(s):  
Nafila Mahida Sukmono ◽  
Ramzi Amin
Keyword(s):  
Visual Acuity ◽  
Blood Vessels ◽  
Retinal Vein Occlusion ◽  
Branch Retinal Vein Occlusion ◽  
Laser Action ◽  
Vitreous Hemorrhage ◽  
Posterior Segment ◽  
Vein Occlusion ◽  
The Right ◽  
Photocoagulation Laser

Introduction Retinal vein occlusion is the largest group of retinal blood vessels after diabetic retinopathy. Occlusion occurring in the retinal vein is divided into central retinal vein occlusion (CRVO) occlusion and branch retinal vein occlusion (BRVO) occlusion. The Beijing Eye Study, reported a higher incidence of BRVO than CRVO, where 10-year incidents for BRVO were 1.6 per 100 subjects, and CRVO was only 0.3% 100 subjects.1 To report a case of Branch Retinal Vein Occlusion with vitreous hemorrhage identified during intraoperative vitrectomy Method: A 49-year-old woman with a history of 15 years of hypertension had right eye vision complaints, increasingly blurred since last 2 months. The right eye visual acuity 2/60 cannot be corrected and left eye 6/30 cannot be corrected. The posterior segment on right eye is difficult to assess. USG B-Scan right eye found vitreous echospike appearance of vitreous bleeding. We manage with vitrectomy and during intraoperative we identified bleeding and ghost vessel in superotemporal area. Bleeding in the superotemporal quadrant is done by photocoagulation laser action. Results: First day postoperative there was increased in visual acuity to 6/60 with a posterior segment that could be assessed, obtained tortous blood vessels, slight bleeding and ghost vessel in the superotemporal area with laser injury. Conclusion: In this case report, patients with BRVO with complications of vitreous hemorrhage performed vitrectomy with additional endolaser in the ischemic area. The result of this action of visual acuity improvement in patient.

scholarly journals Vitreous haemorrhage caused by unusual giant macular tear: a case report

2020 ◽  
Vol 48 (9) ◽  
pp. 030006052095949
Author(s):  
Chunling Lei ◽  
Li Chen
Keyword(s):  
Retinal Vein Occlusion ◽  
Branch Retinal Vein Occlusion ◽  
Pars Plana Vitrectomy ◽  
Vision Loss ◽  
Silicone Oil ◽  
Vitreous Haemorrhage ◽  
Partial Restoration ◽  
Vein Occlusion ◽  
Pars Plana ◽  
The Right

Macular tears rarely occur without trauma. Here, we describe a patient with vitreous haemorrhage, which was caused by an unusual giant macular tear secondary to existing branch retinal vein occlusion. A 60-year-old woman presented with vision loss in the right eye because of vitreous haemorrhage. She had a history of branch retinal vein occlusion and had been treated with retinal photocoagulation 3 years prior. As treatment for vitreous haemorrhage, the patient underwent 23-gauge pars plana vitrectomy combined with silicone oil tamponade. During the operation, a large jagged tear was observed in the macula. We presumed that stretching of the fibrous proliferating membrane secondary to branch retinal vein occlusion was responsible for the macular tear and vitreous haemorrhage. Eventually, the results of pars plana vitrectomy led to anatomical closure of the macular tear and partial restoration of visual acuity.

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