scholarly journals Psychosocial, behavioural, and health determinants of successful smoking cessation: a longitudinal study of Danish adults

1998 ◽  
Vol 7 (3) ◽  
pp. 262-267 ◽  
Author(s):  
M. Osler ◽  
E. Prescott
2016 ◽  
Vol 25 (Suppl 1) ◽  
pp. i90-i95 ◽  
Author(s):  
Yue-Lin Zhuang ◽  
Sharon E Cummins ◽  
Jessica Y Sun ◽  
Shu-Hong Zhu

2019 ◽  
Vol 29 (3-4) ◽  
pp. 556-566
Author(s):  
Ka Yan Ho ◽  
Ho Cheung William Li ◽  
Katherine Ka Wai Lam ◽  
Man Ping Wang ◽  
Wei Xia ◽  
...  

BMJ Open ◽  
2020 ◽  
Vol 10 (6) ◽  
pp. e033776
Author(s):  
Lorenzo Paglione ◽  
Laura Angelici ◽  
Marina Davoli ◽  
Nera Agabiti ◽  
Giulia Cesaroni

ObjectivesSocioeconomic inequalities have a strong impact on population health all over the world. Occupational status is a powerful determinant of health in rich societies. We aimed at investigating the association between occupation and mortality in a large metropolitan study.DesignCohort study.SettingRome, capital of Italy.ParticipantsWe used the Rome Longitudinal Study, the administrative cohort of residents in Rome at the 2001 general census, followed until 2015. We selected residents aged 15–65 years at baseline. For each subject, we had information on sex, age and occupation (occupational status and type of job) according to the Italian General Census recognition.Main outcome measuresWe investigated all-cause, cancer, cardiovascular and accidental mortality, major causes of death in the working-age population. We used Cox proportional hazards models to investigate the association between occupation and all-cause and cause-specific mortality in men and women.ResultsWe selected 1 466 726 subjects (52.1% women). 42 715 men and 29 915 women died during the follow-up. In men, 47.8% of deaths were due to cancer, 26.7% to cardiovascular causes and 6.4% to accidents, whereas in women 57.8% of deaths were due to cancer, 19.3% to cardiovascular causes and 3.5% to accidents. We found an association between occupational variables and mortality, more evident in men than in women. Compared with employed, unemployed had a higher risk of mortality for all causes with an HR=1.99 (95% CI 1.92 to 2.06) in men and an HR=1.49 (95% CI 1.39 to 1.60) in women. Compared with high-qualified non-manual workers, non-specialised manual workers had a higher mortality risk (HR=1.68, 95% CI 1.59 to 1.77 and HR=1.30, 95% CI 1.20 to 1.40, for men and women, respectively).ConclusionsThis study shows the importance of occupational variables as social health determinants and provides evidence for policy-makers on the necessity of integrated and preventive policies aimed at improving the safety of the living and the working environment.


2020 ◽  
Vol 24 (6) ◽  
pp. 672-680
Author(s):  
Esme Fuller-Thomson ◽  
Z. Saab ◽  
K. M. Davison ◽  
S. Lamson Lin ◽  
V. Taler ◽  
...  

Addiction ◽  
2002 ◽  
Vol 97 (2) ◽  
pp. 205-215 ◽  
Author(s):  
Martin Lindström ◽  
Sven-Olof Isacsson ◽  
The Malmö Shoulder-Neck Study Group

2006 ◽  
Vol 30 (2) ◽  
pp. 217-223
Author(s):  
Shoichi Mizuno ◽  
Masako Tomita ◽  
Takashi Murayama

BMJ Open ◽  
2020 ◽  
Vol 10 (3) ◽  
pp. e032918
Author(s):  
Marta Prieto ◽  
Laura Vicente-Vicente ◽  
Alfredo G Casanova ◽  
Maria Teresa Hernández-Sánchez ◽  
Manuel A Gomez-Marcos ◽  
...  

IntroductionTobacco causes kidney damage that can progress to chronic kidney disease. However, the diagnostic parameters used in clinics are not effective in identifying smokers at risk. Our first objective is to more effectively detect subclinical renal damage in smokers. In addition, we hypothesise that tobacco consumption can predispose smokers to renal damage on exposure to other potentially nephrotoxic events (drugs, diagnostic procedures and so on). We will test this hypothesis in our second objective by investigating whether certain predisposition markers (GM2 ganglioside activator protein (GM2AP), transferrin and t-gelsolin) are able to detect smokers who are predisposed to kidney damage. Finally, in our third objective, we will study whether smoking cessation reduces subclinical and/or predisposition to renal damage.Methods and analysisFor our first objective, a prospective cross-sectional study will be carried out with patients from a primary healthcare centre. The influence of tobacco on renal damage, in patients both with and without additional risk factors, will be studied using a panel of early biomarkers (albuminuria, N-acetyl-beta-D-glucosaminidase, kidney injury molecule-1 and neutrophil gelatinase-associated lipocalin). For our second objective, a prospective longitudinal study will be carried out with patients recruited for our first objective. We will study whether certain predisposition biomarkers (GM2AP, transferrin and t-gelsolin) are able to detect smokers predisposed to renal damage. For our third objective, a prospective longitudinal study will be carried out with patients from a smoking cessation unit. We will study the evolution of the markers described above following smoking cessation.Ethics and disseminationThe study has been approved by the Clinical Research Ethics Committee of the Healthcare Area of Salamanca. All study participants will sign an informed consent form in compliance with the Declaration of Helsinki and the WHO standards for observational studies. Results will be presented at conferences and submitted to peer-reviewed journals.Trial registration numberNCT03850756.


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