The Use of Acupuncture in Ophthalmology

The object of this study is to answer the question: Is acupuncture of any use in ophthalmology? Ophthalmology today can boast of 95% accuracy in diagnosis, but modern therapeutics has not caught up with diagnosis. Despite an inability to explain in modern scientific terms the healing power of acupuncture, documentation of over 500 cases treated shows that this modality can be successful in the treatment of eye diseases, especially in cases of retinitis pigmentosa, high myopia, cataracts, surgical aphakia, controlled glaucoma and re-attached ablation retinae. We notice in these cases that improvement occurs in the central acuity, and no improvement in the visual field, color-vision and nightblindness. Apparently acupuncture acts on the macula alone and not on the entire retina. Acupuncture is not intended to substitute for established medication or surgery in ophthalmology. It cannot restore a tissue or organ damaged beyond regeneration, "incurable" eye diseases per se are not cured by acupuncture. Only symptoms are relieved and dimness of vision is a symptom which can be alleviated in many cases with periodic treatment.

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Visual Function and Functional Vision of Retinitis Pigmentosa

Annals of Optometry and Contact Lens ◽

10.52725/aocl.2021.20.3.99 ◽

2021 ◽

Vol 20 (3) ◽

pp. 99-104

Author(s):

Hyeong Gon Yu ◽

Chang Ki Yoon

Keyword(s):

Clinical Trials ◽

Visual Field ◽

Retinitis Pigmentosa ◽

Visual Impairment ◽

Contrast Sensitivity ◽

Color Vision ◽

Visual Function ◽

Functional Vision

Patients with retinitis pigmentosa have defects in visual function, including the visual field, color vision, contrast sensitivity, and impaired functional vision, such as reading and driving. An assessment of visual function is required when interpreting the outcome of clinical trials of retinitis pigmentosa. Various defects of functional vision should be considered when assessing visual impairment for patients with retinitis pigmentosa.

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Cerebral trauma-induced dyschromatopsia in the left hemifield: case presentation

BMC Ophthalmology ◽

10.1186/s12886-020-01800-7 ◽

2021 ◽

Vol 21 (1) ◽

Author(s):

Yoko Mase ◽

Yoshitsugu Matsui ◽

Eriko Uchiyama ◽

Hisashi Matsubara ◽

Masahiko Sugimoto ◽

...

Keyword(s):

Visual Field ◽

Color Vision ◽

Temporal Lobe ◽

Fusiform Gyrus ◽

Ganglion Cell Complex ◽

Automated Perimetry ◽

Subjective Symptoms ◽

Anterior Temporal Lobe ◽

Case Presentation ◽

Cerebral Trauma

Abstract Background Acquired color anomalies caused by cerebral trauma are classified as either achromatopsias or dyschromatopsias (Zeki, Brain 113:1721–1777, 1990). The three main brain regions stimulated by color are V1, the lingual gyrus, which was designated as human V4 (hV4), and the fusiform gyrus, designated as V4α. (Zeki, Brain 113:1721–1777, 1990). An acquired cerebral color anomaly is often accompanied by visual field loss (hemi- and quadrantanopia), facial agnosia, prosopagnosia, visual agnosia, and anosognosia depending on the underlying pathology (Bartels and Zeki, Eur J Neurosci 12:172–193, 2000), (Meadows, Brain 97:615–632, 1974), (Pearman et al., Ann Neurol 5:253–261, 1979). The purpose of this study was to determine the characteristics of a patient who developed dyschromatopsia following a traumatic injury to her brain. Case presentation The patient was a 24-year-old woman who had a contusion to her right anterior temporal lobe. After the injury, she noticed color distortion and that blue objects appeared green in the left half of the visual field. Although conventional color vision tests did not detect any color vision abnormalities, short wavelength automated perimetry (SWAP) showed a decrease in sensitivity consistent with a left hemi-dyschromatopsia. Magnetic resonance imaging (MRI) detected abnormalities in the right fusiform gyrus, a part of the anterior temporal lobe. At follow-up 14 months later, subjective symptoms had disappeared, but the SWAP abnormalities persisted and a thinning of the sectorial ganglion cell complex (GCC) was detected. Conclusion The results indicate that although the subjective symptoms resolved early, a reduced sensitivity of SWAP remained and the optical coherence tomography (OCT) showed GCC thinning. We conclude that local abnormalities in the anterior section of fusiform gyrus can cause mild cerebral dyschromatopsia without other symptoms. These findings indicate that it is important to listen to the symptoms of the patient and perform appropriate tests including the SWAP and OCT at the early stage to objectively prove the presence of acquired cerebral color anomaly.

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Concentric division of 10° visual field tests in retinitis pigmentosa

Japanese Journal of Ophthalmology ◽

10.1007/s10384-013-0235-y ◽

2013 ◽

Vol 57 (3) ◽

pp. 268-274 ◽

Cited By ~ 2

Author(s):

Ken Ogino ◽

Atsushi Otani ◽

Akio Oishi ◽

Masafumi Kurimoto ◽

Takuro Sekiya ◽

...

Keyword(s):

Visual Field ◽

Retinitis Pigmentosa ◽

Field Tests

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Visual field defects and myopic macular degeneration in Singapore adults with high myopia

British Journal of Ophthalmology ◽

10.1136/bjophthalmol-2020-318674 ◽

2021 ◽

pp. bjophthalmol-2020-318674

Author(s):

Carla Lanca ◽

Chen Hsin Sun ◽

Rachel Chong ◽

Yee Ling Wong ◽

Monisha Esther Nongpiur ◽

...

Keyword(s):

Visual Field ◽

Macular Degeneration ◽

High Myopia ◽

Cross Sectional Study ◽

Visual Field Defects ◽

Mean Deviation ◽

Cross Sectional ◽

Sensitivity Loss ◽

Fundus Photographs ◽

Generalised Estimating Equations

AimsTo characterise the association between visual field (VF) defects and myopic macular degeneration (MMD) in highly myopic adults without glaucoma.MethodsParticipants (n=106; 181 eyes) with high myopia (HM; spherical equivalent ≤−5.0 D or axial length (AL) ≥26 mm), after excluding glaucoma and glaucoma suspects, from the Singapore Epidemiology of Eye Diseases-HM study were included in this cross-sectional study. Humphrey VF (central 24–2 threshold), cup-disc ratio (CDR) and intraocular pressure (IOP) measurements were performed. Mean deviation (MD) and pattern SD (PSD), VF defects (normal or abnormal; p<0.05 in ≥3 non-edge contiguous locations) and pattern (eg, generalised sensitivity loss) were analysed. MMD presence was diagnosed from fundus photographs. Generalised estimating equations were used for analysing factors (MD, PSD, VF defects, CDR and IOP) associated with MMD.ResultsMean age was 55.4±9.9 years and 51.9% were women (AL=26.7±1.1 mm). MMD eyes had lower MD (−3.8±2.9 dB vs −1.1±1.4 dB) and higher PSD (2.8±1.7 dB vs 1.7±0.6 dB). A higher percentage of MMD eyes (n=48) had abnormal VF (62.5% vs 28.6%; p<0.001) compared with no MMD (n=133 eyes). VF pattern in MMD eyes was significantly different from eyes without MMD (p=0.001) with greater generalised sensitivity loss (53.3% vs 10.5%) and arcuate defects (16.7% vs 10.5%). In multivariate analyses, MD (OR=1.52) and PSD (OR=1.67) were significantly (p=0.003) associated with MMD, but VF defects were not associated with MMD.ConclusionHighly myopic adults with MMD may have VF loss when compared with highly myopic patients without MMD even in adults without glaucoma.

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A novel mutation in the PRPH2 gene in a Chinese pedigree with retinitis pigmentosa and angle-closure glaucoma

BMC Ophthalmology ◽

10.1186/s12886-021-02064-5 ◽

2021 ◽

Vol 21 (1) ◽

Author(s):

Wei-ning Li ◽

Xiu-juan Du ◽

Yu-ting Zhang ◽

Le-yi Wang ◽

Jing Zhu

Keyword(s):

Visual Field ◽

Retinitis Pigmentosa ◽

Novel Mutation ◽

Genetic Mutation ◽

Angle Closure Glaucoma ◽

Angle Closure ◽

The Novel ◽

Progressive Loss ◽

Whole Exome ◽

Transversion Mutation

Abstract Background Retinitis pigmentosa (RP) is a rare, progressive, and hereditary disorder that leads to the progressive loss of vision and visual field, and in some cases blindness. The specific relationship between RP and glaucoma has been debated for decades. Methods In this study, we examined a Han RP family with concomitant angle-closure glaucoma (ACG), performed an inductive analysis of their clinical features and assistant results, and applied whole-exome sequencing (WES) technology for a molecular diagnosis. Results A novel transversion mutation (c.626 T > A) was identified in the peripherin-2 (PRPH2) gene in the proband, resulting in the substitution of Valine to aspartic acid in codon 209. A full ophthalmic examination showed that the proband with the c.626 T > A mutation had a typical RP manifestation, with close angles; however, the proband’s elder brother, who lacked the novel mutation, had a normal fundus and open angles. Conclusion Our results extend the genetic mutation spectrum of PRPH2 in RP, and provide evidence to support a genetic correlation between RP and ACG.

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