Urinary Porphyrins Among PVC Workers

2015 ◽  
pp. 352-355 ◽  
Author(s):  
C.-E. Lange ◽  
H. Bloch ◽  
G. Veltman ◽  
M. Doss
Keyword(s):  
2014 ◽  
pp. 1333-1348 ◽  
Author(s):  
Janet K. Kern ◽  
David A. Geier ◽  
Lisa Sykes ◽  
Mark Geier

2019 ◽  
Vol 493 ◽  
pp. S455-S456
Author(s):  
C. Castillo Perez ◽  
F.J. Illana Camara ◽  
M.J. Torrejon Martinez

1970 ◽  
Vol 39 (2) ◽  
pp. 159-168 ◽  
Author(s):  
P. Goldswain ◽  
E. Dowdle ◽  
Norma Spong ◽  
L. Eales

1. The specific activities of urinary uroporphyrin and coproporphyrin were measured as functions of time following the administration of a single oral dose of [4-14C] δ-aminolaevulic acid (ALA) to six patients with symptomatic porphyria and one control subject. 2. The peak specific activity of coproporphyrin preceded that of uroporphyrin in all subjects studied and exceeded that of uroporphyrin in the patients with symptomatic porphyria. 3. These results are interpreted as indicating the existence of two distinct metabolic pathways in the liver for the disposal of ALA, rather than as contradicting the generally accepted role of uroporphyrinogen as a precursor of coproporphyrinogen.


1970 ◽  
Vol 16 (4) ◽  
pp. 331-334 ◽  
Author(s):  
J Fischl ◽  
F Eichhorn ◽  
A Ruttenberg ◽  
Ch Major

Abstract Urinary porphyrins are concentrated by adsorption on talc, from which they are eluted with ammonium hydroxide. The eluate is electrophoresed on a membrane filter medium in an alkaline buffer (pH 10.5) for 10 to 20 min. Porphobilinogen and δ-aminolevulinic acid in urine are separated within 90 min by "small-volume" electrophoresis in a pH 2.2 buffer.


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