scholarly journals Angiopoietin-2 exacerbates cardiac hypoxia and inflammation after myocardial infarction

2018 ◽  
Vol 128 (11) ◽  
pp. 5018-5033 ◽  
Author(s):  
Seung-Jun Lee ◽  
Choong-kun Lee ◽  
Seok Kang ◽  
Intae Park ◽  
Yoo Hyung Kim ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Angiopoietin 2 ◽  
Cardiac Hypoxia

scholarly journals Elevated plasma thrombomodulin and angiopoietin-2 predict the development of acute kidney injury in patients with acute myocardial infarction

Critical Care ◽  
2014 ◽  
Vol 18 (3) ◽  
pp. R100 ◽  
Author(s):  
Kuan-Liang Liu ◽  
Kuang-Tso Lee ◽  
Chih-Hsiang Chang ◽  
Yung-Chang Chen ◽  
Shu-Min Lin ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Acute Kidney Injury ◽  
Kidney Injury ◽  
Elevated Plasma ◽  
Angiopoietin 2

Angiopoietin-2 in acute myocardial infarction complicated by cardiogenic shock-a biomarker substudy of the IABP-SHOCK II-Trial

2015 ◽  
Vol 17 (11) ◽  
pp. 1152-1160 ◽  
Author(s):  
Janine Pöss ◽  
Georg Fuernau ◽  
Daniel Denks ◽  
Steffen Desch ◽  
Ingo Eitel ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Cardiogenic Shock ◽  
Angiopoietin 2

Enhanced expression of angiopoietin-2 and the Tie2 receptor but not angiopoietin-1 or the Tie1 receptor in a rat model of myocardial infarction

2004 ◽  
Vol 11 (2) ◽  
pp. 163-171 ◽  
Author(s):  
Kou-Gi Shyu ◽  
Yao-Jen Liang ◽  
Hang Chang ◽  
Bao-Wei Wang ◽  
Jyh-Gang Leu ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Rat Model ◽  
Tie2 Receptor ◽  
Enhanced Expression ◽  
Angiopoietin 2 ◽  
Angiopoietin 1

An electron microscopic study of the intra-myocardial lesions in cases of acute myocardial infarction

1978 ◽  
Vol 36 (2) ◽  
pp. 484-485
Author(s):  
Masahiro Ono ◽  
Kaoru Aihara ◽  
Gompachi Yajima
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Coronary Vessels ◽  
Vascular Wall ◽  
Vascular Changes ◽  
Electron Microscopic ◽  
Main Trunk ◽  
Mechanical Destruction ◽  
Transmission Electron ◽  
Myocardial Lesions

The pathogenesis of the arteriosclerosis in the acute myocardial infarction is the matter of the extensive survey with the transmission electron microscopy in experimental and clinical materials. In the previous communication,the authors have clarified that the two types of the coronary vascular changes could exist. The first category is the case in which we had failed to observe no occlusive changes of the coronary vessels which eventually form the myocardial infarction. The next category is the case in which occlusive -thrombotic changes are observed in which the myocardial infarction will be taken placed as the final event. The authors incline to designate the former category as the non-occlusive-non thrombotic lesions. The most important findings in both cases are the “mechanical destruction of the vascular wall and imbibition of the serous component” which are most frequently observed at the proximal portion of the coronary main trunk.

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