Comparison of Cognitive Impairment Associated With Major Depression Following Stroke Versus Traumatic Brain Injury

2002 ◽  
Vol 43 (4) ◽  
pp. 295-301 ◽  
Author(s):  
Amane Tateno ◽  
Yuichi Murata ◽  
Robert G. Robinson
Keyword(s):  
Traumatic Brain Injury ◽  
Cognitive Impairment ◽  
Brain Injury ◽  
Major Depression

Cognitive Impairment Associated With Major Depression Following Mild and Moderate Traumatic Brain Injury

2005 ◽  
Vol 17 (1) ◽  
pp. 61-65 ◽  
Author(s):  
Mark J. Rapoport ◽  
Scott McCullagh ◽  
Prathiba Shammi ◽  
Anthony Feinstein
Keyword(s):  
Traumatic Brain Injury ◽  
Cognitive Impairment ◽  
Brain Injury ◽  
Major Depression ◽  
Moderate Traumatic Brain Injury

NEUROPSYCHOLOGICAL CHANGES IN PATIENTS WITH A CONSEQUENCE OF TRAUMATIC BRAIN INJURY

2020 ◽  
Vol 3 (1) ◽  
pp. 44-46
Author(s):  
Istatillo Shodjalilov ◽  
◽  
Saoda Igamova ◽  
Aziza Djurabekova
Keyword(s):  
Traumatic Brain Injury ◽  
Cognitive Impairment ◽  
Brain Injury ◽  
Neuropsychological Testing ◽  
Anxiety And Depression ◽  
Psychopathological Symptoms ◽  
Neuropsychological Changes ◽  
The Relationship

The incidence of cognitive impairment in TBI is high, depending on the severity. At the same time, psychopathological symptoms in the form of asthenia, increased anxiety and depression are encountered among patients with TBI. The work studied the relationship between cognitive and psychopathological symptoms in patients with TBI using neuropsychological testing on scales.

scholarly journals Major Depression Following Traumatic Brain Injury

2004 ◽  
Vol 61 (1) ◽  
pp. 42 ◽  
Author(s):  
Ricardo E. Jorge ◽  
Robert G. Robinson ◽  
David Moser ◽  
Amane Tateno ◽  
Benedicto Crespo-Facorro ◽  
...  
Keyword(s):  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Major Depression

scholarly journals Pioglitazone attenuates mitochondrial dysfunction, cognitive impairment, cortical tissue loss, and inflammation following traumatic brain injury

2011 ◽  
Vol 227 (1) ◽  
pp. 128-135 ◽  
Author(s):  
Andrew Sauerbeck ◽  
Jianxin Gao ◽  
Ryan Readnower ◽  
Mei Liu ◽  
James R. Pauly ◽  
...  
Keyword(s):  
Traumatic Brain Injury ◽  
Cognitive Impairment ◽  
Brain Injury ◽  
Mitochondrial Dysfunction ◽  
Tissue Loss ◽  
Cortical Tissue

Traumatic Brain Injury

2008 ◽  
Author(s):  
Vani Rao
Keyword(s):  
Traumatic Brain Injury ◽  
Brain Injury ◽  
Major Depression ◽  
Psychiatric Disorders ◽  
Financial Burden ◽  
Treatment Strategies ◽  
Chronic Alcoholism ◽  
The United States ◽  
Psychiatric Disturbances ◽  
Psychotic Features

Traumatic brain injury (TBI) is a significant cause of disability in the United States, with an incidence of about 1.5 million cases per year (National Institutes of Health Consensus Development Panel, 1999). It is associated with both neurologic and psychiatric consequences. Although the neurologic problems usually stabilize with time, the psychiatric disorders often continue to remit and relapse. Factors associated with the development of psychiatric disorders include older age, arteriosclerosis, and chronic alcoholism, all of which interfere with the reparative process within the central nervous system. Other contributors to psychiatric disability include a pre-TBI history of psychiatric illness, illicit drug abuse, and lack of social support. Because post-TBI psychiatric disturbances interfere with rehabilitation and cause emotional and financial burden for patients and caregivers, early diagnosis and treatment are important. Post-TBI psychiatric disturbances are best classified according to their clinical presentation. These disturbances are discussed below and their pharmacologic and nonpharmacologic treatment strategies are recommended. The mood disturbances most commonly associated with TBI are major depression, mania, anxiety, and apathy. Major depression is seen in about 25% of people with TBI. Symptoms of major depression include persistent sadness; guilt; feelings of worthlessness; hopelessness; suicidal thoughts; anhedonia; and changes in patterns of sleep, appetite, and energy. Sometimes these symptoms may be associated with psychotic features such as delusions and hallucinations. It is important to remember that changes in sleep, appetite, or energy are not specific to the syndrome of major depression and may be due to the brain injury itself, or to the noise, stimulation, or deconditioning associated with hospitalization. If due to the latter conditions, gradual improvement occurs with time in most patients. Sadness in excess of the severity of injury and poor participation in rehabilitation are strong indicators of the presence of major depression. The presence of poor social functioning pre-TBI and left dorsolateral frontal and/or left basal ganglia lesion have been associated with an increased probability of developing major depression following brain injury ( Jorge et al., 1993a; Jorge et al., 2004). Major depression should be differentiated from demoralization, primary apathy syndrome, and pathologic crying.

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