scholarly journals Cathepsins in Human Obesity: Changes in Energy Balance Predominantly Affect Cathepsin S in Adipose Tissue and in Circulation

2010 ◽  
Vol 24 (3) ◽  
pp. 680-680
Author(s):  
Nadia Naour ◽  
Christine Rouault ◽  
Soraya Fellahi ◽  
Marie-Eve Lavoie ◽  
Christine Poitou ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Energy Balance ◽  
Cathepsin S ◽  
Human Obesity

scholarly journals Cathepsins in Human Obesity: Changes in Energy Balance Predominantly Affect Cathepsin S in Adipose Tissue and in Circulation

2010 ◽  
Vol 95 (4) ◽  
pp. 1861-1868 ◽  
Author(s):  
Nadia Naour ◽  
Christine Rouault ◽  
Soraya Fellahi ◽  
Marie-Eve Lavoie ◽  
Christine Poitou ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Energy Balance ◽  
Cathepsin S ◽  
Human Obesity

Transcriptomic and Metabolomic Profiling of Chicken Adipose Tissue: Dual Purpose Benefit for Human Obesity and Poultry Production

2018 ◽  
Vol 6 (2) ◽  
pp. 96-102
Author(s):  
Ronique C. Beckford ◽  
Eric D. Tague ◽  
Shawn R. Campagna ◽  
Brynn H. Voy
Keyword(s):  
Adipose Tissue ◽  
Poultry Production ◽  
Human Obesity ◽  
Dual Purpose ◽  
Metabolomic Profiling

Adipose Tissue and Circulating Endothelial Cell Specific Molecule-1 in Human Obesity

2006 ◽  
Vol 38 (1) ◽  
pp. 28-33 ◽  
Author(s):  
J. Janke ◽  
S. Engeli ◽  
K. Gorzelniak ◽  
M. Feldpausch ◽  
U. Heintze ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Endothelial Cell ◽  
Human Obesity

Role of thermogenesis in the regulation of energy balance in relation to obesity

1989 ◽  
Vol 67 (4) ◽  
pp. 394-401 ◽  
Author(s):  
Jean Himms-Hagen
Keyword(s):  
Adipose Tissue ◽  
Energy Balance ◽  
Energy Expenditure ◽  
Brown Adipose Tissue ◽  
Thermal Balance ◽  
Prominent Feature ◽  
Thermic Effect Of Food ◽  
Brown Adipose ◽  
Effect Of Food ◽  
Thermic Effect

Obligatory thermogenesis is a necessary accompaniment of all metabolic processes involved in maintenance of the body in the living state, and occurs in ail organs. It includes energy expenditure involved in ingesting, digesting, and processing food (thermic effect of food (TEF)). At certain life stages extra energy expenditure for growth, pregnancy, or lactation would also be obligatory. Facultative thermogenesis is superimposed on obligatory thermogenesis and can be rapidly switched on and rapidly suppressed by the nervous system. Facultative thermogenesis is important in both thermal balance, in which control of thermoregulatory thermogenesis (shivering in muscle, nonshivering in brown adipose tissue (BAT)) balances neural control of heat loss mechanisms, and in energy balance, in which control of facultative thermogenesis (exercise-induced in muscle, diet-induced thermogenesis (DIT) in BAT) balances control of energy intake. Thermal balance (i.e., body temperature) is much more stringently controlled than energy balance (i.e., body energy stores). Reduced energy expenditure for thermogenesis is important in two types of obesity in laboratory animals. In the first type, deficient DIT in BAT is a prominent feature of altered energy balance. It may or may not be associated with hyperphagia. In a second type, reduced cold-induced thermogenesis in BAT as well as in other organs is a prominent feature of altered thermal balance. This in turn results in altered energy balance and obesity, exacerbated in some examples by hyperphagia. In some of the hyperphagic obese animals it is likely that the exaggerated obligatory thermic effect of food so alters thermal balance that BAT thermogenesis is suppressed. In all obese animals, deficient hypothalamic control of facultative thermogenesis and (or) food intake is implicated.Key words: thermogenesis, brown adipose tissue, energy balance, obesity, cold, thermoregulation, diet.

scholarly journals Adipose tissue fibrosis, hypertrophy, and hyperplasia: Correlations with diabetes in human obesity

Obesity ◽  
2016 ◽  
Vol 24 (3) ◽  
pp. 597-605 ◽  
Author(s):  
Lindsey A. Muir ◽  
Christopher K. Neeley ◽  
Kevin A. Meyer ◽  
Nicki A. Baker ◽  
Alice M. Brosius ◽  
...  
Keyword(s):  
Adipose Tissue ◽  
Human Obesity ◽  
Tissue Fibrosis

Neuroendocrine regulation of energy balance: Implications on the development and surgical treatment of obesity

2017 ◽  
Vol 23 (3) ◽  
pp. 131-146 ◽  
Author(s):  
Gisele Farias ◽  
Bárbara Dal Molin Netto ◽  
Solange Cravo Bettini ◽  
Ana Raimunda Dâmaso ◽  
Alexandre Coutinho Teixeira de Freitas
Keyword(s):  
Weight Loss ◽  
Adipose Tissue ◽  
Gastrointestinal Tract ◽  
Energy Balance ◽  
Public Health Problem ◽  
Neuroendocrine Regulation ◽  
Calorie Intake ◽  
Hormone Signaling ◽  
Sustained Weight Loss

Introduction: Obesity, a serious public health problem, occurs mainly when food consumption exceeds energy expenditure. Therefore, energy balance depends on the regulation of the hunger–satiety mechanism, which involves interconnection of the central nervous system and peripheral signals from the adipose tissue, pancreas and gastrointestinal tract, generating responses in short-term food intake and long-term energy balance. Increased body fat alters the gut- and adipose-tissue-derived hormone signaling, which promotes modifications in appetite-regulating hormones, decreasing satiety and increasing hunger senses. With the failure of conventional weight loss interventions (dietary treatment, exercise, drugs and lifestyle modifications), bariatric surgeries are well-accepted tools for the treatment of severe obesity, with long-term and sustained weight loss. Bariatric surgeries may cause weight loss due to restriction/malabsorption of nutrients from the anatomical alteration of the gastrointestinal tract that decreases energy intake, but also by other physiological factors associated with better results of the surgical procedure. Objective: This review discusses the neuroendocrine regulation of energy balance, with description of the predominant hormones and peptides involved in the control of energy balance in obesity and all currently available bariatric surgeries. Conclusions: According to the findings of our review, bariatric surgeries promote effective and sustained weight loss not only by reducing calorie intake, but also by precipitating changes in appetite control, satiation and satiety, and physiological changes in gut-, neuro- and adipose-tissue-derived hormone signaling.

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