Idiopathic familial facial nerve paralysis

2000 ◽  
Vol 114 (2) ◽  
pp. 132-134 ◽  
Author(s):  
W. A. Clement ◽  
A. White
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Facial Palsy ◽  
Facial Nerve Paralysis ◽  
Unknown Aetiology ◽  
Nerve Paralysis ◽  
Minimal Residual

A 26-year-old man was seen one day after developing a left facial palsy of unknown aetiology. He had previously had a left facial palsy at age 14 and a right facial palsy at 19, both with minimal residual paresis. Both his mother and grandmother have had facial palsies. The role of hereditary influences in idiopathic facial paralysis, as well as the treatment of this condition, is discussed.

S230 – MRI Findings in Delayed Facial Palsy after Stapedectomy

2008 ◽  
Vol 139 (2_suppl) ◽  
pp. P152-P152 ◽  
Author(s):  
Marc Cohen ◽  
Claudia Kirsch ◽  
Abie Mendelsohn ◽  
Akira Ishiyama
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Facial Palsy ◽  
Rare Complication ◽  
Internal Auditory Canal ◽  
Facial Nerve Paralysis ◽  
Mri Findings ◽  
Nerve Paralysis ◽  
Oral Corticosteroids ◽  
Magnetic Resonance Imaging Mri

Objectives 1) To retrospectively review the pathophysiology and radiologic features of delayed facial palsy after stapedectomy. 2) To discuss the management strategy of this unusual problem. Methods 450 stapedectomies performed at our institution were retrospectively reviewed from 1997 to 2007. A total of 2 patients in this series developed a delayed facial paralysis postoperatively. The clinical presentation, radiographic characteristics on magnetic resonance imaging (MRI), and the management of these patients are presented. Results A total of 2 patients out of 450 stapedectomies within a 10-year period developed the rare complication of delayed facial paralysis (postoperative days 12 and 41, respectively). Both patients developed rapidly progressive complete facial nerve paralysis preceded by periauricular pain and dysguesia. No sign of infection was present in the operated ear. MRI with gadolinium of the internal auditory canal demonstrated gadolinium enhancement of the labyrinthine portion of the facial nerve as seen in Bell's palsy. Both patients were treated with oral corticosteroids and antiviral medications. Subsequent imaging revealed decreased enhancement of the facial nerve with complete resolution of facial paralysis. Conclusions In the rare complication of delayed facial paralysis after stapedectomy, MR imaging is a useful diagnostic tool to dictate the treatment strategy. Delayed facial paralysis following stapedectomy can be treated medically when the MRI scan demonstrates enhancement of the facial nerve in the labyrinthine segment.

scholarly journals Role of topodiagnostic tests in evaluation of facial nerve palsy in head injury

2020 ◽  
Vol 6 (10) ◽  
pp. 1874
Author(s):  
Manish Munjal ◽  
Hem Lata Badyal ◽  
Anju Mehndiratta ◽  
Shubham Munjal ◽  
Iti Bharadwaj ◽  
...  
Keyword(s):  
Head Injury ◽  
Facial Nerve ◽  
Facial Palsy ◽  
Nerve Palsy ◽  
Facial Nerve Palsy ◽  
Facial Nerve Paralysis ◽  
Acoustic Reflex ◽  
Nerve Paralysis ◽  
Medical College

<p class="Normal1"><strong>Background:</strong> Head injury with temporal bone trauma manifests with facial nerve palsy. The site of lesion can be assessed by various tests.</p><p class="Normal1"><strong>Methods: </strong>A retrospective study of 500 cases of head injury was undertaken to study the role of topodiagnostic tests in localising the site of lesion in 48 patients of facial palsy. The study was undertaken by the otology services of Dayanand medical college and hospital, Ludhiana during a period of one year.</p><p class="Normal1">Cases: 500 cases admitted with head injury were screened. The cases with facial nerve paralysis were then enrolled in the study.</p><p class="Normal1"><strong>Results: </strong>In 48 patients of facial palsy, taste sensation was diminished in 67% (21 cases); acoustic reflex absent in 86.8% (33 cases) and Schirmer’s test showed reduced lacrimation in 29.1% (14 cases).</p><p class="Normal1"><strong>Conclusions: </strong>The topodiagnostic tests do not always localise the site of lesion in head injury.</p>

Recurrent bilateral peripheral facial palsy

1989 ◽  
Vol 103 (1) ◽  
pp. 117-119 ◽  
Author(s):  
N. Stahl ◽  
T. Ferit
Keyword(s):  
Facial Nerve ◽  
Facial Palsy ◽  
Common Finding ◽  
Facial Nerve Paralysis ◽  
Nerve Paralysis ◽  
Peripheral Facial Palsy ◽  
Local Disease ◽  
Idiopathic Facial Palsy

AbstractFacial nerve paralysis is a common otolaryngological diagnosis. Recurrent unilateral peripheral facial palsy is found in about 7 per cent of the cases. Simultaneous bilateral facial palsy is relatively uncommon and occurs in 0.3–2.0 per cent of cases of facial palsy. Recurrent. simultaneous, bilateral, idiopathic facial palsy to the best of our knowledge has never been reported. A case of recurrent, simultaneous, bilateral, idiopathic facial palsy is presented. No evidence of systemic or local disease was found in both attacks of peripheral facial palsies. The association with states of stress is the only common finding between the two attacks.

Facial Nerve Paralysis Induced by Herpes Simplex Virus in Mice: An Animal Model of Acute and Transient Facial Paralysis

1995 ◽  
Vol 104 (7) ◽  
pp. 574-581 ◽  
Author(s):  
Toshiaki Sugita ◽  
Yasuo Fujiwara ◽  
Shingo Murakami ◽  
Yoshinari Hirata ◽  
Naoaki Yanagihara ◽  
...  
Keyword(s):  
Herpes Simplex Virus ◽  
Herpes Simplex ◽  
Facial Nerve ◽  
Facial Paralysis ◽  
Bell’S Palsy ◽  
Facial Nerve Paralysis ◽  
Bell's Palsy ◽  
Vacuolar Degeneration ◽  
Nerve Paralysis ◽  
Simplex Virus

We have been the first to succeed in producing an acute and transient facial paralysis simulating Bell's palsy, by inoculating herpes simplex virus into the auricles or tongues of mice. The KOS strain of the virus was injected into the auricle of 104 mice and the anterior two thirds of the tongue in 30 mice. Facial paralysis developed between 6 and 9 days after virus inoculation, continued for 3 to 7 days, and then recovered spontaneously. The animals were painlessly sacrificed between 6 and 20 days after inoculation for histopathologic and immunocytochemical study. Histopathologically, severe nerve swelling, inflammatory cell infiltration, and vacuolar degeneration were manifested in the affected facial nerve and nuclei. Herpes simplex virus antigens were also detected in the facial nerve, geniculate ganglion, and facial nerve nucleus. The pathophysiologic mechanisms of the facial paralysis are discussed in light of the histopathologic findings, in association with the causation of Bell's palsy.

Facial Nerve Paralysis with Benign Parotid Masses

1989 ◽  
Vol 98 (8) ◽  
pp. 644-647 ◽  
Author(s):  
Howard L. DeLozier ◽  
Martin J. Spinella ◽  
Glenn D. Johnson
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Case Reports ◽  
Facial Nerve Paralysis ◽  
Local Inflammation ◽  
Nerve Paralysis ◽  
Seventh Nerve ◽  
Rapid Enlargement ◽  
Pathologic Findings ◽  
Physical Findings

Facial paralysis in the presence of a parotid mass has been associated classically with a presumed diagnosis of malignancy. However, isolated case reports have documented the occurrence of paresis or paralysis secondary to pathologically benign, nonneurogenic parotid lesions. These previous cases have been reviewed and three additional cases are described. Comparisons are made on age, sex, symptoms, physical findings, pathologic findings, and prognosis. Involvement of the seventh nerve may be explained on the basis of compression, especially in association with local inflammation. Although facial paralysis still should be considered indicative of a malignancy, it also may be caused by benign masses, particularly those associated with rapid enlargement and/or infection.

S236 – Novel Biomarker Use in the Evaluation of Facial Nerve Palsy

2008 ◽  
Vol 139 (2_suppl) ◽  
pp. P154-P154
Author(s):  
Carrie Flanagan ◽  
Shaw Gerry ◽  
Rick Odland
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Computer Software ◽  
Correlation Coefficients ◽  
Acute Onset ◽  
Facial Nerve Paralysis ◽  
Linear Regression Models ◽  
Serum Samples ◽  
Nerve Paralysis ◽  
Student’S T

Objectives 1) To determine whether a novel biomarker (hyperphosphorlyated neurofilament-heavy subunit) is able to be quantitated in patients with idiopathic facial nerve paralysis. 2) To determine whether correlation exists between biomarker quantitation and clinical parameters in patients with idiopathic facial nerve paralysis. Methods This is a prospective pilot study that assesses the utility of the serum biomarker hyperphosphorylated neurofilament-heavy subunit protein(NF-H) in evaluating patients with idiopathic facial nerve paralysis. From May 2006 to August 2007, 12 patients that presented to the Emergency, Neurology, and Otolaryngology departments at a county hospital with acute onset unilateral facial paralysis were enrolled into the study. Serum samples at the time of presentation were obtained, and the following additional data was recorded and analyzed: age, sex, severity of paralysis at presentation, duration between time of onset of paralysis and presentation, side of paresis, associated comorbidities, time to recovery, and ancillary testing results. Data analysis was performed using Student's T-test and analysis of variance; linear regression models and correlation coefficients were calculated using Microsoft Excel computer software. Results There was no significant relationship between patient age (r2=0.20), sex (p=0.19), and side of paralysis (p=0.49) and biomarker level. The biomarker level increased with increasing facial paresis severity (p=0.002, r2=0.17), and had a more pronounced correlation when patients presented within the first 24 to 48 hours of paralysis (p=0.006, r2=0.59). There was no correlation between biomarker and prognosis or ancillary testing results. Conclusions Hyperphosphorylated neurofilament-heavy subunit levels correlated with initial severity of facial paralysis, but were time-dependent.

scholarly journals Intratemporal Facial Nerve Paralysis- A Three Year Study

2016 ◽  
Vol 24 (2) ◽  
pp. 94-99
Author(s):  
Anirban Ghosh ◽  
Sankar Prasad Bera ◽  
Somnath Saha
Keyword(s):  
Facial Nerve ◽  
Facial Palsy ◽  
Bell’S Palsy ◽  
Facial Nerve Paralysis ◽  
Bell's Palsy ◽  
Prospective Longitudinal Study ◽  
Nerve Paralysis ◽  
Excellent Outcome ◽  
Stapedial Reflex ◽  
Prospective Longitudinal

Introduction This study on intratemporal facial paralysis is an attempt to understand the aetiology of facial nerve paralysis, effect of different management protocols and the outcome after long-term follow-up. Materials and Methods A prospective longitudinal study was conducted from September 2005 to August 2008 at the Department of Otorhinolaryngology of a medical college in Kolkata comprising 50 patients of intratemporal facial palsy. All cases were periodically followed up for at least 6 months and their prognostic outcome along with different treatment options were analyzed. Result Among different causes of facial palsy, Bell’s palsy is the commonest cause; whereas cholesteatoma and granulation were common findings in otogenic facial palsy. Traumatic facial palsies were exclusively due to longitudinal fracture of temporal bone running through geniculate ganglion. Herpes zoster oticus and neoplasia related facial palsies had significantly poorer outcome. Discussion Otogenic facial palsy showed excellent outcome after mastoid exploration and facial decompression. Transcanal decompression was performed in traumatic facial palsies showing inadequate recovery. Complete removal of cholesteatoma over dehiscent facial nerve gave better postoperative recovery. Conclusion The stapedial reflex test is the most objective and reproducible of all topodiagnostic tests. Return of the stapedial reflex within 3 weeks of injury indicates good prognosis. Bell’s palsy responded well to conservative measures. All traumatic facial palsies were due to longitudinal fracture and 2/3rd of these patients showed favourable outcome with medical therapy.

scholarly journals Ionophoresis and LASER applications in facial nerve palsy

2019 ◽  
Vol 10 (Vol 10 No. 4) ◽  
pp. 551-554
Author(s):  
Cristina Octaviana DAIA ◽  
Ștefana CROITORU ◽  
Ioana CAZACU ◽  
Ruxandra SCUTUROIU ◽  
Octaviana DAIA ◽  
...  
Keyword(s):  
Complete Remission ◽  
Facial Nerve ◽  
Temporomandibular Joint ◽  
Facial Palsy ◽  
Nerve Palsy ◽  
Facial Nerve Palsy ◽  
Facial Nerve Paralysis ◽  
Nerve Paralysis ◽  
Combined Application ◽  
Number Of Patients

Background and aim of the study. The aim of this article is to present the functional gain of the specific rehabilitation program in patients with facial nerve paralysis, irrespective of etiology, following a complex physiotherapeutic treatment, consisting of the combined application of ionophoresis and LASER. Material and Method. We performed a retrospective analysis of 26 patients having their consent and The Theaching Emergency Hospital “Bagdasar-Arseni” (TEHBA) ethics Committee’s approval, N.O. 683/21.02.2019. The patients were admitted to the Neuro-Muscular Rehabilitation Clinic Division of TEHBA between April 2011 and March 2019. The patients were over the age of 18, diagnosed with facial nerve palsy and received physiotherapy consisting of the combined application of: ionophoresis with 1% potassium iodide, applied to positive electrode, preauricular, and the negative electrode, retroauricular, for 20 minutes and LASER, to the temporomandibular joint on the affected part, 4 points : radiant exposure per dose 3 J/cm2, frequency 5 Hz, probe area 1 cm2, power 62 mW, for 10 sessions and two sets of the same formula at the temporomandibular joint on the opposite side. Results. From the etiological point of view, the study included an equal number of patients with peripheric facial palsy (PFP) and with central facial palsy (CFP), respectively 13 in each group. Overall, at least 1 patient in 2 had a positive response to physiotherapy. In the patients with PFP, 77% of patients responded to the treatment and 46% had complete remission. In the patients with CFP, only 30% responded to treatment and only one had complete remission. Approximately half of the patients did not have any adverse reactions (46%), while the rest showed temporary erythema (46%), or local tingling sensation over a variable period of time (8%). Conclusion. Applying the combination of ionophoresis and LASER as physiotherapic treatment is particularly effective in peripheric facial nerve paresis. The method is safe and well tolerated, therefore we propose its use in this type of pathology. Key words: Laser therapy, facial nerve paralysis, ionophoresis,

Acute suppurative parotitis and facial paralysis

1996 ◽  
Vol 110 (1) ◽  
pp. 91-92 ◽  
Author(s):  
Y. T. Pang ◽  
C. H. Raine
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Rare Complication ◽  
Facial Nerve Paralysis ◽  
Nerve Paralysis

AbstractA case of facial nerve paralysis secondary to acute suppurative parotitis is described. This is a rare complication in the absence of malignant processes in the parotid.

scholarly journals Rare Presentation of Rhino-Orbital-Cerebral Zygomycosis: Bilateral Facial Nerve Palsy

2011 ◽  
Vol 2011 ◽  
pp. 1-2 ◽  
Author(s):  
Alireza Mohebbi ◽  
Hesam Jahandideh ◽  
Ali Amini Harandi
Keyword(s):  
Facial Nerve ◽  
Facial Paralysis ◽  
Nerve Palsy ◽  
Facial Nerve Palsy ◽  
Interesting Case ◽  
Facial Nerve Paralysis ◽  
Rare Presentation ◽  
Nerve Paralysis ◽  
Healthy Man ◽  
Immunocompromised Individual

Rhino-orbital-cerebral zygomycosis afflicts primarily diabetics and immunocompromised individual, but can also occur in normal hosts rarely. We here presented an interesting case of facial nerve palsy and multiple cold abscesses of neck due to rhino-orbital-cerebral zygomycosis in an otherwise healthy man. Although some reports of facial nerve paralysis in conjunction with rhino-orbital-cerebral zygomycosis exist, no case of bilateral complete facial paralysis has been reported in the literature to date.

Sign in / Sign up

Export Citation Format

Share Document