Ultrasonicarly induced nuclear aberrations in anin vitromulticellular tumour system

1982 ◽  
Vol 55 (653) ◽  
pp. 362-365 ◽  
Author(s):  
Peter G. Sacks ◽  
Morton W. Miller
Keyword(s):  
Tumour System

Transition metals in an experimental tumour system

1979 ◽  
Vol 35 (4) ◽  
pp. 460-461 ◽  
Author(s):  
S. S. Ranade ◽  
Smita Shah ◽  
Pallavi Haria
Keyword(s):  
Transition Metals ◽  
Experimental Tumour ◽  
Tumour System

Detection of an Increase in Ascorbate Radical in an Irradiated Experimental Tumour System Using ESR

1995 ◽  
Vol 68 (4) ◽  
pp. 467-473 ◽  
Author(s):  
H. Isoda ◽  
K. Akagi ◽  
T. Hasegawa ◽  
Y. Tanaka ◽  
T. Kihara ◽  
...  
Keyword(s):  
Experimental Tumour ◽  
Tumour System

scholarly journals Comparison Between Different Techniques of Immunoprotection in the A-SW Mouse/SEWA Tumour System

1974 ◽  
Vol 29 (4) ◽  
pp. 300-304
Author(s):  
R Favre ◽  
Y Carcassonne ◽  
G Meyer
Keyword(s):  
Tumour System

scholarly journals A discoordinate increase in the cellular amount of 3-hydroxy-3-methylglutaryl-CoA reductase results in the loss of rate-limiting control over cholesterogenesis in a tumour cell-free system

1989 ◽  
Vol 258 (2) ◽  
pp. 421-425 ◽  
Author(s):  
N I Azrolan ◽  
P S Coleman
Keyword(s):  
Cholesterol Biosynthesis ◽  
Normal Liver ◽  
Preliminary Evidence ◽  
Free System ◽  
Normal Rat ◽  
Morris Hepatoma ◽  
Coa Reductase ◽  
Rate Limiting ◽  
Hydroxymethylglutaryl Coa Reductase ◽  
Tumour System

Cholesterol biosynthesis was characterized in cell-free post-mitochondrial supernatant systems prepared from both normal rat liver and Morris hepatoma 3924A. The rate of cholesterol synthesis per cell was 9-fold greater in the tumour system than in that from normal liver, and the tumour systems showed the loss of rate-limiting control at the hydroxymethylglutaryl-CoA reductase (HMGR)-catalysed step. The apparent absence of rate-limiting control over cell-free tumour cholesterogenesis was traced primarily to a discoordinate and dramatic increase in the amount of HMGR in the tumour relative to the liver system. Preliminary evidence for an altered control of the post-lanosterol portion of the pathway was also obtained with the tumour system.

Thermal enhancement of low dose rate irradiation in a murine tumour system

1989 ◽  
Vol 5 (4) ◽  
pp. 509-523 ◽  
Author(s):  
Ellen L. Jones ◽  
Bernard E. Lyons ◽  
Evan B. Douple ◽  
Bradley J. Dain
Keyword(s):  
Dose Rate ◽  
Low Dose ◽  
Murine Tumour ◽  
Low Dose Rate ◽  
Thermal Enhancement ◽  
Tumour System

scholarly journals In SilicoModelling of Treatment-Induced Tumour Cell Kill: Developments and Advances

2012 ◽  
Vol 2012 ◽  
pp. 1-16 ◽  
Author(s):  
Loredana G. Marcu ◽  
Wendy M. Harriss-Phillips
Keyword(s):  
Tumour Cell ◽  
Health Professionals ◽  
Treatment Modality ◽  
Tumour Growth ◽  
Comprehensive Overview ◽  
Fractionated Radiotherapy ◽  
The Past ◽  
Cell Kill ◽  
Relevant Field ◽  
Tumour System

Mathematical and stochastic computer (in silico) models of tumour growth and treatment response of the past and current eras are presented, outlining the aims of the models, model methodology, the key parameters used to describe the tumour system, and treatment modality applied, as well as reported outcomes from simulations. Fractionated radiotherapy, chemotherapy, and combined therapies are reviewed, providing a comprehensive overview of the modelling literature for current modellers and radiobiologists to ignite the interest of other computational scientists and health professionals of the ever evolving and clinically relevant field of tumour modelling.

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