scholarly journals Characteristics of Borderline Personality Disorder in a Community Sample: Comorbidity, Treatment Utilization, and General Functioning

2014 ◽  
Vol 28 (5) ◽  
pp. 734-750 ◽  
Author(s):  
Rachel L. Tomko ◽  
Timothy J. Trull ◽  
Phillip K. Wood ◽  
Kenneth J. Sher
Author(s):  
Soheil Zahediabghari ◽  
Philippe Boursiquot ◽  
Paul Links

Borderline Personality Disorder (BPD) significantly impairs functioning. Fortunately, effective treatments are available for borderline symptoms but their effect on functioning should be assessed. The objective of this meta-analysis is to assess the effect of specifically-designed versus non-specifically designed psychotherapies on function in adult patients with BPD. The reference list of Cristea et al. 2017 was used to identify the randomized controlled trials (RCTs) assessing the BPD-specifically-designed psychotherapy versus non-specific psychotherapies in adult BPD patients. Among those, RCTs assessing post-treatment functioning using the Global Assessment of Functioning, Social Adjustment Scale–Self-Report and Inventory of Interpersonal Problems were included. Ten trials (880 participants) were included. Summary effect size was calculated using the measured Hedge’s g. The results indicate the BPD patients in the intervention group had a significantly higher (g = 0.41; 95% CI, 0.09–0.73) level of psychosocial functioning after receiving the specifically-designed psychotherapies in comparison with BPD patients in control groups after receiving non-specific psychotherapies. Specifically-designed psychotherapies can improve psychosocial functioning although improvement in measurement of function (i.e., more objective and universal tools) and improvement in psychotherapies (i.e., more focused on general functioning) will be helpful.


2010 ◽  
Vol 16 (3) ◽  
pp. 155-163 ◽  
Author(s):  
Marianne Goodman ◽  
Uday Patil ◽  
Lauren Steffel ◽  
Jennifer Avedon ◽  
Scott Sasso ◽  
...  

2019 ◽  
Author(s):  
Taylor Barber ◽  
Whitney R. Ringwald ◽  
Aidan G.C. Wright ◽  
Stephen N. Manuck

There is growing interest in relationships between borderline personality disorder (BPD) pathology and physical health outcomes. Diagnostic BPD and BPD-related traits, for instance, have been shown to associate with self-reported cardiovascular disease and various cardiometabolic risk factors. However, potential confounding of these associations by comorbid depression, which itself contributes to risk for heart disease, remains unresolved, and prior research is limited by nearly uniform reliance on self-reported health status. In the present study, we examine the association of BPD traits and contemporaneously assessed depressive mood with instrumented measures of cardiometabolic risk in a midlife community sample (N = 1,295). BPD pathology was measured using dimensional, multi-informant trait measures; depressive symptomology was self-reported; and cardiometabolic risk was indexed via multiple indicators of insulin resistance, adiposity, dyslipidemia, and blood pressure. Structural equation modeling was used to estimate the effects of BPD traits and depressive symptoms on aggregated cardiometabolic risk, adjusting for their shared variance. Results showed both BPD pathology features and depressive symptomatology related to extent of cardiometabolic risk; when examined simultaneously, only BPD associated independently with risk indicators. In further supporting a link between BPD pathology and cardiovascular disease risk, these findings warrant future work to elucidate intervening behavioral and biological mechanisms.


2019 ◽  
Vol 10 (1) ◽  
pp. 204380871881887 ◽  
Author(s):  
Kibby McMahon ◽  
Kwanguk Kim ◽  
Caitlin M. Fang ◽  
Andrada D. Neacsiu ◽  
M. Zachary Rosenthal

Previous studies have demonstrated abnormalities in emotion recognition within individuals diagnosed with borderline personality disorder (BPD). However, it is yet unknown how much these abnormalities can be attributed to emotional states or affect. Therefore, the current study aimed to investigate the independent effects of BPD, positive affect, and negative affect on emotion recognition sensitivity. We recruited a mixed, transdiagnostic community sample of 118 adults diagnosed with either a personality disorder, only an affective disorder, or without psychopathology. Participants completed self-report assessments of positive and negative affect and two behavioral assessments of emotion recognition sensitivity. We found that both positive and negative affect predict lower overall emotion recognition sensitivity in both tasks, beyond the effect of BPD. We did not find a significant, independent effect of the diagnosis of BPD. Additionally, we found that the diagnosis of BPD moderated the relationship between negative affect and emotion recognition sensitivity within one task. Findings from the present study suggest that sensitivity to other people’s emotional expressions may be influenced by affect beyond the effect of the BPD diagnosis. The implications for future research efforts on emotion recognition and BPD are discussed.


2020 ◽  
Vol 10 (12) ◽  
pp. 180
Author(s):  
Victor Suarez ◽  
Guillem Feixas

Borderline personality disorder (BPD) represents a severe mental condition that is usually characterized by distressing identity disturbances. Although most prevailing explanatory models and psychotherapy approaches consider and intervene on self-concept, they seem not to recognize or explore idiosyncratic cognitive conflicts that patients may experience. These conflicts, which have been conceptualized as “implicative dilemmas” and “dilemmatic constructs” by personal construct theorists, could be considered as key elements of the explanatory model for BPD to provide a better understanding of this disorder and possibly enhance the effectiveness of contemporary psychotherapeutic approaches. The current study (Identifier: NCT04498104) aims to examine the characteristics of the interpersonal cognitive system of a group of patients diagnosed with BPD, using the repertory grid technique, and to compare them with those of a community sample. We will test if BPD participants are more affected by cognitive conflicts than controls. Additionally, we will gauge the association between cognitive conflicts and symptom severity as well as their predictive capacity of treatment outcome. The obtained results will be a necessary step to determine if cognitive conflicts have a substantial role on the explanation of BPD. It could also help to consider the development of a conflict resolution intervention module for this disorder.


2009 ◽  
Vol 21 (4) ◽  
pp. 1335-1353 ◽  
Author(s):  
Marina A. Bornovalova ◽  
Brian M. Hicks ◽  
William G. Iacono ◽  
Matt McGue

AbstractAlthough personality disorders are best understood in the context of lifetime development, there is a paucity of work examining their longitudinal trajectory. An understanding of the expected course and the genetic and environmental contributions to these disorders is necessary for a detailed understanding of risk processes that lead to their manifestation. The current study examined the longitudinal course and heritability of borderline personality disorder (BPD) over a period of 10 years starting in adolescence (age 14) and ending in adulthood (age 24). In doing so, we built on existing research by using a large community sample of adolescent female twins, a sensitive dimensional measure of BPD traits, an extended follow-up period, and a longitudinal twin design that allowed us to investigate the heritability of BPD traits at four discrete ages spanning midadolescence to early adulthood. Results indicated that mean-level BPD traits significantly decline from adolescence to adulthood, but rank order stability remained high. BPD traits were moderately heritable at all ages, with a slight trend for increased heritability from age 14 to age 24. A genetically informed latent growth curve model indicated that both the stability and change of BPD traits are highly influenced by genetic factors and modestly by nonshared environmental factors. Our results indicate that as is the case for other personality dimensions, trait BPD declines as individuals mature from adolescence to adulthood, and that this process is influenced in part by the same genetic factors that influence BPD trait stability.


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