Analysis of Factors Related to Recurrence of Colorectal Adenomatous Polyps After Endoscopic Treatment

2021 ◽  
Vol 7 (5) ◽  
pp. 2924-2928
Author(s):  
Hong Xiao ◽  
Xingguo Fang ◽  
Hai Zhang

Objective: To study and analyze the related factors of recurrence after endoscopic treatment of colorectal adenomatous polyps. Methods: From March 2017 to March 2018, a total of 600 colorectal adenomatous polyps treated with endoscopy in our hospital were selected as the study objects. The clinical data of all patients were analyzed retrospectively, including gender, age, occupation, income, drinking history, smoking history, whether they were infected with Helicobacter pylori, polyp location, polyp number, adenoma type, adenoma base, gland Tumor diameter, atypical hyperplasia, history of diabetes mellitus, history of hypertension, dyslipidemia (cholesteremia, low-density lipoprotein, hypertriglyceridemia), treatment methods, the relationship between the above risk factors and recurrence were analyzed, and the relevant factors of recurrence of colorectal adenomatous polyps were screened. Result: Age, occupation, income, drinking, smoking, polyp type, polyp diameter, Helicobacter pylori infection, treatment, diabetes, hypertension, adenoma type, adenoma base, atypical hyperplasia and dyslipidemia were all related factors (P < 0.05). Conclusion: Patients with colorectal adenomatous polyps undergo recurrence after endoscopic treatment with age, occupation, income, alcohol consumption, smoking, polyp type, Helicobacter pylori infection, treatment, diabetes, hypertension, adenoma type, adenoma base, A typical hyperplasia and dyslipidemia are associated. The more the above factors occur, the greater the chance of recurrence. The corresponding preventive measures should be taken for the above factors.

2001 ◽  
Vol 120 (5) ◽  
pp. A128-A128 ◽  
Author(s):  
H MALATY ◽  
D GRAHAM ◽  
A ELKASABANY ◽  
S REDDY ◽  
S SRINIVASAN ◽  
...  

2002 ◽  
Vol 18 (1) ◽  
pp. 26-33 ◽  
Author(s):  
Lila Belhoussine-Idrissi ◽  
Edgar C. Boedeker

2012 ◽  
Vol 55 (2) ◽  
pp. 209-216 ◽  
Author(s):  
Ximena Duque ◽  
Jenny Vilchis ◽  
Robertino Mera ◽  
Belem Trejo-Valdivia ◽  
Karen J. Goodman ◽  
...  

2019 ◽  
Vol 8 (4) ◽  
pp. 293-298
Author(s):  
Naoko Tsuji ◽  
Yasuko Umehara ◽  
Mamoru Takenaka ◽  
Yasunori Minami ◽  
Tomohiro Watanabe ◽  
...  

Abstract Background There have been few studies in the English literature regarding verrucous gastritis (VG). The present study investigated the clinical and endoscopic features of verrucous antral gastritis, especially focusing on Helicobacter pylori infection, nutrition, and gastric atrophy. Methods We performed a retrospective study of patients who underwent routine endoscopy with indigo carmine chromoendoscopy and a comparative study was conducted between VG-positive and VG-negative groups. VG was subdivided into classical and numerous types based on the number and distribution of verrucous lesions. Demographic, clinical, and endoscopic data including body mass index (BMI), serum albumin and cholesterol, gastric atrophy, reflux oesophagitis, Barrett’s oesophagus, and H. pylori status were collected. Univariate and multivariable analyses were performed to identify factors associated with VG. Results We analysed the data of 621 patients undergoing routine endoscopy and found that VG (n = 352) was significantly associated with increased BMI (1.12 [1.05–1.18], P &lt; 0.01), reflux esophagitis (1.96 [1.10–3.28], P &lt; 0.01), and H. pylori negativity with or without a history of eradication (9.94 [6.00–16.47] and 6.12 [3.51–10.68], P &lt; 0.001, respectively). Numerous-type (n = 163) VG was associated with both closed- and open-type gastric atrophy (9.9 [4.04–21.37] and 8.10 [3.41–19.24], P &lt; 0.001, respectively). There were no statistical differences between groups regarding age, sex, total cholesterol, albumin, and bile-colored gastric juice. Conclusions Verrucous antral gastritis was related to increased BMI, reflux esophagitis, and H. pylori negativity. Numerous-type verrucous lesions were associated with gastric atrophy. These indicate that VG may be a physiological phenomenon due to high gastric acidity, mechanical overload, and vulnerability of background mucosa.


Helicobacter ◽  
2009 ◽  
Vol 14 (5) ◽  
pp. 478-486 ◽  
Author(s):  
Krystyna Stec-Michalska ◽  
Lukasz Peczek ◽  
Blazej Michalski ◽  
Maria Wisniewska-Jarosinska ◽  
Agnieszka Krakowiak ◽  
...  

Sign in / Sign up

Export Citation Format

Share Document