Variation in Collar Spine Number of Echinostoma revolutum from the Muskrat

1969 ◽  
Vol 55 (2) ◽  
pp. 380
Author(s):  
George P. Doerksen
Keyword(s):  
Collar Spine ◽  
Echinostoma Revolutum ◽  
Spine Number

Single- and five-worm infections of Echinostoma revolutum (trematoda) in the golden hamster

1988 ◽  
Vol 18 (2) ◽  
pp. 179-181 ◽  
Author(s):  
Bernard Fried ◽  
Jane E. Huffman ◽  
Jose Franco
Keyword(s):  
Golden Hamster ◽  
Echinostoma Revolutum

The adaptive significance of dorsal spine variation in the fourspine stickleback, Apeltes quadracus, I. Geographic variation in spine number

1984 ◽  
Vol 62 (7) ◽  
pp. 1329-1339 ◽  
Author(s):  
D. M. Blouw ◽  
D. W. Hagen
Keyword(s):  
Genetic Differentiation ◽  
Geographic Variation ◽  
Adaptive Significance ◽  
Maritime Provinces ◽  
Selective Agents ◽  
Apeltes Quadracus ◽  
Remarkable Result ◽  
Fourspine Stickleback ◽  
Very High Frequencies ◽  
Spine Number

The goal of our research is to investigate the adaptive significance of a polymorphism for the number of dorsal spines in Apeltes quadracus, the fourspine stickleback. One approach we take is to search for correlations between phenotypes and environments. To this end we collected Apeltes and scored environments at 570 sites in the Maritime Provinces of Canada. In this paper we describe geographic variation in spine number and evaluate how reliably it reflects genetic differentiation among sites. Morph frequencies are highly differentiated geographically. We describe four kinds of variation: relatively constant frequencies, gentle clines, steep clines, and remarkably abrupt changes (called "intrusions") where frequencies at some sites differ greatly from those at a larger number of surrounding sites. Most of the variation among sites is due to differences in the frequencies of the four- and five-spined morphs. However, a remarkable result is that the three-spined morph, which is rare or absent elsewhere in the range, reaches very high frequencies in Bras D'Or Lake. Our evidence suggests this variation among sites reflects substantial genetic differentiation. The differentiation is favorable for detecting selective agents, if indeed selection is responsible.

Thin-Layer Chromatographic Analysis of Phospholipids in Echinostoma revolutum (Trematoda) Adults

1979 ◽  
Vol 65 (2) ◽  
pp. 243 ◽  
Author(s):  
Bernard Fried ◽  
Irwin L. Shapiro
Keyword(s):  
Thin Layer ◽  
Chromatographic Analysis ◽  
Echinostoma Revolutum ◽  
Thin Layer Chromatographic Analysis

Thin layer chromatographic analyses of amino acids in Echinostoma revolutum (Trematoda) adults

1977 ◽  
Vol 7 (6) ◽  
pp. 497-499 ◽  
Author(s):  
Robert S. Bailey ◽  
Bernard Fried
Keyword(s):  
Amino Acids ◽  
Thin Layer ◽  
Echinostoma Revolutum

The effect of DDT and rearing temperature on pecten spine number in larvae of the mosquito Aedes aegypti L.

Genetica ◽  
1990 ◽  
Vol 46 (1) ◽  
pp. 45-48 ◽  
Author(s):  
R. J. Wood
Keyword(s):  
Aedes Aegypti ◽  
Rearing Temperature ◽  
Spine Number

Experiments on Regeneration in the Trematode, Echinostoma revolutum

1937 ◽  
Vol 23 (4) ◽  
pp. 423 ◽  
Author(s):  
Paul Beaver
Keyword(s):  
Echinostoma Revolutum

Histochemical Glycogen Studies on Echinostoma revolutum

1968 ◽  
Vol 54 (5) ◽  
pp. 942 ◽  
Author(s):  
Bernard Fried ◽  
Marshall D. Kramer
Keyword(s):  
Echinostoma Revolutum

scholarly journals BDNF signaling during the lifetime of dendritic spines

2020 ◽  
Vol 382 (1) ◽  
pp. 185-199 ◽  
Author(s):  
Marta Zagrebelsky ◽  
Charlotte Tacke ◽  
Martin Korte
Keyword(s):  
Dendritic Spines ◽  
Dendritic Spine ◽  
Neurological Disorders ◽  
Current Knowledge ◽  
Conclusive Evidence ◽  
Excitatory Synapses ◽  
Neuronal Homeostasis ◽  
Bdnf Signaling ◽  
Spine Number

Abstract Dendritic spines are tiny membrane specialization forming the postsynaptic part of most excitatory synapses. They have been suggested to play a crucial role in regulating synaptic transmission during development and in adult learning processes. Changes in their number, size, and shape are correlated with processes of structural synaptic plasticity and learning and memory and also with neurodegenerative diseases, when spines are lost. Thus, their alterations can correlate with neuronal homeostasis, but also with dysfunction in several neurological disorders characterized by cognitive impairment. Therefore, it is important to understand how different stages in the life of a dendritic spine, including formation, maturation, and plasticity, are strictly regulated. In this context, brain-derived neurotrophic factor (BDNF), belonging to the NGF-neurotrophin family, is among the most intensively investigated molecule. This review would like to report the current knowledge regarding the role of BDNF in regulating dendritic spine number, structure, and plasticity concentrating especially on its signaling via its two often functionally antagonistic receptors, TrkB and p75NTR. In addition, we point out a series of open points in which, while the role of BDNF signaling is extremely likely conclusive, evidence is still missing.

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