scholarly journals Cardiac Arrhythmia Associated with Remdesivir in COVID-19

Author(s):  
Niraula Sristee ◽  
Oli Shital ◽  
Lee Janette
Keyword(s):  
2005 ◽  
Vol 250 (02) ◽  
Author(s):  
L Hutsol ◽  
M Hutsol ◽  
I Tsymbal
Keyword(s):  

Diabetes ◽  
2019 ◽  
Vol 68 (Supplement 1) ◽  
pp. 2207-PUB
Author(s):  
CARLA MUSSO ◽  
NOELIA S. SFORZA ◽  
YANINA JIMENA MOROSAN ALLO ◽  
ROMINA G. CLEMENTE ◽  
ADRIAN PAVESI ◽  
...  
Keyword(s):  

Medicine ◽  
2018 ◽  
Vol 97 (50) ◽  
pp. e13629
Author(s):  
Yu-Hong Man ◽  
Xiao-Li Meng ◽  
Ting-Min Yu ◽  
Gang Yao

2021 ◽  
Vol 77 (18) ◽  
pp. 308
Author(s):  
Chanavuth Kanitsoraphan ◽  
Chol Techorueangwiwat ◽  
Jakrin Kewcharoen ◽  
Pattara Rattanawong ◽  
Thomas Bunch ◽  
...  

Author(s):  
Hadeel AbdElraheem Altejani Badawi ◽  
Maysaa AbdAlgader Abdalrahman Megdar ◽  
Mohammed A. Zarrouq Yousif ◽  
Ebtisam Muawia MohammedKhair Mustafa ◽  
Najwan Othman Mohammed Abdalrheem

Author(s):  
Shanna Hamilton ◽  
Roland Veress ◽  
Andriy Belevych ◽  
Dmitry Terentyev

AbstractSudden cardiac death due to malignant ventricular arrhythmias remains the major cause of mortality in the postindustrial world. Defective intracellular Ca2+ homeostasis has been well established as a key contributing factor to the enhanced propensity for arrhythmia in acquired cardiac disease, such as heart failure or diabetic cardiomyopathy. More recent advances provide a strong basis to the emerging view that hereditary cardiac arrhythmia syndromes are accompanied by maladaptive remodeling of Ca2+ homeostasis which substantially increases arrhythmic risk. This brief review will focus on functional changes in elements of Ca2+ handling machinery in cardiomyocytes that occur secondary to genetic mutations associated with catecholaminergic polymorphic ventricular tachycardia, and long QT syndrome.


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