scholarly journals Neurotransmitters and nitric oxide are released in various brain areas according to ultradian and infradian rhythms

2020 ◽  
Vol 8 (2) ◽  
pp. 233-236
Author(s):  
Athineos Philippu
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Brain Regions ◽  
Ultradian Rhythms ◽  
Brain Areas ◽  
Nitric Oxide Release ◽  
Infradian Rhythms

The push-pull superfusion technique was used to investigate release of endogenous neurotransmitters and nitric oxide in various brain regions. The transmitters catecholamines, histamine, GABA, glutamate, as well and nitric oxide are released according to ultradian rhythms which generate fluctuations of vegetative functions such as blood pressure and arousal. More frequent infradian oscillations of neurotransmitter and nitric oxide release fates are implicated in mutual interneural control.

scholarly journals Neurotransmitters and nitric oxide are released in various brain areas according to ultradian and infradian rhythms

2020 ◽  
Vol 8 (2) ◽  
pp. 233-236
Author(s):  
Athineos Philippu
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Brain Regions ◽  
Ultradian Rhythms ◽  
Brain Areas ◽  
Nitric Oxide Release ◽  
Infradian Rhythms

The push-pull superfusion technique was used to investigate release of endogenous neurotransmitters and nitric oxide in various brain regions. The transmitters catecholamines, histamine, GABA, glutamate, as well and nitric oxide are released according to ultradian rhythms which generate fluctuations of vegetative functions such as blood pressure and arousal. More frequent infradian oscillations of neurotransmitter and nitric oxide release fates are implicated in mutual interneural control.

L-Arginine Infusion Reduces Blood Pressure in Preeclamptic Women Through Nitric Oxide Release

1999 ◽  
Vol 6 (4) ◽  
pp. 202-207 ◽  
Author(s):  
F. Facchinetti ◽  
M. Longo ◽  
F. Piccinini ◽  
I. Neri ◽  
A. Volpe
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Arginine Infusion ◽  
Nitric Oxide Release

scholarly journals ANTIHYPERTENSION STUDY OF ANREDERA CORDIFOLIA (TEN). V. STEENIS EXTRACT AND ITS FRACTIONS IN RATS THROUGH DEXAMETHASONE INDUCTION AND NITRIC OXIDE RELEASE.

2018 ◽  
Vol 11 (1) ◽  
pp. 278
Author(s):  
Afrillia Nuryanti Garmana ◽  
Elin Yulinah Sukandar ◽  
Irda Fidrianny
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Antihypertensive Effect ◽  
Ethanol Extract ◽  
Ethyl Acetate Fraction ◽  
Pressure System ◽  
Rat Serum ◽  
Nitric Oxide Release ◽  
Water Fraction ◽  
Hypertensive Rat

 Objective: The aims of this study were to prove the antihypertensive effect of Anredera cordifolia (Madeira vine) in dexamethasone-induced hypertensive rat and to determine the release of nitric oxide (NO).Methods: The rat’s blood pressure was measured by CODA® tail-cuff blood pressure system. A hypertensive rat model was developed on day 7 after administration of dexamethasone injection 0.5 mg/kg body weight (bw). NO levels were measured by spectrophotometry at a wavelength of 546 nm after reacting the serum sample with Griess reagent.Results: Ethanol extract of Madeira vine (EEMV), ethyl acetate fraction (EF), and water fraction (WF) could reduce systolic blood pressure at day 14 with a diastolic blood pressure (DBP) reduction of 26.8, 34.1, and 40.5 mmHg, respectively. DBP began to decrease from day 8 in the EEMV group with a DBP reduction of 24.1 mmHg. In the HF, EF, and WF groups, decreasing in DBP occurred on day 14 which were 22.0, 24.5, and 35.4 mmHg, respectively. NO level in rat serum was increased significantly at 90 min after administration of EEMV 100 mg/kg bw and WF 40.73 mg/kg bw. Increasing in NO levels due to EF with a dose of 1.66 mg/kg bw was not significantly different to control group.Conclusion: Ethanol extract of A. cordifolia had the antihypertensive effect in dexamethasone-induced hypertensive rats, so does its WF and EF. The mechanism of ethanol extract of Madeira vine leaves and its WF most likely due to vasodilation effect through NO-pathway, whereas EF could have other mechanism(s) of action. 

scholarly journals ANTIHYPERTENSION STUDY OF ANREDERA CORDIFOLIA (TEN). V. STEENIS EXTRACT AND ITS FRACTIONS IN RATS THROUGH DEXAMETHASONE INDUCTION AND NITRIC OXIDE RELEASE.

2018 ◽  
Vol 11 (1) ◽  
pp. 278
Author(s):  
Afrillia Nuryanti Garmana ◽  
Elin Yulinah Sukandar ◽  
Irda Fidrianny
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Antihypertensive Effect ◽  
Ethanol Extract ◽  
Ethyl Acetate Fraction ◽  
Pressure System ◽  
Rat Serum ◽  
Nitric Oxide Release ◽  
Water Fraction ◽  
Hypertensive Rat

 Objective: The aims of this study were to prove the antihypertensive effect of Anredera cordifolia (Madeira vine) in dexamethasone-induced hypertensive rat and to determine the release of nitric oxide (NO).Methods: The rat’s blood pressure was measured by CODA® tail-cuff blood pressure system. A hypertensive rat model was developed on day 7 after administration of dexamethasone injection 0.5 mg/kg body weight (bw). NO levels were measured by spectrophotometry at a wavelength of 546 nm after reacting the serum sample with Griess reagent.Results: Ethanol extract of Madeira vine (EEMV), ethyl acetate fraction (EF), and water fraction (WF) could reduce systolic blood pressure at day 14 with a diastolic blood pressure (DBP) reduction of 26.8, 34.1, and 40.5 mmHg, respectively. DBP began to decrease from day 8 in the EEMV group with a DBP reduction of 24.1 mmHg. In the HF, EF, and WF groups, decreasing in DBP occurred on day 14 which were 22.0, 24.5, and 35.4 mmHg, respectively. NO level in rat serum was increased significantly at 90 min after administration of EEMV 100 mg/kg bw and WF 40.73 mg/kg bw. Increasing in NO levels due to EF with a dose of 1.66 mg/kg bw was not significantly different to control group.Conclusion: Ethanol extract of A. cordifolia had the antihypertensive effect in dexamethasone-induced hypertensive rats, so does its WF and EF. The mechanism of ethanol extract of Madeira vine leaves and its WF most likely due to vasodilation effect through NO-pathway, whereas EF could have other mechanism(s) of action. 

BLOOD PRESSURE REGULATION AND NITRIC OXIDE RELEASE BY S-NITROSO HUMAN SERUM ALBUMIN-STUDIES IN THE RABBIT.

Shock ◽  
1999 ◽  
Vol 12 (Supplement) ◽  
pp. 43
Author(s):  
H. Gasser ◽  
S. Hallström ◽  
C. Vogl ◽  
V. Brovkovych ◽  
T. Malinski;
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Human Serum Albumin ◽  
Serum Albumin ◽  
Human Serum ◽  
Blood Pressure Regulation ◽  
Pressure Regulation ◽  
Nitric Oxide Release

Role of Nitric Oxide in Buffering Short-Term Blood Pressure Fluctuations

Physiology ◽  
2000 ◽  
Vol 15 (5) ◽  
pp. 229-233 ◽  
Author(s):  
Harald M. Stauss ◽  
Pontus B. Persson
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Shear Stress ◽  
Pressure Fluctuations ◽  
Initial Increase ◽  
Short Term ◽  
Nitric Oxide Release ◽  
Arterial Blood ◽  
Endothelial Shear Stress

Blood pressure instability may promote cardiovascular morbidity. Recent data suggest a role of nitric oxide in stabilizing arterial blood pressure. A rise in blood pressure enhances endothelial shear stress and nitric oxide release. The resulting vasodilation antagonizes the initial increase in blood pressure. This system can respond within 2–10 seconds.

Dipeptidyl Peptidase-4 Inhibition With Saxagliptin Enhanced Nitric Oxide Release and Reduced Blood Pressure and sICAM-1 Levels in Hypertensive Rats

2012 ◽  
Vol 60 (5) ◽  
pp. 467-473 ◽  
Author(s):  
R. Preston Mason ◽  
Robert F. Jacob ◽  
Ruslan Kubant ◽  
Aleksander Ciszewski ◽  
J. Jose Corbalan ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Blood Pressure ◽  
Dipeptidyl Peptidase ◽  
Hypertensive Rats ◽  
Dipeptidyl Peptidase 4 ◽  
Nitric Oxide Release
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