Microliter Determinations of Total and Direct Bilirubin, Alkaline Phosphatase, Potassium, Sodium and Urea Nitrogen in Blood Plasma During the Early Neonatal Period

During an experiment on white outbread mail rats with different models of hepatorenal syndrome, evaluations of liver functional state biochemical findings in blood plasma were studied. The results of the study showed obvious hepatic disorders manifested by decrease in blood plasma albumin and urea content, transaminitis, changes of Ritis coefficient and increased alkaline phosphatase. It was determined that the disorders were more pronounced in cases of hepatorenal syndrome modelling by means of carbon tetrachloride administering.

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Utilization of the Bone/Liver Alkaline Phosphatase Activity Ratio in Blood Plasma as an Indicator of Ascorbate Deficiency in Salmonid Fish

Experimental Biology and Medicine ◽

10.3181/00379727-212-43990 ◽

1996 ◽

Vol 212 (1) ◽

pp. 44-51 ◽

Cited By ~ 7

Author(s):

M. Matusiewicz ◽

K. Dabrowski

Keyword(s):

Alkaline Phosphatase ◽

Blood Plasma ◽

Phosphatase Activity ◽

Alkaline Phosphatase Activity ◽

Activity Ratio ◽

Salmonid Fish ◽

In Blood Plasma

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A Case of Congenital Glucose Galactose Malabsorption with a New Mutation in the SLC5A1 Gene

Journal of Pediatric Genetics ◽

10.1055/s-0040-1719161 ◽

2020 ◽

Author(s):

Hasan Akduman ◽

Dilek Dilli ◽

Serdar Ceylaner

Keyword(s):

Mutation Analysis ◽

Autosomal Recessive ◽

Glucose Transporter ◽

Neonatal Period ◽

Autosomal Recessive Disorder ◽

Homozygous Mutation ◽

New Mutation ◽

Early Neonatal Period ◽

Sodium Dependent ◽

Hypernatremic Dehydration

AbstractCongenital glucose-galactose malabsorption (CGGM) is an autosomal recessive disorder originating from an abnormal transporter mechanism in the intestines. It was sourced from a mutation in the SLC5A1 gene, which encodes a sodium-dependent glucose transporter. Here we report a 2-day-old girl with CGGM who presented with severe hypernatremic dehydration due to diarrhea beginning in the first hours of life. Mutation analysis revealed a novel homozygous mutation NM_000343.3 c.127G > A (p.Gly43Arg) in the SLC5A1 gene. Since CGGM can cause fatal diarrhea in the early neonatal period, timely diagnosis of the disease seems to be essential.

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The Balance of Pro-Oxidants - Antixidants No Change in Blood Plasma Following a Carbohydrate Meal

Advances in Obesity Weight Management & Control ◽

10.15406/aowmc.2017.06.00162 ◽

2017 ◽

Vol 6 (4) ◽

Author(s):

Karterolioti Chrysavgi

Keyword(s):

Blood Plasma ◽

Carbohydrate Meal ◽

In Blood Plasma

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FEATURES OF CARDIORESPIRATORY ADAPTATION OF LATER PRETERM INFANTS IN THE EARLY NEONATAL PERIOD

World of Medicine and Biology ◽

10.26724/2079-8334-2018-4-66-100-103 ◽

2018 ◽

Vol 14 (66) ◽

pp. 100

Author(s):

G. A. Solovyova ◽

V. I. Pokhilko ◽

S. N. Tsvirenko ◽

N. I. Gasyuk ◽

Yu. Yu. Klimchuk

Keyword(s):

Preterm Infants ◽

Neonatal Period ◽

Early Neonatal Period

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State of hormonal regulation in experimental animals exposed to noise and vibration

Russian Journal of Occupational Health and Industrial Ecology ◽

10.31089/1026-9428-2019-59-9-680-681 ◽

2020 ◽

pp. 680-681

Author(s):

A. V. Lizarev ◽

V. A. Pankov

Keyword(s):

Blood Plasma ◽

Hormonal Regulation ◽

Adrenocorticotropic Hormone ◽

Vibration Exposure ◽

Noise And Vibration ◽

Experimental Animals ◽

In Blood Plasma

When exposed to noise and vibration in experimental animals there was a decrease in the content of threeiodinethyronine, thyroxin and adrenocorticotropic hormone in blood plasma after 15 and 30 days of experience. An increase in loads led to an increase in the level of threeiodinethyronine and thyroxin under vibration exposure and was normalized with noise. The content of adrenocorticotropic hormone leveled in both cases.

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