Congenital neuroblastoma with symptoms of epidural compression at birth

2016 ◽  
Vol 33 (2) ◽  
pp. 94-101 ◽  
Author(s):  
A. R. Gigliotti ◽  
M. A. De Ioris ◽  
E. De Grandis ◽  
M. Podda ◽  
M. Cellini ◽  
...  
Author(s):  
O. B. Malevich ◽  
T. V. Shamanskaya ◽  
D. Yu. Kachanov ◽  
S. P. Homyakova ◽  
G. M. Muftakhova ◽  
...  

Neurosurgery ◽  
1988 ◽  
Vol 23 (5) ◽  
pp. 662-665 ◽  
Author(s):  
Alan Hirschfeld ◽  
William Beutler ◽  
Juliet Seigle ◽  
Herbert Manz

Abstract We present two cases in which spinal epidural compression was caused by the expansion of bony elements into the spinal canal as a result of osteoblastic metastases. The precise nature of the compression was appreciated only on computed tomography. One patient had immediate and sustained neurological improvement after laminectomy. The other benefited temporarily, but widespread involvement of his spine ultimately led to paraplegia despite two more decompressive procedures. We think that bony expansion of the spine secondary to osteoblastic metastasis is not reversible with radiation therapy alone and is, therefore, an absolute indication for surgical decompression.


1972 ◽  
Vol 36 (1) ◽  
pp. 43-49 ◽  
Author(s):  
Arthur M. Gerber ◽  
Robert A. Moody

✓ Experiments were carried out on rhesus monkeys to determine what physiological parameters were most closely correlated with death due to craniocerebral missile injuries. Observations of intracranial pressure, blood pressure, carotid flow, blood gases, respiratory rate, depth and volume, and electroencephalograms were made. These parameters were compared in survivors and nonsurvivors as were the pathological injuries. The most important single parameter that correlated with death was the drop in carotid flow. As this same correlation has been observed in epidural compression experiments in the monkey, there is a strong suspicion that reduced blood flow to the brain as measured by carotid flow is a common factor in craniocerebral missile injuries and epidural compression injuries.


2018 ◽  
Vol 9 (1) ◽  
pp. 209 ◽  
Author(s):  
ZacharyA Smith ◽  
Maia Winkel ◽  
CortD Lawton ◽  
OlabisiR Sanusi ◽  
CraigM Horbinski ◽  
...  

2017 ◽  
Vol 77 (1) ◽  
pp. 1-17
Author(s):  
Tzu‑Yin Yeh ◽  
Guo‑Fang Tseng ◽  
Chi‑Yu Tseng ◽  
Yung‑Hsin Huang ◽  
Pei‑Hsin Liu

1987 ◽  
pp. 229-239
Author(s):  
K.-D. Bachmann ◽  
H. Chr. Dominick

2009 ◽  
Vol 15 (11) ◽  
Author(s):  
Ignatius Edwin D'souza ◽  
Ramesh Chandra Patidar ◽  
Ravichandran Mani ◽  
Mahmoud Shamseldeen ◽  
Joyce Jose

2013 ◽  
Vol 35 (4) ◽  
pp. 260-266 ◽  
Author(s):  
Maria Capasso ◽  
Giuseppe Cinalli ◽  
Anna Nastro ◽  
Maria Giuliano ◽  
Maria E. Errico ◽  
...  
Keyword(s):  

1978 ◽  
Vol 24 (11) ◽  
pp. 1899-1902 ◽  
Author(s):  
F A Muskiet ◽  
H J Jeuring ◽  
G T Nagel ◽  
H W de Bruyn ◽  
B G Wolthers

Abstract We describe the determination of 3-methoxy-4-hydroxy-phenylacetic acid, 3-methoxy-4-hydroxyphenylamandelic acid, and 3-methoxy-4-hydroxyphenylethylene glycol in amniotic fluid by means of mass fragmentography, with use of deuterated internal standards. The results expressed in terms of absolute concentration and creatinine concentration, are given as a function of gestational age. In the 15th to 17th week, concentrations in amniotic fluid are a reflection of those in the mother's serum, whereas in the 32nd to 40th week, these concentrations, expressed in terms of creatinine, are similar to those found in the urine of newborns. We discuss the possible usefulness of the determination of catecholamine metabolites in amniotic fluid in the diagnosis of congenital neuroblastoma, maternal pheochromocytoma, and underdevelopment.


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