scholarly journals Elevated pCO2 Affects Feeding Behavior and Acute Physiological Response of the Brown Crab Cancer pagurus

2018 ◽  
Vol 9 ◽  
Author(s):  
Youji Wang ◽  
Menghong Hu ◽  
Fangli Wu ◽  
Daniela Storch ◽  
Hans-Otto Pörtner
1993 ◽  
Vol 9 (3) ◽  
pp. 369-382 ◽  
Author(s):  
S. McChesney Gillette ◽  
C. A. Dawson ◽  
R. J. Scott ◽  
D. A. Rickaby ◽  
B. E. Powers ◽  
...  

2018 ◽  
Vol 2018 ◽  
pp. 1-11 ◽  
Author(s):  
Scott McGuire ◽  
Elizabeth Jane Horton ◽  
Derek Renshaw ◽  
Alofonso Jimenez ◽  
Nithya Krishnan ◽  
...  

Acute haemodynamic instability is a natural consequence of disordered cardiovascular physiology during haemodialysis (HD). Prevalence of intradialytic hypotension (IDH) can be as high as 20–30%, contributing to subclinical, transient myocardial ischemia. In the long term, this results in progressive, maladaptive cardiac remodeling and impairment of left ventricular function. This is thought to be a major contributor to increased cardiovascular mortality in end stage renal disease (ESRD). Medical strategies to acutely attenuate haemodynamic instability during HD are suboptimal. Whilst a programme of intradialytic exercise training appears to facilitate numerous chronic adaptations, little is known of the acute physiological response to this type of exercise. In particular, the potential for intradialytic exercise to acutely stabilise cardiovascular hemodynamics, thus preventing IDH and myocardial ischemia, has not been explored. This narrative review aims to summarise the characteristics and causes of acute haemodynamic instability during HD, with an overview of current medical therapies to treat IDH. Moreover, we discuss the acute physiological response to intradialytic exercise with a view to determining the potential for this nonmedical intervention to stabilise cardiovascular haemodynamics during HD, improve coronary perfusion, and reduce cardiovascular morbidity and mortality in ESRD.


2004 ◽  
Vol 18 (15) ◽  
pp. 1934-1936 ◽  
Author(s):  
G. Mandolesi ◽  
F. Madeddu ◽  
Y. Bozzi ◽  
L. Maffei ◽  
G. M. Ratto

1990 ◽  
Vol 25 (6) ◽  
pp. 587-593 ◽  
Author(s):  
Timothy M. Crombleholme ◽  
Michael R. Harrison ◽  
Michael T. Longaker ◽  
Jacob C. Langer ◽  
N. Scott Adzick ◽  
...  

2014 ◽  
Vol 9 (4) ◽  
pp. 661-666 ◽  
Author(s):  
Dietmar Wallner ◽  
Helmut Simi ◽  
Gerhard Tschakert ◽  
Peter Hofmann

Purpose:To analyze the acute physiological response to aerobic short-interval training (AESIT) at various high-intensity running speeds. A minor anaerobic glycolytic energy supply was aimed to mimic the characteristics of slow continuous runs.Methods:Eight trained male runners (maximal oxygen uptake [VO2max] 55.5 ± 3.3 mL · kg−1 · min−1) performed an incremental treadmill exercise test (increments: 0.75 km · h−1 · min−1). Two lactate turn points (LTP1, LTP2) were determined. Subsequently, 3 randomly assigned AESIT sessions with high-intensity running-speed intervals were performed at speeds close to the speed (v) at VO2max (vVO2max) to create mean intensities of 50%, 55%, and 60% of vLTP1. AESIT sessions lasted 30 min and consisted of 10-s work phases, alternated by 20-s passive recovery phases.Results:To produce mean velocities of 50%, 55%, and 60% of vLTP1, running speeds were calculated as 18.6 ± 0.7 km/h (93.4% vVO2max), 20.2 ± 0.6 km/h (101.9% vVO2max), and 22.3 ± 0.7 km/h (111.0% vVO2max), which gave a mean blood lactate concentration (La) of 1.09 ± 0.31 mmol/L, 1.57 ± 0.52 mmol/L, and 2.09 ± 0.99 mmol/L, respectively. La at 50% of vLTP1 was not significantly different from La at vLTP1 (P = .8894). Mean VO2 was found at 54.0%, 58.5%, and 64.0% of VO2max, while at the end of the sessions VO2 rose to 71.1%, 80.4%, and 85.6% of VO2max, respectively.Conclusion:The results showed that AESIT with 10-s work phases alternating with 20 s of passive rest and a running speed close to vVO2max gave a systemic aerobic metabolic profile similar to slow continuous runs.


1992 ◽  
Vol 73 (3) ◽  
pp. 1202-1206 ◽  
Author(s):  
R. C. Wetzel ◽  
C. J. Herold ◽  
E. A. Zerhouni ◽  
J. L. Robotham

Recent advances in computed tomographic imaging provide a unique method to serially and directly visualize acute physiological response in the lung. To directly investigate the airway response to hypoxia, high-resolution computed tomographic scans of the lungs of eight intact anesthetized minipigs were serially repeated before, during, and after ventilation with a hypoxic gas mixture (inspired fraction of O2 congruent to 0.07). This approach demonstrated an acute reversible 56 +/- 8% (SE) dilation in large airways (greater than 2 mm diam) and a 90 +/- 15% dilation in small airways (less than 1.99 mm diam) with decreased inspired O2 tension. Of the airways studied, 70 of 76 dilated. Hypoxic bronchodilation may interact with hypoxic pulmonary vasoconstriction in the fundamental physiological process of ventilation-perfusion matching in the lung.


2021 ◽  
Vol Publish Ahead of Print ◽  
Author(s):  
Gwan-Jin Park ◽  
So Yeon Kong ◽  
Dong-Min Shin ◽  
Dae In Lee ◽  
Beom-Seok Ku ◽  
...  

Author(s):  
Scott McGuire ◽  
Elizabeth Jane Horton ◽  
Derek Renshaw ◽  
Klaris Chan ◽  
Nithya Krishnan ◽  
...  

Background: Hemodialysis is associated with numerous symptoms and side effects which, in part, maybe due to sub-clinical hypoxia. However, acute cardio-pulmonary and metabolic physiology during hemodialysis is not well defined. Intra-dialytic and inter-dialytic exercise appear to be beneficial and may alleviate these side effects. To better understand these potential benefits, the acute physiological response to exercise should be evaluated. The aim of this study was to compare and characterise the acute physiological response during hemodialysis, intra-dialytic and inter-dialytic exercise. Methods: Cardiopulmonary physiology was evaluated during three conditions; 1) hemodialysis without exercise (HD), 2) intra-dialytic exercise (IDEx), and 3) inter-dialytic exercise (Ex). Exercise consisted of 30 minutes constant load cycle ergometry at 90% VO2AT. Central hemodynamics (via non-invasive bio-reactance) and ventilatory gas exchange were recorded during each experimental condition. Results: Twenty participants (59 ± 12 yrs, 16/20 male) completed the protocol. Cardiac output (Δ = -0.7 L/min), O2 uptake (Δ = -1.4 ml.kg-1.min-1) and arterial-venous O2 difference (Δ = -2.0 ml/O2/100ml) decreased significantly during HD. Respiratory exchange ratio exceeded 1.0 throughout HD and IDEx. Minute ventilation was lower (p = 0.001) during IDEx (16.5 ± 1.1 L/min) compared to Ex (19.8 ± 1.0 L/min). Arterial-venous O2 difference was partially restored further to IDEx (4.6 ± 1.9 ml/O2/100ml) compared to HD (3.5 ± 1.2 ml/O2/100ml). Conclusion: Hemodialysis altered cardiopulmonary and metabolic physiology, suggestive of hypoxia. This dysregulated physiology contributed to a greater physiological demand during intra-dialytic compared to inter-dialytic exercise. Despite this, intra-dialytic exercise partly normalised physiology during treatment.


Author(s):  
Christopher R.J. Fennell ◽  
James G. Hopker

Purpose: There has been paucity in research investigating the individualization of recovery interval duration during cycling-based high-intensity interval training (HIIT). The main aim of the study was to investigate whether individualizing the duration of the recovery interval based upon the resolution of muscle oxygen consumption would improve the performance during work intervals and the acute physiological response of the HIIT session, when compared with a standardized (2:1 work recovery ratio) approach. Methods: A total of 16 well-trained cyclists (maximal oxygen consumption: 60 [7] mL·kg−1·min−1) completed 6 laboratory visits: (Visit 1) incremental exercise test, (Visit 2) determination of the individualized (IND) recovery duration, using the individuals’ muscle oxygen consumption recovery duration to baseline from a 4- and 8-minute work interval, (Visits 3–6) participants completed a 6 × 4- and a 3 × 8-minute HIIT session twice, using the IND and standardized recovery intervals. Results: Recovery duration had no effect on the percentage of the work intervals spent at >90% and >95% of maximal oxygen consumption, maximal minute power output, and maximal heart rate, during the 6 × 4- and 3 × 8-minute HIIT sessions. Recovery duration had no effect on mean work interval power output, heart rate, oxygen consumption, blood lactate, and rating of perceived exertion. There were no differences in reported session RPE between recovery durations for the 6 × 4- and 3 × 8-minute HIIT sessions. Conclusion: Individualizing HIIT recovery duration based upon the resolution of muscle oxygen consumption to baseline levels does not improve the performance of the work intervals or the acute physiological response of the HIIT session, when compared with standardized recovery duration.


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