scholarly journals Does the origin of ablated premature ventricular contractions determine the level of left ventricle remodeling?

2020 ◽  
Author(s):  
Agnieszka Wojdyła-Hordyńska ◽  
Patrycja Pruszkowska-Skrzep ◽  
Philipp Sommer ◽  
Gerhard Hindricks ◽  
Piotr Feusette ◽  
...  
Global Heart ◽  
2018 ◽  
Vol 13 (4) ◽  
pp. 450-451
Author(s):  
A. Tomasik ◽  
G. Banasik ◽  
D. Kawecki ◽  
C. Wojciechowska ◽  
W. Jacheć ◽  
...  

Author(s):  
Marija Zdravkovic ◽  
Branislav Milovanovic ◽  
Mirjana Krotin ◽  
Sinisa Dimnkovic ◽  
Dragan Lovic ◽  
...  

2020 ◽  
Vol 20 (1) ◽  
Author(s):  
Elisabeth Walsh-Wilkinson ◽  
Marie-Claude Drolet ◽  
Marie Arsenault ◽  
Jacques Couet

2018 ◽  
Vol 18 (4) ◽  
pp. 128-131
Author(s):  
Nobutaka Masunaga ◽  
Yasuharu Matsunaga-Lee ◽  
Sen Matsumoto ◽  
Kouichi Tachibana ◽  
Yuzuru Takano

2017 ◽  
Vol 14 (1) ◽  
pp. 75-85 ◽  
Author(s):  
Jen-Te Hsu ◽  
Chang-Min Chung ◽  
Chi-Ming Chu ◽  
Yu-Shen Lin ◽  
Kuo-Li Pan ◽  
...  

2019 ◽  
Vol 40 (Supplement_1) ◽  
Author(s):  
E Radzik ◽  
K T Pigon ◽  
G B Banasik ◽  
A T Tomasik ◽  
W J Jachec ◽  
...  

Abstract Introduction Permanent right ventricle pacing leads to left ventricle remodeling, its systolic dysfunction and symptomatic heart failure in the long run. Valsartan is well known for its preventive anti-remodeling function in the post infarction heart remodeling. Objectives To assess the effect of valsartan on left ventricle remodeling in patients with second and third degree atrioventricular block with first-time implantation of dual chamber pacemaker. Methods This was a randomized, double-blind, placebo controlled single center study. One hundred eligible patients were assigned in a 1:1:1 fashion to receive placebo, valsartan 80mg or 160mg once daily, respectively. Echocardiographic assessment of left ventricle geometry, its systolic and diastolic function was performed at baseline and at twelve months. One patient from placebo group suffered stroke. We present the baseline date for 100 enrolled patients and follow-up data for 88 patients who have completed the study. Data in valsartan arms are pooled in one group. Concentration of soluble ST2 was measured in duplicates with Aspect Reader (Critical Diagnostics). Results Results are presented in table. Data are presented as mean and standard deviation. Table 1 Placebo (n=28) Valsartan (n=60) ANOVA Baseline 12 mths Baseline 12 mths sST2, ng/mL 36.0±16.0 39.4±15.3 35.1±15.2 19.9±6.5 0.01 LVEF, % 60±8 55±9 60±8 58±8 0.01 LVEDD, mm 48±5 50±4 48±6 49±5 NS LVESD, mm 29±4 32±5 29±5 31±4 NS LVEDV, mL 79±12 84±13 80±12 81±13 NS LVESV, mL 32±5 38±7 31±5 34±6 0.01 E/A 0.94±0.12 0.92±0.13 0.94±0.15 0.95±0.15 NS DecT, ms 211±38 226±43 223±45 218±37 0.01 IVRT, ms 98±14 108±17 101±17 99±18 0.01 Conclusion Valsartan has protective effect of left ventricle remodeling and function. It may be useful in prevention of pacing induced heart failure. Decrease in soluble ST2 concentration may help explain the alternative mechanism for protective role of valsartan. (NCT01805804)


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