scholarly journals Landscape depressions can create silent zones in noise polluted parks

2021 ◽  
Vol 263 (4) ◽  
pp. 2550-2554
Author(s):  
Timothy Van Renterghem ◽  
Pieter Thomas ◽  
Dick Botteldooren

Excessive road traffic noise exposure in (sub)urban parks hinders its restorative function and will negatively impact the number of visitors. Especially in such green environments, noise abatements by natural means, well integrated in the landscape, are the most desired solutions. Although dense vegetation bordering the park or raised berms could come first in mind, local landscape depressions are typically underused. In this work, a case-study of a small suburban park, squeezed in between two major arterial roads, is analyzed. The spatially dependent road traffic noise exposure in the park is assessed in detail by mobile sound pressure level measurements. Local reductions of up to 6-7 dBA are found at landscape depressions of only a few meters deep. It can therefore be concluded that this is an efficient measure and should be added to the environmental noise control toolbox for noise polluted parks.

Author(s):  
Saeha Shin ◽  
Li Bai ◽  
Tor H. Oiamo ◽  
Richard T. Burnett ◽  
Scott Weichenthal ◽  
...  

Background Exposure to road traffic noise has been linked to cardiometabolic complications, such as elevated blood pressure and glucose dysregulation. However, epidemiologic evidence linking road traffic noise to diabetes mellitus and hypertension remains scarce. We examined associations between road traffic noise and the incidence of diabetes mellitus and hypertension in Toronto, Canada. Methods and Results Using the Ontario Population Health and Environment Cohort, we conducted a retrospective, population‐based cohort study of long‐term residents of Toronto, aged 35 to 100 years, who were registered for provincial publicly funded health insurance, and were without a history of hypertension (n=701 174) or diabetes mellitus (n=914 607). Road traffic noise exposure levels were assessed by the equivalent continuous A‐weighted sound pressure level (dBA) for the 24‐hour day and the equivalent continuous A‐weighted sound pressure level for the night (11 pm –7 am) . Noise exposures were assigned to subjects according to their annual residential postal codes during the 15‐year follow‐up. We used random‐effect Cox proportional hazards models adjusting for personal and area‐level characteristics. From 2001 to 2015, each interquartile range increase in the equivalent continuous A‐weighted sound pressure level (dBA) for the 24‐hour day (10.0 dBA) was associated with an 8% increase in incident diabetes mellitus (95% CI, 1.07–1.09) and a 2% increase in hypertension (95% CI, 1.01–1.03). We obtained similar estimates with the equivalent continuous A‐weighted sound pressure level for the night (11 pm –7 am) . These results were robust to all sensitivity analyses conducted, including further adjusting for traffic‐related air pollutants (ultrafine particles and nitrogen dioxide). For both hypertension and diabetes mellitus, we observed stronger associations with the equivalent continuous A‐weighted sound pressure level (dBA) for the 24‐hour day among women and younger adults (aged <60 years). Conclusions Long‐term exposure to road traffic noise was associated with an increased incidence of diabetes mellitus and hypertension in Toronto.


2002 ◽  
Vol 33 (8) ◽  
pp. 16-24
Author(s):  
Jesús Alba Fernández ◽  
Marcelino Ferri García ◽  
Jaime Ramis Soriano ◽  
Juan Antonio Martínez Mora

In environmental acoustics the knowledge of the time dependency of the sound level provides relevant information about a sound event. In this sense, it may be said that conventional sound level metres have frequently implemented programs to calculate the fractiles (percentiles) of the distribution of instantaneous sound levels; and there are several indexes to evaluate the noise pollution, based on different statistical parameters. For further analysis of sound, and to obtain the commented indexes, it is accepted that this distribution is normal or gaussian. The questions we've tried to solve in this work are the following: First of all, whether the time dependent distribution of the variable sound pressure level should be considered as Gaussian in general cases or only in some particular ones. On the other hand, we have studied how the frequency of the sampling affects the resulting distribution of a given a sound event. To these ends, a set of road traffic noise events has been evaluated. Furthermore, even in gaussian distributions of sound pressure levels, the average of the distribution will not be coincident with the equivalent sound pressure level; that is the level of the average quadratic pressure. The difference between this parameter, and its dependence on the standard deviation, is studied.


2021 ◽  
Vol 263 (3) ◽  
pp. 3282-3293
Author(s):  
Jens Forssén ◽  
Andreas Gustafson ◽  
Meta Berghauser Pont ◽  
Marie Haeger-Eugensson ◽  
Christine Achberger ◽  
...  

For a model set of 31 different building morphologies in an urban setting, road traffic noise exposure has been calculated and analysed. For five of the building morphologies also vegetation surfaces on facades and roofs were studied. Facade exposures were analysed for both smaller (single-sided) flats and larger (floor-through) flats, considering the direct exposure from the roads as well as the non-direct exposure at noise-shielded positions like inner yards, applying a noise mapping software in combination with a prediction model for the non-direct exposure. Using noise indicators Lden and Lnight, the disease burden, in terms of DALY (Disability-Adjusted Life Years) per person, was estimated and analysed, via predictions of annoyance and sleep disturbance. The resulting effects of varying the building morphology and adding vegetation are shown and discussed, including effects of a bonus model for flats having additional facade elements with lower noise exposure.


2021 ◽  
pp. 2004594
Author(s):  
Shuo Liu ◽  
Youn-Hee Lim ◽  
Marie Pedersen ◽  
Jeanette T. Jørgensen ◽  
Heresh Amini ◽  
...  

BackgroundWhile air pollution has been linked to the development of chronic obstructive pulmonary disease (COPD), evidence on the role of environmental noise is just emerging. We examined the associations of long-term exposure to air pollution and road traffic noise with COPD incidence.MethodsWe defined COPD incidence for 24 538 female nurses from the Danish Nurse Cohort (age>44 years) as the first hospital contact between baseline (1993 or 1999) and 2015. We estimated residential annual mean concentrations of particulate matter with diameter<2.5 µm (PM2.5) since 1990 and nitrogen dioxide (NO2) since 1970 by the Danish DEHM/UBM/AirGIS modeling system, and road traffic noise (Lden) since 1970 by the Nord2000 model. Time-varying Cox regression models were applied to assess the associations of air pollution and road traffic noise with COPD incidence.Results977 nurses developed COPD during 18.6 years’ mean follow-up. We observed associations with COPD for all three exposures with hazard ratios (HRs) and 95% confidence intervals (CIs) of 1.19 (1.01, 1.41) per 6.26 µg·m−3 for PM2.5, 1.13 (1.05, 1.20) per 8.19 µg·m−3 for NO2, and 1.15 (1.06, 1.25) per 10 dB for Lden. Associations with NO2 and Lden attenuated slightly after mutual adjustment, but were robust to adjustment for PM2.5. Associations with PM2.5 were attenuated to null after adjustment for either NO2 or Lden. No potential interaction effect was observed between air pollutants and noise.ConclusionsLong-term exposure to air pollution, especially traffic-related NO2, and road traffic noise were independently associated with COPD.


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