scholarly journals Androgen receptor gene CAG repeat polymorphism and risk of isolated hypospadias: results from a meta-analysis

2015 ◽  
Vol 14 (1) ◽  
pp. 1580-1588 ◽  
Author(s):  
G. Huang ◽  
W. Shan ◽  
L. Zeng ◽  
L. Huang
Keyword(s):  
Androgen Receptor ◽  
Meta Analysis ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Repeat Polymorphism

Association between androgen receptor gene CAG repeat polymorphism and breast cancer risk: a meta-analysis

2010 ◽  
Vol 124 (3) ◽  
pp. 815-820 ◽  
Author(s):  
YouJin Hao ◽  
Rafael Montiel ◽  
BingHui Li ◽  
Enyi Huang ◽  
Lewie Zeng ◽  
...  
Keyword(s):  
Breast Cancer ◽  
Androgen Receptor ◽  
Breast Cancer Risk ◽  
Cancer Risk ◽  
Meta Analysis ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Repeat Polymorphism

The CAG repeat polymorphism of androgen receptor gene and prostate cancer: a meta-analysis

2011 ◽  
Vol 39 (3) ◽  
pp. 2615-2624 ◽  
Author(s):  
Mingliang Gu ◽  
Xiaoqun Dong ◽  
Xuezhi Zhang ◽  
Wenquan Niu
Keyword(s):  
Prostate Cancer ◽  
Androgen Receptor ◽  
Meta Analysis ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Repeat Polymorphism

Association Between Androgen Receptor Gene CAG Repeat Polymorphism and Plasma Testosterone Levels in Postmenopausal Women

2005 ◽  
Vol 12 (2) ◽  
pp. 135-141 ◽  
Author(s):  
Ilma Simoni Brum ◽  
Poli Mara Spritzer ◽  
Franyoise Paris ◽  
Maria Augusta Maturana ◽  
Franyoise Audran ◽  
...  
Keyword(s):  
Androgen Receptor ◽  
Postmenopausal Women ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Plasma Testosterone ◽  
Repeat Polymorphism ◽  
Testosterone Levels

Association of androgen receptor gene CAG and GGN repeat polymorphism with cryptorchidism: A meta-analysis

Andrologia ◽  
2017 ◽  
Vol 50 (3) ◽  
pp. e12909 ◽  
Author(s):  
Qi Wang ◽  
Xing Ge ◽  
Heng-Xue Wang ◽  
Qiao-Mei Shi ◽  
Zhen Ding ◽  
...  
Keyword(s):  
Androgen Receptor ◽  
Meta Analysis ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Repeat Polymorphism

scholarly journals Androgen Receptor Gene CAG Repeat Polymorphism in the Development of Ovarian Hyperandrogenism

2003 ◽  
Vol 88 (7) ◽  
pp. 3333-3338 ◽  
Author(s):  
Lourdes Ibáñez ◽  
Ken K. Ong ◽  
Nigel Mongan ◽  
Jarmo Jääskeläinen ◽  
Maria Victoria Marcos ◽  
...  
Keyword(s):  
Androgen Receptor ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Repeat Polymorphism ◽  
Ovarian Hyperandrogenism

Association of the CAG repeat polymorphisms in androgen receptor gene with polycystic ovary syndrome: A systemic review and meta-analysis

Gene ◽  
2013 ◽  
Vol 524 (2) ◽  
pp. 161-167 ◽  
Author(s):  
Tao Zhang ◽  
Wenqing Liang ◽  
Min Fang ◽  
Jianmin Yu ◽  
Ya Ni ◽  
...  
Keyword(s):  
Androgen Receptor ◽  
Polycystic Ovary Syndrome ◽  
Polycystic Ovary ◽  
Meta Analysis ◽  
Receptor Gene ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Systemic Review ◽  
Ovary Syndrome

CAG Repeat Polymorphism in the Androgen Receptor Gene in Women with Rheumatoid Arthritis

2011 ◽  
Vol 39 (1) ◽  
pp. 10-17 ◽  
Author(s):  
VIOLETTA DZIEDZIEJKO ◽  
MATEUSZ KURZAWSKI ◽  
KRZYSZTOF SAFRANOW ◽  
ANDRZEJ OSSOWSKI ◽  
JAROSLAW PIATEK ◽  
...  
Keyword(s):  
Rheumatoid Arthritis ◽  
Androgen Receptor ◽  
White Women ◽  
Receptor Gene ◽  
Study Groups ◽  
Androgen Receptor Gene ◽  
Cag Repeat ◽  
Cag Repeats ◽  
Repeat Polymorphism ◽  
Erosive Disease

Objective.Rheumatoid arthritis (RA) is the most common chronic, autoimmune, inflammatory disease, with a genetic and hormonal background. The prevalence of women among patients with RA suggests the important role of sex hormones in the pathogenesis of RA. We examined the association between CAG repeat polymorphism in the androgen receptor (AR) gene and susceptibility to RA and its clinical features in white women.Methods.The study groups consisted of 325 female patients with RA and 238 female controls. CAG repeat polymorphism was determined using polymerase chain reaction and subsequent fragment analysis by capillary electrophoresis.Results.The number of CAG repeats in patients did not differ from that of controls (22.1 ± 2.9 vs 21.9 ± 2.9, respectively; p = 0.26), but the presence of articular erosions was associated with a lower number of repeats in the shorter allele of patients with RA (20.4 ± 2.2 vs 21.2 ± 2.4; p = 0.031). When alleles with < 22 CAG were classified as short (S) and those with ≥ 22 CAG as long (L), the age at diagnosis of RA was lower in women with S-S genotype in comparison to combined S-L + L-L genotypes (43.0 ± 14.6 yrs vs 47.6 ± 12.5 yrs; p = 0.021). In patients with the L-L genotype, the frequency of erosive disease (OR 0.45, 95% CI 0.25–0.80, p = 0.0085) and extraarticular manifestations (OR 0.50, 95% CI 0.26–0.98, p = 0.047) was lower in comparison to carriers of the S allele. In multivariate analysis, the L-L genotype was an independent factor associated with a lower risk of erosions (OR 0.44, 95% CI 0.22–0.90, p = 0.024).Conclusion.The results suggest the association of short AR (CAG)n alleles with earlier onset and a more aggressive course of RA.

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