scholarly journals Unexpected cause of involuntary muscle movements: Reel syndrome

2021 ◽  
Vol 25 (7) ◽  
pp. 515-516
Author(s):  
Gorkem Kus ◽  
◽  
Goksel Cagirci ◽  
Cagin Mustafa Ureyen ◽  
Nermin Bayar ◽  
...  
Keyword(s):  
Involuntary Muscle

Opioid-Induced Muscle Activity: Implications for Managing Chronic Pain

1995 ◽  
Vol 29 (11) ◽  
pp. 1118-1121 ◽  
Author(s):  
Robert K Sylvester ◽  
Ralph Levitt ◽  
Preston D Steen
Keyword(s):  
Breakthrough Pain ◽  
Oral Morphine ◽  
Opioid Therapy ◽  
Chronic Opioid Therapy ◽  
Transdermal Fentanyl ◽  
Management Options ◽  
Morphine Infusion ◽  
Involuntary Muscle ◽  
Metastatic Lung ◽  
Morphine Solution

Objective: To increase awareness of opioid-induced involuntary muscle hyperactivity and to present management options. Case Summary: A ventilator-dependent 71-year-old man presented with pain caused by metastatic lung cancer. Transdermal fentanyl therapy was titrated to 200 μg/h. Two days later a continuous morphine infusion was initiated because of frequent administration of oral morphine solution for breakthrough pain. The patient became progressively less responsive and began exhibiting involuntary muscle hyperactivity thought to represent breakthrough pain. Despite the inability to assess pain control effectively in this unresponsive patient, the morphine infusion rate was increased from 22 to 717 mg/h within 7 days. No change in muscle hyperactivity was observed. Discussion: Over the last decade involuntary muscle hyperactivity has been documented as an adverse effect of chronic opioid therapy. The literature describing the incidence of this toxicity, possible risk factors for its development, and recommendations for its management are discussed. Conclusions: The occurrence of muscle hyperactivity in an unresponsive patient receiving chronic opioid therapy may represent opioid toxicity. Recommendations for managing opioid-induced muscle hyperactivity include reduction of the opioid dosage and/or administration of clonazepam therapy.

scholarly journals MECHANICS OF VOLUNTARY AND INVOLUNTARY MUSCLE ACTIONS

1989 ◽  
Vol 21 (Supplement) ◽  
pp. S68 ◽  
Author(s):  
B. Hather ◽  
M. Duvoisin ◽  
R. Harris ◽  
P. Buchanan ◽  
G. Dudley
Keyword(s):  
Involuntary Muscle ◽  
Muscle Actions

Basic and Translational Neuroscience of Childhood-Onset Dystonia: A Control-Theory Perspective

2018 ◽  
Vol 41 (1) ◽  
pp. 41-59 ◽  
Author(s):  
Terence D. Sanger
Keyword(s):  
Muscle Activation ◽  
Distributed Network ◽  
Childhood Onset ◽  
Specific Mechanism ◽  
Involuntary Muscle ◽  
Postural Stabilization ◽  
Reflex Gain ◽  
Cortical Regions ◽  
Mechanism Of Failure ◽  
The Relationship

Dystonia is a collection of symptoms with involuntary muscle activation causing hypertonia, hyperkinetic movements, and overflow. In children, dystonia can have numerous etiologies with varying neuroanatomic distribution. The semiology of dystonia can be explained by gain-of-function failure of a feedback controller that is responsible for stabilizing posture and movement. Because postural control is maintained by a widely distributed network, many different anatomic regions may be responsible for symptoms of dystonia, although all features of dystonia can be explained by uncontrolled activation or hypersensitivity of motor cortical regions that can cause increased reflex gain, inserted postures, or sensitivity to irrelevant sensory variables. Effective treatment of dystonia in children requires an understanding of the relationship between etiology, anatomy, and the specific mechanism of failure of postural stabilization.

An Introductory Snapshot

2016 ◽  
Author(s):  
Michael J. Aminoff
Keyword(s):  
Nervous System ◽  
Medical Students ◽  
Clinical Sign ◽  
Facial Palsy ◽  
Muscle Spasm ◽  
Involuntary Muscle ◽  
Clinician Educator ◽  
The Subject ◽  
Fundamental Law

The name of Charles Bell has been given to a nerve, a facial palsy, a clinical sign, an involuntary muscle spasm, a muscle, and a fundamental law of physiology, making it well known to physicians, surgeons, and medical students alike, even though they generally have little knowledge or appreciation of his actual, very solid accomplishments. He suggested, in fact, new ways to look at—and to make sense of—the nervous system. Bell was revered by some contemporaries for his achievements; to others, however, his name and brilliance were tarnished by charges of intellectual dishonesty and fraud. This chapter introduces Charles Bell, summarizing his achievements and failings so that the reader has a general understanding of the subject of this biography. He is considered as a scientist–surgeon, as a clinician–educator, and as a polymath.

The Clinical Effects of Cold Application on the Production of Electrically Induced Involuntary Muscle Contractions

2003 ◽  
Vol 12 (3) ◽  
pp. 240-248 ◽  
Author(s):  
Bonnie L. Van Lunen ◽  
Clayton Carroll ◽  
Kristen Gratias ◽  
Doug Straley
Keyword(s):  
Outcome Measures ◽  
Repeated Measures ◽  
Peak Torque ◽  
Pain Tolerance ◽  
Outpatient Rehabilitation ◽  
Clinical Effects ◽  
Output Intensity ◽  
Involuntary Muscle ◽  
Cold Application ◽  
Rehabilitation Clinic

Context:Rehabilitation.Objective:To determine the effects of a 20-min ice treatment on pain tolerance and peak torque.Design:A 2 × 2 × 6 factorial with repeated measures on 1 factor.Setting:Outpatient rehabilitation clinic.Participants:20 men and 15 women.Intervention:The participants were randomly assigned to an experimental (ice bag) or control (no ice bag) group.Main Outcome Measures:Peak electrical-stimulation output intensity (ESOI) was recorded in mV, and isokinetic peak torque (IPT), in N · m, every 4 min for 20 min.Results:ESOI and IPT increased over time. ESOI for the experimental condition was greater than for the control and within the experimental condition at 12, 16, and 20 min. No other differences were found for the IPT measures. There were no differences for ESOI and IPT between genders.Conclusions:Cryotherapy enables patients to tolerate greater output intensities but does not result in increased peak torque

MECHANICS OF VOLUNTARY AND INVOLUNTARY MUSCLE ACTIONS

1980 ◽  
Vol 21 (Supplement) ◽  
pp. S68
Author(s):  
B. Hather ◽  
M. Duvoisin ◽  
R. Harris ◽  
P. Buchanan ◽  
G. Dudley
Keyword(s):  
Involuntary Muscle ◽  
Muscle Actions

scholarly journals On the ultimate nerve-fibres distributed to muscle and some other tissues, with observations upon the structure and probable mode of action of a nervous mechanism

1865 ◽  
Vol 14 ◽  
pp. 229-268
Keyword(s):  
Mode Of Action ◽  
Nerve Fibres ◽  
Voluntary Muscle ◽  
Striped Muscle ◽  
Involuntary Muscle

Introduction. Of the movements occurring in the tissues of living beings, and of contractility.—The distribution of nerves to involuntary muscle. Distribution of nerves to the muscular fibres of the frog’s bladder. Distribution of nerves to the muscular fibres in the walls of arteries, veins, the intestine, ducts of glands, &c. —The distribution of nerves to striped muscle. Of the arrangement of the dark-bordered nerve-fibres distributed to voluntary muscle and other tissues.

scholarly journals Teaching Video NeuroImages: Involuntary muscle contractions in Hoffman syndrome

Neurology ◽  
2010 ◽  
Vol 75 (9) ◽  
pp. 836-836
Author(s):  
C. Loomis ◽  
S. J. Bird ◽  
J. M. Levine
Keyword(s):  
Muscle Contractions ◽  
Involuntary Muscle ◽  
Teaching Video
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