Role of susceptibility weighted imaging (SWI) in the diagnosis of abdominal wall endometriosis

2016 ◽  
pp. 325-9
Author(s):  
Canan Çimşit ◽  
Tevfik Yoldemir ◽  
İhsan Nuri Akpınar
Keyword(s):  
Abdominal Wall ◽  
Susceptibility Weighted Imaging ◽  
Abdominal Wall Endometriosis

scholarly journals The intricate role of mast cell proteases and the annexin A1-FPR1 system in abdominal wall endometriosis

2014 ◽  
Vol 46 (1) ◽  
pp. 33-43 ◽  
Author(s):  
Rubens Paula ◽  
Antonio H. Oliani ◽  
Denise C. M. Vaz-Oliani ◽  
Solange C. G. P. D’Ávila ◽  
Sonia M. Oliani ◽  
...  
Keyword(s):  
Mast Cell ◽  
Abdominal Wall ◽  
Annexin A1 ◽  
Abdominal Wall Endometriosis

Abdominal wall endometriosis: Is structure in imaging related to nodule localisation? A retrospective study

2021 ◽  
pp. 228402652110096
Author(s):  
Claire Figuier ◽  
Pierre-François Montoriol ◽  
Bruno Pereira ◽  
Pauline Chauvet ◽  
Nicolas Bourdel ◽  
...  
Keyword(s):  
Retrospective Study ◽  
Surgical Treatment ◽  
Disease Progression ◽  
Abdominal Wall ◽  
Significant Relationship ◽  
Subcutaneous Fat ◽  
Nodule Size ◽  
Abdominal Wall Endometriosis ◽  
The Relationship

Objective: Investigate the relationship between the structure of abdominal wall endometriotic nodules in MRI and their localisation in abdominal wall layers in order to better understand nodule origins. Design: Women who had an MRI prior to surgical treatment of an abdominal wall endometriotic nodule between 2005 and 2016. Population: Thirty-six patients including four patients with two nodules. Methods: MRI images were reviewed. Each nodule was analysed according to its structure (fibrous, cystic, mixed), localisation (subcutaneous fat, intra muscular, intermediary position), and size. Results: Forty nodules were analysed in MRI with no relationship found between localisation and nodule structure ( p = 0.48). 87.5% of mixed nodules were revealed to have a cystic superficial rim extending towards the subcutaneous fat layer. This finding suggests that the glandular part of the nodule is the active part of the disease from which nodule progression occurs. Intermediary and intramuscular nodules were respectively statistically larger than subcutaneous fat nodules indicating a relationship between nodule size and localisation (35 mm (22–53) vs 17 mm (17–23)) ( p = 0.03). Conclusion: Despite differences in environments surrounding the nodules, no significant relationship between nodule structure in imaging and abdominal wall localisation was found. Data from mixed nodules indicate however the possible role of nodule environment on structure and that the mechanism of nodule growth may be linked to development of cystic superficial rims, at the forefront of disease progression, abdominal wall nodules growing from deep to superficial. Studies are required to further investigate our findings and enable greater understanding of the origins of AWE.

scholarly journals Evaluation of Cases of Abdominal Wall Endometriosis at Universidade Estadual de Campinas in a period of 10 Years

2017 ◽  
Vol 39 (08) ◽  
pp. 403-407 ◽  
Author(s):  
Daniela Yela ◽  
Lucas Trigo ◽  
Cristina Benetti-Pinto
Keyword(s):  
Cesarean Section ◽  
Abdominal Wall ◽  
Recurrence Rate ◽  
Clinical Manifestations ◽  
Disease Recurrence ◽  
Surgical Margins ◽  
Abdominal Wall Endometriosis ◽  
Postoperative Control ◽  
Epidemiological Characteristics ◽  
Previous Cesarean

Purpose To determine the clinical and epidemiological characteristics of abdominal wall endometriosis (AWE), as well as the rate and recurrence factors for the disease. Methods A retrospective study of 52 women with AWE was performed at Universidade Estadual de Campinas from 2004 to 2014. Of the 231 surgeries performed for the diagnosis of endometriosis, 52 women were found to have abdominal wall endometriosis (AWE). The frequencies, means and standard deviations of the clinical characteristics of these women were calculated, as well as the recurrence rate of AWE. To determine the risk factors for disease recurrence, Fisher's exact test was used. Results The mean age of the patients was 30.71 ± 5.91 years. The main clinical manifestations were pain (98%) and sensation of a mass (36.5%). We observed that 94% of these women had undergone at least 1 cesarean section, and 73% had used medication for the postoperative control of endometriosis. The lesion was most commonly located in the cesarean section scar (65%). The recurrence rate of the disease was of 26.9%. All 14 women who had relapsed had surgical margins compromised in the previous surgery. There was no correlation between recurrent AWE and a previous cesarean section (p = 0.18), previous laparotomy (p = 0.11), previous laparoscopy (p = 0.12) and postoperative hormone therapy (p = 0.51). Conclusion Women with previous cesarean sections with local pain or lumps should be investigated for AWE. The recurrence of AWE is high, especially when the first surgery is not appropriate and leaves compromised surgical margins.

Abdominal wall endometriosis: a surgeon's perspective and review of 445 cases

2008 ◽  
Vol 196 (2) ◽  
pp. 207-212 ◽  
Author(s):  
John D. Horton ◽  
Kent J. DeZee ◽  
Eric P. Ahnfeldt ◽  
Michel Wagner
Keyword(s):  
Abdominal Wall ◽  
Abdominal Wall Endometriosis

Abdominal wall endometriosis

2021 ◽  
Vol Publish Ahead of Print ◽  
Author(s):  
Sarah E. Allen ◽  
Noah B. Rindos ◽  
Suketu Mansuria
Keyword(s):  
Abdominal Wall ◽  
Abdominal Wall Endometriosis

Expression of p63 Differs in Peritoneal Endometriosis, Endometriomas, Adenomyosis, Rectovaginal Septum Endometriosis, and Abdominal Wall Endometriosis

2007 ◽  
Vol 131 (7) ◽  
pp. 1099-1102
Author(s):  
Omero B. Poli Neto ◽  
Hebert M. Ferreira; ◽  
Leandra N. Z. Ramalho ◽  
Júlio C. Rosa e Silva ◽  
Francisco J. Candido dos Reis ◽  
...  
Keyword(s):  
Abdominal Wall ◽  
Task Force ◽  
Tertiary Care ◽  
Female Genital Tract ◽  
University Hospital ◽  
Rectovaginal Septum ◽  
Tissue Samples ◽  
Abdominal Wall Endometriosis ◽  
Peritoneal Endometriosis ◽  
Accompanying Symptoms

Abstract Context.—Although there is evidence that endometriosis results from basal endometrium dislocation, the underlying biology is not fully understood. One protein that plays an important role in regulating epithelial proliferation and differentiation is the 63-kDa membrane protein (p63), which is also a marker of basal and reserve cells in the female genital tract. Objective.—To determine whether p63 is expressed differently in peritoneal endometriosis, endometriomas, and adenomyosis, as well as in deep endometriotic nodules of the rectovaginal septum and abdominal wall. Design.—This study includes a prospective series of consecutive patients (Canadian Task Force classification II-2) from a tertiary care university hospital. Specimens collected from 83 patients (15 peritoneal endometriosis specimens, 22 endometrioma specimens, 36 adenomyosis specimens, and 10 rectovaginal septum/abdominal wall specimens) were evaluated. Diagnostic and operative laparoscopies or laparotomies were performed, and tissue samples were obtained. Immunohistochemistry was used to evaluate p63 expression. Results.—Positivity for p63 was detected in 93.3% of the peritoneal endometriosis specimens, 81.8% of the endometrioma specimens, 36.1% of the adenomyosis specimens, and none of the rectovaginal/abdominal wall endometriosis specimens (P < .001). Distribution of p63 immunostaining in the positive specimens was homogeneous. Conclusions.—Endometriotic lesions express p63 differently, and some retain the basal/reserve cell immunophenotype. Nevertheless, it remains unclear whether the lack of p63 expression in some lesions is related to the extent of the disease, to its clinical behavior, or to exacerbation of the accompanying symptoms.

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