Early Signs of Thiamine Deficiency: A Case Report

2020 ◽  
Vol 173 (1) ◽  
pp. 72-73 ◽  
Author(s):  
Jorge C. Kattah
Psihiatru ro ◽  
2019 ◽  
Vol 58 (3) (1) ◽  
pp. 18-20
Author(s):  
Cătălina Crişan ◽  
Laura Grosu ◽  
Oana Vanţa

Gayet-Wernicke encephalopathy is an acute neuropsychiatric condition caused by thiamine deficiency. Only a small percentage of patients experience all three symptoms, with ophtalmoplegia, ataxia and confusion, and the full triad occurs more frequently among those who have overused alcohol. The evolution is toward full recovery, Korsakoff syndrome, dementia or death. We present the case of a 56-year-old patient, known with a diagnostic of alcoholism, who was admitted for a complicated withdrawal syndrome with delirium and who developed encephalopathy and dementia syndrome.


Author(s):  
Sujoy Kundu

<p class="abstract"><span lang="EN-IN">We present a case of fracture intra capsular neck of femur in a young adult osteopenic patient, who had a nonunion and early signs of avascular necrosis even after the initial surgical intervention. Teriparatide was administered for two monthly doses after diagnosis of nonunion and avascular necrosis of femoral neck. 90% successful union was achieved in 2 months without further surgical intervention, and no adverse events related to the use of teriparatide were observed. Our case report shows that teriparatide can be an alternative to surgical intervention in nonunion of ICNF with early signs of avascular necrosis.</span></p>


Author(s):  
Catherine Crecraft ◽  
Jennifer Prittie

ABSTRACT Three dogs that presented to the emergency service in severely emaciated body conditions were admitted to the hospital for monitoring and refeeding. During their hospitalization, all three dogs developed electrolyte derangements or required supplementation to prevent hypophosphatemia and hypomagnesemia. Additionally, all dogs developed hyperlactatemia, which was suspected to be secondary to thiamine deficiency. Two dogs were reported to have cardiac abnormalities, including cardiac arrhythmias, systolic dysfunction, and spontaneous echogenic contrast. These cases highlight the complexity of refeeding syndrome and its associated complications that extend beyond electrolyte deficiencies.


2018 ◽  
Vol 23 (5) ◽  
pp. 384-386 ◽  
Author(s):  
Stamatis Karakonstantis ◽  
Despoina Galani ◽  
Dafni Korela ◽  
Sevasti Maragou ◽  
Despoina Arna ◽  
...  

2017 ◽  
Vol 5 (1) ◽  
Author(s):  
Genri Numata ◽  
Satoshi Kodera ◽  
Hiroyuki Kiriyama ◽  
Atsuko Nakayama ◽  
Eisuke Amiya ◽  
...  

1994 ◽  
Vol 9 (5) ◽  
pp. 337-340 ◽  
Author(s):  
Olivier Multon ◽  
Olivier Sibony ◽  
Lionel Carbillon ◽  
Jean Michel Guerin ◽  
Catherine Nessman ◽  
...  

2020 ◽  
Vol 2020 (6) ◽  
Author(s):  
L Hiffler ◽  
K Escajadillo ◽  
M Rocaspana ◽  
S Janet

Abstract In paediatrics, the overall clinical picture of thiamine deficiency (TD) is not easy to recognize, because it mimics or can be confused with other diseases even in cases of classic beriberi. Unsurprisingly, the likelihood of misdiagnosis of TD is even greater where beriberi has not been described. Critically ill patients have increased thiamine body consumption and dextrose-based IV fluid increases thiamine cellular demand even further. Consequently, severe acute conditions may result in TD, or trigger TD signs in patients with borderline thiamine status, with life-threatening consequences. Here, we describe the case of a young patient admitted to a West African hospital where TD is not well documented and diagnosed with severe pneumonia who responded dramatically to thiamine injection. The lack of rapid diagnostic capacity and the severe outcome of TD justify the use of a therapeutic thiamine challenge in cases with high clinical suspicion. Increased awareness about TD and low threshold for thiamine use should guide clinicians in their practice.


Sign in / Sign up

Export Citation Format

Share Document