scholarly journals Diminishing of Myocardial Damage Using Impella CP for ST-Elevation Myocardial Infarction Involving the Left Main Trunk

2021 ◽  
Vol 27 (1) ◽  
pp. 33-36
Author(s):  
Shunichi Doi ◽  
Yasuhiro Tanabe ◽  
Yuki Ishibashi ◽  
Yoshihiro J Akashi
Keyword(s):  
Myocardial Infarction ◽  
Myocardial Damage ◽  
St Elevation Myocardial Infarction ◽  
Left Main ◽  
Main Trunk ◽  
Left Main Trunk ◽  
St Elevation

Infarto agudo de miocardio con elevación del ST secundario a oclusión aguda del tronco principal de la coronaria izquierda: reporte de cinco casos.

2013 ◽  
Vol 38 (2) ◽  
pp. 83-85
Author(s):  
Franco Javier Vallejo García ◽  
Juan Manuel Senior Sanchez ◽  
Andres Fernandez Cadavid ◽  
Arturo Rodriguez Dimuro
Keyword(s):  
Myocardial Infarction ◽  
Acute Myocardial Infarction ◽  
Clinical Presentation ◽  
Primary Angioplasty ◽  
Left Main ◽  
Main Trunk ◽  
Left Main Trunk ◽  
Palabras Clave ◽  
St Elevation ◽  
Low Probability

Resumen La oclusión del TPI es poco observada durante la realización de angioplastia primaria en infarto agudo de miocardio, posiblemente en parte por la baja probabilidad de sobrevivir al evento el tiempo suficiente para llegar a un hospital (1). Reportamos cinco casos de pacientes tratados con angioplastia primaria con presentación y evolución clínica diferentes. Palabras clave: Infarto con elevación del ST, angioplastia primaria, oclusión aguda del tronco principal izquierdo. Abstract The left main trunk occlusion is rarely observed during primary angioplasty in acute myocardial infarction, possibly in part by the low probability of surviving the event long enough to reach a hospital. We report five cases of patients treated with primary angioplasty with different clinical presentation and course. Keywords: ST-elevation infarction, primary angioplasty, acute occlusion of the left main trunk.

Circulating Erythrocyte Microparticles and the Biochemical Extent of Myocardial Injury in ST Elevation Myocardial Infarction

Cardiology ◽  
2016 ◽  
Vol 136 (1) ◽  
pp. 15-20 ◽  
Author(s):  
Georgios Giannopoulos ◽  
Dimitrios A. Vrachatis ◽  
Georgios Oudatzis ◽  
Georgios Paterakis ◽  
Christos Angelidis ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Myocardial Injury ◽  
Troponin I ◽  
Multivariable Analysis ◽  
Area Under The Curve ◽  
Myocardial Damage ◽  
Primary Angioplasty ◽  
St Elevation Myocardial Infarction ◽  
Vascular Effects ◽  
St Elevation

Objectives: Red blood cell microparticles (RBCm) have potential adverse vascular effects and they have been shown to be elevated in ST elevation myocardial infarction (STEMI). The purpose of this study is to investigate their relationship with biochemical infarct size. Methods: RBCm were quantified with flow cytometry in blood drawn from 60 STEMI patients after a primary angioplasty. The creatine kinase-myocardial brain fraction (CK-MB) was measured at predefined time points and the area under the curve (AUC) was calculated. Results: RBCm count was correlated with CK-MB AUC (Spearman's ρ = 0.83, p < 0.001). The CK-MB AUC values per RBCm quartile (lower to upper) were: 3,351 (2,452-3,608), 5,005 (4,450-5,424), 5,903 (4,862-10,594), and 8,406 (6,848-12,782) ng × h/ml, respectively. From lower to upper quartiles, the maximal troponin I values were: 42.2 (23.3-49.3), 49.6 (28.8-54.1), 59.2 (41.4-77.3), and 69.1 (48.0-77.5) ng/ml (p = 0.005). In multivariable analysis, RBCm remained a significant predictor of CK-MB AUC (standardized β = 0.63, adjusted p = 0.001). Conclusions: Erythrocyte microparticles appear to be related to the total myocardial damage biomarker output. The exact pathophysiologic routes, if any, for this interaction remain to be identified. However, these results suggest that erythrocytes may be a - thus far virtually ignored - player in the pathogenesis of ischemic injury.

CLINICAL CHARACTERISTICS AND OUTCOMES OF LEFT MAIN ST-ELEVATION MYOCARDIAL INFARCTION

2018 ◽  
Vol 71 (11) ◽  
pp. A1282
Author(s):  
Ameera Ahmed ◽  
Ross Garberich ◽  
Frank Aguirre ◽  
Jenny Chambers ◽  
Yader Sandoval ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Clinical Characteristics ◽  
St Elevation Myocardial Infarction ◽  
Left Main ◽  
St Elevation

scholarly journals P669 Functional remodeling of the left atrium after first acute ST-elevation myocardial infarction: a 3D echocardiography study

2020 ◽  
Vol 21 (Supplement_1) ◽  
Author(s):  
F Jarjour ◽  
S Civera ◽  
A Vijiiac ◽  
B Elnagar ◽  
C Palermo ◽  
...  
Keyword(s):  
Myocardial Infarction ◽  
Left Atrium ◽  
Longitudinal Strain ◽  
Circumferential Strain ◽  
Troponin I ◽  
Myocardial Damage ◽  
St Elevation Myocardial Infarction ◽  
P Value ◽  
Functional Remodeling ◽  
St Elevation

Abstract Background Left atrium (LA) is a dynamic structure which is functionally coupled with the left ventricle and modulates its function in many cardiac conditions. The geometric and functional remodeling of the LA occurring early after myocardial infarction are poorly understood. Purpose We sought to evaluate the early changes in LA geometry and function occurring in survivors of a first acute ST-elevation myocardial infarction (STEMI), using three-dimensional echocardiography (3DE). Methods LA phasic volumes and strain (both longitudinal and circumferential) were measured using a dedicated automated software package in 54 patients at pre-discharge after STEMI, and in 54 age- and sex-matched healthy volunteers (controls), (figure 1). Results In STEMI patients, both maximal (LAV max) and minimal (LAV min) LA volumes were significantly larger than in controls 63 ± 15 vs. 53 ±11 ml; p = 0,002 and 38 ± 15 ml vs. 25 ± 6; p &lt;0.0001 (respectively). Moreover, when compared to controls (Table 1). Both longitudinal (LASr) and circumferential strain reservoirs showed a significant negative correlation with peak cardiac troponin I values (r=-0.344; p = 0.007 and r=-0.357; p = 0.005, respectively) as an estimate of the extent of myocardial damage. Conclusion STEMI was associated to significant geometrical and functional remodeling of the LA which was correlated with the extent of myocardial damage. Table 1 Controls STEMI patients P-value Longitudinal% LASr 21.8 ± 8.4 13.72 ± 8.27 &lt;0.0001 LAScd -12.8 ± 8.48 -6.43 ± 4.74 &lt;0.0001 LASct -9.73 ± 6.04 -7.26 ± 5.87 0.05 Circumferential % LASr-c 27.31 ± 8.07 18.92 ± 9.16 &lt;0.0001 LAScd-c -11.2 ± 5.93 -6.46 ± 5.68 0.0002 LASct-c -16.22 ± 6.33 -12.41 ± 5.94 0.004 LASr longitudinal strain reservoir, LAScd: longitudinal strain conduit, LAScd: longitudinal strain contraction, LASr-c: circumferential strain reservoir, LAScd-c: circumferential strain conduit , LASct-c:circumferential strain contraction Abstract P669 Figure 1

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