Cortical ablation induces time-dependent changes in rat pituitary somatotrophs and upregulates growth hormone receptor expression in the injured cortex

2014 ◽  
Vol 92 (10) ◽  
pp. 1338-1349 ◽  
Author(s):  
Irena Lavrnja ◽  
Vladimir Ajdzanovic ◽  
Svetlana Trifunovic ◽  
Danijela Savic ◽  
Verica Milosevic ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Hormone Receptor ◽  
Growth Hormone Receptor ◽  
Receptor Expression ◽  
Time Dependent ◽  
Cortical Ablation ◽  
Rat Pituitary ◽  
Hormone Receptor Expression

Growth Hormone Receptor Expression in the Rat Gastrointestinal Tract*

Endocrinology ◽  
1990 ◽  
Vol 126 (1) ◽  
pp. 299-306 ◽  
Author(s):  
PETER E. LOBIE ◽  
WINRICH BREIPOHL ◽  
MICHAEL J. WATERS
Keyword(s):  
Growth Hormone ◽  
Gastrointestinal Tract ◽  
Hormone Receptor ◽  
Growth Hormone Receptor ◽  
Receptor Expression ◽  
Hormone Receptor Expression

scholarly journals Growth hormone receptor expression in human gluteal versus abdominal subcutaneous adipose tissue: Association with body shape

Obesity ◽  
2016 ◽  
Vol 24 (5) ◽  
pp. 1090-1096 ◽  
Author(s):  
Kalypso Karastergiou ◽  
Miriam A. Bredella ◽  
Mi-Jeong Lee ◽  
Steven R. Smith ◽  
Susan K. Fried ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Adipose Tissue ◽  
Hormone Receptor ◽  
Subcutaneous Adipose Tissue ◽  
Body Shape ◽  
Growth Hormone Receptor ◽  
Receptor Expression ◽  
Hormone Receptor Expression ◽  
Subcutaneous Adipose

Cardiac Insulin-like Growth Factor I and Growth Hormone Receptor Expression in Renal Hypertension

Hypertension ◽  
1996 ◽  
Vol 27 (3) ◽  
pp. 636-642 ◽  
Author(s):  
Gregor Guron ◽  
Peter Friberg ◽  
Anna Wickman ◽  
Camilla Brantsing ◽  
Britt Gabrielsson ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Growth Factor ◽  
Hormone Receptor ◽  
Growth Hormone Receptor ◽  
Renal Hypertension ◽  
Receptor Expression ◽  
Insulin Like Growth Factor ◽  
Factor I ◽  
Growth Factor I ◽  
Hormone Receptor Expression

Growth hormone receptor expression in the dunning R 3327 prostatic carcinoma of the rat

The Prostate ◽  
1991 ◽  
Vol 19 (4) ◽  
pp. 273-278 ◽  
Author(s):  
F. Sinowatz ◽  
W. Breipohl ◽  
M. I. Waters ◽  
D. Lincoln ◽  
P. E. Lobie ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Hormone Receptor ◽  
Prostatic Carcinoma ◽  
Growth Hormone Receptor ◽  
Receptor Expression ◽  
Hormone Receptor Expression

scholarly journals Identification of a novel antisense RNA that regulates growth hormone receptor expression in chickens

RNA Biology ◽  
2019 ◽  
Vol 16 (5) ◽  
pp. 626-638
Author(s):  
Shudai Lin ◽  
Zihao Zhang ◽  
Tingting Xie ◽  
Bowen Hu ◽  
Zhuohao Ruan ◽  
...  
Keyword(s):  
Growth Hormone ◽  
Hormone Receptor ◽  
Antisense Rna ◽  
Growth Hormone Receptor ◽  
Receptor Expression ◽  
Hormone Receptor Expression

Developmental changes and differential regulation by testosterone and estradiol of growth hormone receptor expression in the rabbit

1996 ◽  
Vol 135 (5) ◽  
pp. 583-590 ◽  
Author(s):  
Yu M Yu ◽  
Horacio M Domené ◽  
Jorge Sztein ◽  
Debra R Counts ◽  
Fernando Cassorla
Keyword(s):  
Growth Hormone ◽  
Hormone Receptor ◽  
Growth Hormone Receptor ◽  
Treated Group ◽  
Differential Regulation ◽  
Receptor Expression ◽  
Developmental Changes ◽  
Mrna Levels ◽  
R Gene ◽  
Hormone Receptor Expression

Yu YM, Domené HM, Sztein J, Counts DR, Cassorla F. Developmental changes and differential regulation by testosterone and estradiol of growth hormone receptor expression in the rabbit. Eur J Endocrinol 1996;135:583–90. ISSN 0804–4643 To investigate the effects of testosterone and estradiol (E2) on growth hormone receptor (GH-R) gene expression, we measured GH-R mRNA levels in relation to the changes of sex steroid concentrations in the normal male rabbits aged 1–12 months and after administration of testosterone or E2 to castrated male rabbits. In the normal animals, E2 levels were below the detection limit in all age groups, and testosterone levels were below the detection limit at 1 month, increased at 2 months and reached the plateau of the adult levels after 4 months. Liver GH-R mRNA levels were low at 1 month, reached a peak at 2 months and then decreased slightly thereafter. In the castrated animals, liver and growth plate GH-R mRNA levels were increased in the testosterone-treated group (162.0 ± 12.0%, p < 0.025; 128.4 ± 7.6%; p < 0.025) and reduced in the E2-treated group (29.6 ± 6.2%, p < 0.005; 53.6 ± 11.3%, p < 0.025). Sex steroid administration did not result in any significant change in GH-R mRNA levels in striated muscle, kidney and heart. Serum GH concentrations were increased in E2 (15.3 ± 7.7 μg/l vs 4.8 ± 2.2 μg/l, p < 0.025) but the increase was not significant in testosterone-treated animals (8.4 ± 7.7 μg/l vs 4.8 ± 2.2 μg/l). Both testosterone and E2 treatment resulted in a reduction of mean serum growth hormone-binding protein (GHBP) levels compared to control animals (1077 ± 422 pmol/l, p < 0.01; 1137 ± 443 pmol/l, p < 0.01; 2308 ± 565 pmol/l). We conclude that in addition to their stimulatory effect on GH secretion, testosterone and E2 have opposite effects on GH-R gene expression in liver and growth plate in the rabbit. The modulation of GH-R expression by sex steroids may be important for growth during sexual maturation in mammals. Yu M Yu, MD, 11600 Bootjack Court, Gaithersburg, MD 20878, USA

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