Sympathetic control of spontaneous defibrillation of the heart

1992 ◽  
Vol 114 (4) ◽  
pp. 1387-1389
Author(s):  
V. I. Kobrin
Keyword(s):  
Sympathetic Control ◽  
Spontaneous Defibrillation

SYMPATHETIC CONTROL OF TISSUE CHANGE

The Lancet ◽  
1932 ◽  
Vol 220 (5697) ◽  
pp. 993-995
Author(s):  
R.Cunliffe Shaw
Keyword(s):  
Sympathetic Control ◽  
Tissue Change

scholarly journals The Effect of Nitric Oxide Inhibition in Spinal Cord Injured Humans with and without Preserved Sympathetic Control of the Vasculature

2016 ◽  
Vol 10 ◽  
Author(s):  
Rachael Brown ◽  
David Celermajer ◽  
Vaughan Macefield ◽  
Mikael Sander
Keyword(s):  
Nitric Oxide ◽  
Spinal Cord ◽  
Nitric Oxide Inhibition ◽  
Sympathetic Control ◽  
Spinal Cord Injured ◽  
Oxide Inhibition

Coordination between and sympathetic control of rabbit distal colonic contraction and potential difference

1992 ◽  
Vol 103 (4) ◽  
pp. 1392
Author(s):  
JM Goldhill ◽  
WH Percy
Keyword(s):  
Potential Difference ◽  
Sympathetic Control

Spinal stimulation to locate preganglionic neurons controlling the kidney, spleen, or intestine

1992 ◽  
Vol 263 (4) ◽  
pp. H1026-H1033 ◽  
Author(s):  
R. B. Taylor ◽  
L. C. Weaver
Keyword(s):  
Spinal Cord ◽  
Sympathetic Outflow ◽  
Renal Nerve ◽  
Homocysteic Acid ◽  
Sympathetic Control ◽  
The Third ◽  
Preganglionic Neurons ◽  
Spinal Segments ◽  
Vascular Beds ◽  
Nerve Discharge

The organization of sympathetic preganglionic neurons may be a substrate for selective control of sympathetic outflow to different vascular beds. This study was done to determine the spinal segments containing preganglionic neurons controlling discharge of renal, splenic, and mesenteric postganglionic nerves. In urethan-anesthetized rats, preganglionic neurons were stimulated by microinjecting D,L-homocysteic acid (3 nl, 0.17 M) into the lateral gray matter of the third thoracic (T3) to the fourth lumbar (L4) spinal segments. Responses from all three nerves could be elicited from segments T4-T13. The greatest increases in renal nerve discharge were evoked from segments T8-T12, the largest increase of 59 +/- 9% elicited from T10. Increases in splenic and mesenteric nerve discharge were smaller and were evoked more uniformly from T4-L3. The largest increases in discharge of splenic and mesenteric nerves were 19 +/- 5% (from T5) and 26 +/- 4% (from T10), respectively. The widely overlapping spinal cord segments controlling these three organs suggest that location of the preganglionic neurons in different spinal segments is not part of the mechanism for selective sympathetic control. However, the larger renal nerve responses demonstrate that sympathetic output to these organs can be differentiated at the level of the spinal cord.

Dissociation of enhanced efficiency of fat deposition during weight recovery from sympathetic control of thermogenesis

1995 ◽  
Vol 269 (2) ◽  
pp. R365-R369 ◽  
Author(s):  
A. G. Dulloo ◽  
J. Seydoux ◽  
L. Girardier
Keyword(s):  
Lower Energy ◽  
Fat Deposition ◽  
Energy Deficit ◽  
Energetic Efficiency ◽  
Sympathetic Control ◽  
Energy Conserving ◽  
And Control ◽  
Body Energy ◽  
Environmental Temperatures ◽  
Very High

Studies reported here examined the extent to which conditions known to suppress or markedly increase the sympathetic control of thermogenesis influence enhanced efficiency of fat deposition during weight recovery after caloric restriction. To this end, measurements of energy balance and changes in body energy compartments during refeeding of rats pair fed with weight-matched controls were conducted over a 2-wk period at 22 degrees C, at thermoneutrality (29 degrees C), or in the cold (6 degrees C). The results indicate that, despite identical (or slightly lower) energy intake relative to the respective controls, the refed animals showed greater gain in body fat (by 2- to 2.5-fold), 10-12% lower energy expenditure, and higher energetic efficiency (60-80%) than the controls at all three environmental temperatures. In contrast, protein gain was not different between the refed and control groups. Thus the energy-conserving mechanism specific to acceleration of fat deposition during weight recovery persists when sympathetically driven thermogenesis is shifted from very low to very high intensity. These findings raise the possibility that this energy-conserving mechanism during refeeding may be distinct from sympathetic-dependent mechanisms underlying adaptive reduction in thermogenesis during severe energy deficit and weight loss.

Studies on the Sympathetic Control of Normal and Diseased Synovial Blood Vessels: The Effect of α and β Receptor Stimulation and Inhibition, Monitored by the 133Xenon Clearance Technique

1971 ◽  
Vol 40 (2) ◽  
pp. 197-209 ◽  
Author(s):  
W. C. Dick ◽  
R. Jubb ◽  
W. W. Buchanan ◽  
J. Williamson ◽  
K. Whaley ◽  
...  
Keyword(s):  
Rheumatoid Arthritis ◽  
Clearance Rate ◽  
Qualitative Difference ◽  
Normal Subjects ◽  
Indirect Measure ◽  
Sympathetic Control ◽  
Blocking Agents ◽  
Disappearance Curve ◽  
Β Receptor ◽  
T Values

1. The half-life (T½) of the disappearance curve of 133Xe from the knee joint has been used as an indirect measure of synovial perfusion. 2. The effects of intra-articularly administered isoprenaline and noradrenaline and their respective blocking agents, propranolol and phentolamine, on the T½ values have been investigated in normal subjects and in patients with osteoarthritis and rheumatoid arthritis. 3. In normal subjects isoprenaline increased the clearance rate and this effect was blocked by propranolol. Noradrenaline decreased the clearance rate and this effect was blocked by phentolamine. 4. The injection of phentolamine in normal subjects caused an increase in synovial perfusion, suggesting the presence of a basal α-constrictor tone. 5. In the patients with osteoarthritis and rheumatoid arthritis quantitative differences were found in the responses to isoprenaline and noradrenaline and their respective blocking agents. 6. There appeared to be a qualitative difference in the case of phentolamine; it is possible that vasoconstrictor tone may be lost in inflamed joints.

Go with the flow: sympathetic control of blood flow during recovery from heart failure

2006 ◽  
Vol 101 (1) ◽  
pp. 3-4 ◽  
Author(s):  
Michael J. Joyner
Keyword(s):  
Heart Failure ◽  
Blood Flow ◽  
Sympathetic Control
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