Stimulation of the DNA replication and murine bone marrow cell proliferation with exogenous zinc-metallothionein

1996 ◽  
Vol 122 (5) ◽  
pp. 1088-1090
Author(s):  
A. N. Koterov ◽  
Z. A. Trebenok ◽  
N. B. Pushkareva ◽  
A. V. Nikol'skii
1987 ◽  
Vol 100 (1-2) ◽  
pp. 223-233 ◽  
Author(s):  
P.H. Hart ◽  
L.K. Spencer ◽  
P.A. Kenny ◽  
A.F. Lopez ◽  
P.J. McDonald ◽  
...  

2010 ◽  
Vol 24 (3) ◽  
pp. 1039-1044 ◽  
Author(s):  
K. Oshida ◽  
E. Iwanaga ◽  
K. Miyamoto ◽  
Y. Miyamoto

1986 ◽  
Vol 127 (3) ◽  
pp. 397-402 ◽  
Author(s):  
Elizabeth J. Waterhouse ◽  
Peter J. Quesenberry ◽  
Gary Balian

1990 ◽  
Vol 580 (1 Structure, Mo) ◽  
pp. 521-523
Author(s):  
BRIAN A. BLUE ◽  
LOUIS J. RULAND III ◽  
RANDALL D. LEARISH ◽  
PETER J. QUESENBERRY ◽  
GARY BALIAN

2020 ◽  
Vol 44 (3) ◽  
pp. 942-950
Author(s):  
Eduardo Madrigal-Bujaidar ◽  
Genaro Ivan Cerón-Montes ◽  
Joan Reyes-Miranda ◽  
Erasto Vergara-Hernández ◽  
Isela Álvarez-González ◽  
...  

Carbon dots derived from nopal significantly increase the number of micronuclei in mouse erythrocytes and inhibit mouse bone marrow cell proliferation.


Blood ◽  
1973 ◽  
Vol 41 (4) ◽  
pp. 539-547 ◽  
Author(s):  
Jack W. Hamer ◽  
Peter H. Fitzgerald

Abstract Cell proliferation studies, utilizing 3H-thymidine radioautography combined with quantitative DNA microdensitometry, demonstrated a gross disturbance of bone marrow cell proliferation in a 17-yr-old youth with primary shunt hyperbilirubinemia. Early erythroid and myeloid cells were arrested at the S, G2 and mitotic phases of the cell cycle, but late normoblasts were not affected. Intramedullary death of the abnormal early erythrmoid cells probably results and is compensated by increased hemopoiesis with a hyperplastic marrow, which 59Fe studies suggest leads to the production and early release of "stress" reticulocytes. The increased early bilirubin production that characterizes this disorder is considered to be derived primarily from intramedullary death of early normoblasts and secondarily from hemolysis in the marrow and spleen of stress reticulocytes.


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