Genetic polymorphisms associated with the prevalence of retinal vein occlusion in a Greek population

2019 ◽  
Vol 39 (11) ◽  
pp. 2637-2648 ◽  
Author(s):  
Aikaterini Christodoulou ◽  
Eleni Bagli ◽  
Maria Gazouli ◽  
Marilita M. Moschos ◽  
Georgios Kitsos
Keyword(s):  
Retinal Vein Occlusion ◽  
Genetic Polymorphisms ◽  
Greek Population ◽  
Vein Occlusion

Analysis of genetic polymorphisms related to thrombosis and other risk factors in patients with retinal vein occlusion

1998 ◽  
Vol 9 (7) ◽  
pp. 617-622 ◽  
Author(s):  
O. Salomon ◽  
J. Moisseiev ◽  
N. Rosenberg ◽  
O. Vidne ◽  
I. Yassur ◽  
...  
Keyword(s):  
Risk Factors ◽  
Retinal Vein Occlusion ◽  
Genetic Polymorphisms ◽  
Vein Occlusion

scholarly journals Genetic polymorphisms and retinal vein occlusion in an Italian population

2015 ◽  
Vol 14 (4) ◽  
pp. 13337-13341
Author(s):  
L. De Polo ◽  
P.E. Maltese ◽  
E. Rigoni ◽  
M. Bertelli ◽  
S. Cecchin ◽  
...  
Keyword(s):  
Retinal Vein Occlusion ◽  
Genetic Polymorphisms ◽  
Italian Population ◽  
Vein Occlusion

Genetic Polymorphisms Associated with Retinal Vein Occlusion: A Greek Case-control Study and Meta-analysis

2013 ◽  
Vol 34 (3) ◽  
pp. 130-139 ◽  
Author(s):  
Georgia G. Yioti ◽  
Orestis A. Panagiotou ◽  
Georgios A. Vartholomatos ◽  
Nikolaos I. Kolaitis ◽  
Chrisavgi N. Pappa ◽  
...  
Keyword(s):  
Retinal Vein Occlusion ◽  
Genetic Polymorphisms ◽  
Case Control Study ◽  
Meta Analysis ◽  
Case Control ◽  
Vein Occlusion ◽  
Control Study

Platelet Coagulant Activities and Retinal Vein Thrombosis

1977 ◽  
Vol 38 (02) ◽  
pp. 0399-0406 ◽  
Author(s):  
Peter N. Walsh ◽  
Richard E. Goldberg ◽  
Richard L. Tax ◽  
Larry E. Magargal
Keyword(s):  
Platelet Aggregation ◽  
Venous Thrombosis ◽  
Retinal Vein Occlusion ◽  
Serotonin Release ◽  
Platelet Factor ◽  
Trigger Mechanism ◽  
Local Conditions ◽  
Plasma Coagulation ◽  
Vein Thrombosis ◽  
Vein Occlusion

SummaryTo determine whether platelets play a role in the pathogenesis of retinal vein occlusion (RVO), platelets and coagulation were evaluated in 28 patients with RVO. Platelet coagulant activities concerned with the initiation and early stages of intrinsic coagulation were 2–4 fold increased in 9 patients with acute primary RVO but not in patients with acute secondary (10 patients) or chronic (9 patients) RVO. Platelet factor 3 activity, platelet aggregation, serotonin release by platelets and plasma coagulation were normal in all patients. Platelets may provide a trigger mechanism for venous thrombosis in the eye when local conditions permit.

Hypercoagulability and High Lipoprotein(a) Levels in Patients with Central Retinal Vein Occlusion

1994 ◽  
Vol 72 (01) ◽  
pp. 039-043 ◽  
Author(s):  
Francesco Bandello ◽  
Silvana Vigano’ D’Angelo ◽  
Mariella Parlavecchia ◽  
Alessandra Tavola ◽  
Patrizia Della Valle ◽  
...  
Keyword(s):  
Retinal Vein Occlusion ◽  
Central Retinal Vein Occlusion ◽  
Specific Treatment ◽  
Fibrin Deposition ◽  
Heparin Cofactor Ii ◽  
Lipoprotein A ◽  
Vein Occlusion ◽  
Acute Event ◽  
D Dimer ◽  
Central Retinal Vein

SummaryA series of coagulation parameters and lipoprotein(a) (Lp(a)) were explored in plasma from 40 patients with central retinal vein occlusion (CRVO, non-ischemic type n = 12; ischemic type n = 28) free of local and systemic predisposing factors, 1 to 12 months after the acute event. Forty age- and sex-matched patients with cataract served as controls. Prothrombin fragment 1.2 (FI.2), D-dimer, FVII:C - but not FVII: Ag - were higher and fibrinogen was lower in CRVO patients than in controls. Patients with non-ischemic CRVO had higher FI .2 and FVII:C and lower heparin cofactor II than patients with ischemic CRVO. Lp(a) levels greater than 300 mg/1 were observed in 12 patients with CRVO and in 4 controls (30% vs 10%, p <0.025). Patients with high Lp(a) - consistently associated with the S2 phenotype - had higher FVII:C, FVII:C/Ag ratio, and fibrinogen than the remaining CRVO patients. Plasma FI.2 and D-dimer correlated fairly in controls (r = 0.41) and patients with normal Lp(a) levels (r = 0.55), but they did not in the group of patients with high Lp(a) (r = 0.19), where the latter parameter was negatively related to D-dimer (r = −0.55). There was no dependence of the abnormalities observed on the time elapsed from vein occlusion. The findings of activated FVII and high FI.2, D-dimer, and Lp(a) are not uncommon in patients with CRVO. Increased thrombin formation with fibrin deposition and impaired fibrinolysis may play a role in the pathophysiology of CRVO and require specific treatment

Prevalence of Factor V Leiden in Young Adults with Retinal Vein Occlusion

1997 ◽  
Vol 77 (01) ◽  
pp. 214-216 ◽  
Author(s):  
Tuuli Linna ◽  
Antti Ylikorkala ◽  
Kimmo E Kontula ◽  
Päivi Puska ◽  
Timo Tervo
Keyword(s):  
Young Adults ◽  
Retinal Vein Occlusion ◽  
Factor V Leiden ◽  
Factor V ◽  
Vein Occlusion
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