Chemically-induced resistance on soybean inhibits nodulation and mycorrhization

2009 ◽  
Vol 329 (1-2) ◽  
pp. 259-268 ◽  
Author(s):  
Ludovic Faessel ◽  
Najat Nassr ◽  
Thierry Lebeau ◽  
Bernard Walter
2005 ◽  
Vol 37 (10) ◽  
pp. 1837-1842 ◽  
Author(s):  
Ilja Sonnemann ◽  
Nele Mika Streicher ◽  
Volkmar Wolters

2001 ◽  
Vol 47 (1) ◽  
pp. 49-54 ◽  
Author(s):  
A. D. Omer ◽  
J. Granett ◽  
R. Karban ◽  
E. M. Villa

1963 ◽  
Vol 56 (2) ◽  
pp. 189-192 ◽  
Author(s):  
J. W. Matteson ◽  
H. M. Taft ◽  
C. F. Rainwater

2002 ◽  
Vol 15 (1) ◽  
pp. 75-81 ◽  
Author(s):  
Chui Eng Wong ◽  
Rachael A. J. Carson ◽  
John P. Carr

Salicylic acid (SA) treatment triggers inhibition of replication or movement of several positive-sense RNA plant viruses in tobacco. This resistance can also be stimulated by nonlethal concentrations of cyanide and antimycin A (AA) without triggering induction of pathogenesis-related PR-1 protein genes. In two ecotypes of Arabidopsis thaliana (Columbia and Nössen), SA-induced resistance to a tobamovirus, Turnip vein clearing virus (TVCV), was also induced by nonlethal concentrations of cyanide and AA without concomitant induction of PR-1 gene expression. Furthermore, chemically induced resistance to TVCV, as well as the induction of the plant mitochondrial alternative oxidase (a potential target for the chemicals), was independent of NPR1, a gene that plays a key role downstream of SA in the induction of PR proteins. The chemically induced resistance to TVCV appeared to be due to inhibition of replication at the site of inoculation. Taken together, these results show that in Arabidopsis, as in tobacco, resistance to viruses can be induced via a distinct branch of the defensive signal transduction pathway. This suggests that the existence of this virus-specific branch may be widespread among plants.


Author(s):  
D.C. Hixson ◽  
J.C. Chan ◽  
J.M. Bowen ◽  
E.F. Walborg

Several years ago Karasaki (1) reported the production of type C virus particles by Novikoff ascites hepatocarcinoma cells. More recently, Weinstein (2) has reported the presence of type C virus particles in cell cultures derived from transplantable and primary hepatocellular carcinomas. To date, the biological function of these virus and their significance in chemically induced hepatocarcinogenesis are unknown. The present studies were initiated to determine a possible role for type C virus particles in chemically induced hepatocarcinogenesis. This communication describes results of studies on the biological and surface properties of type C virus associated with Novikoff hepatocarcinoma cells.Ecotropic and xenotropic murine leukemia virus (MuLV) activity in ascitic fluid of Novikoff tumor-bearing rats was assayed in murine sarcoma virus transformed S+L- mouse cells and S+L- mink cells, respectively. The presence of sarcoma virus activity was assayed in non-virus-producing normal rat kidney (NRK) cells. Ferritin conjugates of concanavalin A (Fer-Con wheat germ agglutinin (Fer-WGA), and Ricinus communis agglutinins I and II (Fer-RCAI and Fer-RCAII) were used to probe the structure and topography of saccharide determinants present on the viral envelope.


2001 ◽  
Vol 120 (5) ◽  
pp. A614-A614
Author(s):  
J HAIER ◽  
U GOLDMANN ◽  
U KEILHOLZ ◽  
H BUHR

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