scholarly journals Brain Ceruloplasmin Expression After Experimental Intracerebral Hemorrhage and Protection Against Iron-Induced Brain Injury

2018 ◽  
Vol 10 (1) ◽  
pp. 112-119 ◽  
Author(s):  
Hongwei Liu ◽  
Ya Hua ◽  
Richard F. Keep ◽  
Guohua Xi
Keyword(s):  
Brain Injury ◽  
Intracerebral Hemorrhage

The role of complement in brain injury following intracerebral hemorrhage: A review

2021 ◽  
pp. 113654
Author(s):  
Katherine Holste ◽  
Fan Xia ◽  
Hugh J.L. Garton ◽  
Shu Wan ◽  
Ya Hua ◽  
...  
Keyword(s):  
Brain Injury ◽  
Intracerebral Hemorrhage

Ginsenoside Rg2 Ameliorates Brain Injury After Intracerebral Hemorrhage in a Rat Model of Preeclampsia

2021 ◽  
Author(s):  
Liying Cai ◽  
Feifei Hu ◽  
Wenwen Fu ◽  
Xiaofeng Yu ◽  
Weijie Zhong ◽  
...  
Keyword(s):  
Brain Injury ◽  
Intracerebral Hemorrhage ◽  
Rat Model

scholarly journals Ischemic Brain Injury After Intracerebral Hemorrhage

Stroke ◽  
2012 ◽  
Vol 43 (8) ◽  
pp. 2258-2263 ◽  
Author(s):  
Shyam Prabhakaran ◽  
Andrew M. Naidech
Keyword(s):  
Brain Injury ◽  
Intracerebral Hemorrhage ◽  
Ischemic Brain Injury ◽  
Ischemic Brain

Metallothionein and brain injury after intracerebral hemorrhage

2009 ◽  
pp. 37-40 ◽  
Author(s):  
S. Yamashita ◽  
M. Okauchi ◽  
Y. Hua ◽  
W. Liu ◽  
R. F. Keep ◽  
...  
Keyword(s):  
Brain Injury ◽  
Intracerebral Hemorrhage

scholarly journals Mitochondria: Novel Mechanisms and Therapeutic Targets for Secondary Brain Injury After Intracerebral Hemorrhage

2021 ◽  
Vol 12 ◽  
Author(s):  
Weixiang Chen ◽  
Chao Guo ◽  
Hua Feng ◽  
Yujie Chen
Keyword(s):  
Brain Injury ◽  
Intracerebral Hemorrhage ◽  
Hemorrhagic Stroke ◽  
Secondary Brain Injury ◽  
Valuable Insight ◽  
Potential Therapeutic Target ◽  
Pathological Mechanism ◽  
Clinical Strategies ◽  
Inflammatory Activation ◽  
Insight Into

Intracerebral hemorrhage (ICH) is a destructive form of stroke that often results in death or disability. However, the survivors usually experience sequelae of neurological impairments and psychiatric disorders, which affect their daily functionality and working capacity. The recent MISTIE III and STICH II trials have confirmed that early surgical clearance of hematomas does not improve the prognosis of survivors of ICH, so it is vital to find the intervention target of secondary brain injury (SBI) after ICH. Mitochondrial dysfunction, which may be induced by oxidative stress, neuroinflammation, and autophagy, among others, is considered to be a novel pathological mechanism of ICH. Moreover, mitochondria play an important role in promoting neuronal survival and improving neurological function after a hemorrhagic stroke. This review summarizes the mitochondrial mechanism involved in cell death, reactive oxygen species (ROS) production, inflammatory activation, blood–brain barrier (BBB) disruption, and brain edema underlying ICH. We emphasize the potential of mitochondrial protection as a potential therapeutic target for SBI after stroke and provide valuable insight into clinical strategies.

T2 and T2* Magnetic Resonance Imaging Sequences Predict Brain Injury After Intracerebral Hemorrhage in Rats

2013 ◽  
pp. 151-155 ◽  
Author(s):  
Hang Jin ◽  
Gang Wu ◽  
Shukun Hu ◽  
Ya Hua ◽  
Richard F. Keep ◽  
...  
Keyword(s):  
Magnetic Resonance Imaging ◽  
Brain Injury ◽  
Magnetic Resonance ◽  
Intracerebral Hemorrhage ◽  
Resonance Imaging
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