First record in Upper Egypt of vascular wilt on pomegranate caused by Fusarium oxysporum, its molecular identification and artificial pathogenicity

2020 ◽  
Author(s):  
Ramadan A. Mohamed ◽  
Osama A. Al-Bedak ◽  
Sedky H. A. Hassan
Keyword(s):  
Fusarium Oxysporum ◽  
Molecular Identification ◽  
First Record ◽  
Vascular Wilt ◽  
Upper Egypt

First record of vascular wilt of flannel flower caused by Fusarium oxysporum

1998 ◽  
Vol 27 (1) ◽  
pp. 49 ◽  
Author(s):  
Suzanne Bullock ◽  
B.A. Summerell ◽  
L. von Richter
Keyword(s):  
Fusarium Oxysporum ◽  
First Record ◽  
Vascular Wilt

scholarly journals The First Record of Echinococcus ortleppi (G5) Tapeworms in Grey Wolf (Canis lupus)

Pathogens ◽  
2021 ◽  
Vol 10 (7) ◽  
pp. 853
Author(s):  
Jacek Karamon ◽  
Małgorzata Samorek-Pieróg ◽  
Jacek Sroka ◽  
Ewa Bilska-Zając ◽  
Joanna Dąbrowska ◽  
...  
Keyword(s):  
Small Intestine ◽  
Molecular Identification ◽  
Canis Lupus ◽  
Sequence Comparison ◽  
First Record ◽  
Grey Wolf ◽  
South Eastern ◽  
Counting Technique ◽  
Small Intestines ◽  
First Time

The aim of this study is to confirm the presence and molecular identification of Echinococcus tapeworms in wolves from south-eastern Poland. An investigation was carried out on the intestines of 13 wolves from south-eastern Poland. The small intestines were divided into three equal segments. Each segment was separately examined using the sedimentation and counting technique (SCT). The detected Echinococcus tapeworms were isolated and identified by PCRs and sequencing (nad1 and cox1 genes). Additionally, DNA isolated from the feces of wolves positive for Echinococcus tapeworms was examined with two diagnostic PCRs. The intestines of one wolf were positive for E. granulosus s.l. when assessed by SCT; the intestine was from a six-year-old male wolf killed in a communication accident. We detected 61 adult tapeworms: 42 in the anterior, 14 in the middle, and 5 in the posterior parts of the small intestine. The PCRs conducted for cox1 and nad1 produced specific products. A sequence comparison with the GenBank database showed similarity to the deposited E. ortleppi (G5) sequences. An analysis of the available phylogenetic sequences showed very little variation within the species of E. ortleppi (G5), and identity ranged from 99.10% to 100.00% in the case of cox1 and from 99.04 to 100.00% in the case of nad1. One of the two diagnostic PCRs used and performed on the feces of Echinococcus-positive animals showed product specific for E. granulosus. This study showed the presence of adult E. ortleppi tapeworms in wolves for the first time.

Molecular aspects of tomato (Solanum lycopersicum) vascular wilt by Fusarium oxysporum f. sp. lycopersici and antagonism by Trichoderma spp.

2021 ◽  
Vol 40 (1) ◽  
Author(s):  
Paula Andrea Castillo-Sanmiguel ◽  
Laura Rocío Cortés-Sánchez ◽  
Jovanna Acero-Godoy
Keyword(s):  
Fusarium Oxysporum ◽  
Solanum Lycopersicum ◽  
Negative Impact ◽  
Vascular Wilt ◽  
Trichoderma Spp ◽  
Genetic Characteristics ◽  
Synthetic Chemicals ◽  
Efficient Alternative ◽  
Molecular Aspects ◽  
The One

<p>Tomato plants (<em>Solanum lycopersicum</em>) are susceptible to the infection by diverse pathogens that cause devastating diseases such as vascular wilt, which causes great losses at the production level. The fungus <em>Fusarium oxysporum</em> f. sp. <em>lycopersici</em> (<em>Fol</em>) is one of the etiologic agents of this disease and its control lies in the use of synthetic chemicals which generate a negative impact in both health and the environment; thus, it is necessary to implement biological control as a healthier and more efficient alternative. The fungus <em>Trichoderma</em> spp. is a favorable option to be employed as a biocontroller against this pathogen thanks to its antagonist mechanisms, determined by metabolic and genetic characteristics. On the one hand, for <em>Fol</em> it is indispensable the activation of signaling routes such as MAPK Fmk1, MAPK Mpk1 y HOG, while <em>Trichoderma</em> spp. uses effectors involved in the interaction with the plant such as proteins, enzymes and secondary metabolites that also strengthen its immune response against infection, determined by both Pathogen Associated Molecular Patterns (PAMP) and effectors. Therefore, this article makes a review about the mentioned characteristics and suggests a greater application of tools and molecular markers for the management of this disease.</p>

Fusarium oxysporum f. sp. elaeidis. [Descriptions of Fungi and Bacteria].

1992 ◽  
Author(s):  
D. Brayford
Keyword(s):  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Contaminated Soil ◽  
Oil Palm ◽  
Ivory Coast ◽  
Plant Material ◽  
Elaeis Guineensis ◽  
Central Africa ◽  
Vascular Wilt ◽  
West And Central Africa

Abstract A description is provided for Fusarium oxysporum f. sp. elaeidis. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Elaeis guineensis (Oil palm). May also infect E. oleifera, E. madagascariensis and E. melanococca. DISEASE: Vascular wilt. GEOGRAPHICAL DISTRIBUTION: West and central Africa: Cameroon, Congo, Ivory Coast, Nigeria, Zaire. Possibly Colombia. TRANSMISSION: Contaminated soil or plant material. Potentially by means of seed (52, 4182).

Fusarium oxysporum f. sp. melonis. [Descriptions of Fungi and Bacteria].

1992 ◽  
Author(s):  
D. Brayford
Keyword(s):  
Saudi Arabia ◽  
Great Britain ◽  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Cucumis Melo ◽  
Vascular Wilt ◽  
Distribution Map

Abstract A description is provided for Fusarium oxysporum f. sp. melonis. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Cucumis melo (Muskmelon, Cantaloupe). DISEASE: Vascular wilt. GEOGRAPHICAL DISTRIBUTION: IMI Distribution Map 496. Africa: Morocco, Zimbabwe. Asia: India, Iraq, Iran, Israel, Japan, Korea, Lebanon, Philippines, Saudi Arabia, Thailand, USSR. Australasia: Australia. Europe: Belgium, France, Germany, Great Britain, Greece, Netherlands, Turkey. America: Canada, USA. TRANSMISSION: The fungus is soil borne and may be tramsmitted by seed.

Fusarium oxysporum f.sp. vasinfectum. [Descriptions of Fungi and Bacteria].

1964 ◽  
Author(s):  
C. Booth
Keyword(s):  
Puerto Rico ◽  
Fusarium Oxysporum ◽  
Central America ◽  
Geographical Distribution ◽  
West Indies ◽  
Central African Republic ◽  
Vascular Wilt ◽  
Vein Clearing ◽  
Cotton Plants ◽  
Cotton Belt

Abstract A description is provided for Fusarium oxysporum f.sp. vasinfectum. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: On Gossypium spp., and species of Cajanus, Coffea, Hevea, Hibiscus, Medicago, Ricinus, Solanum and Vigna. DISEASE: Vascular wilt or Fusariosis of cotton is a disease affecting its host at all stages of its growth. Early symptoms on seedlings consist of vein clearing of the leaves followed by necrosis of the interveinal tissue and death of the leaves. On older plants leaves become chlorotic and the vascular tissues show a brown discolouration. Growth is retarded and the plant eventually wilts. GEOGRAPHICAL DISTRIBUTION: Africa: Congo, Central African Republic, Egypt, Ethiopia, Madagascar, Senegal, Somalia, Sudan, South Africa, Tanganyika, Uganda; Asia: Burma, China, Formosa, India, Indo-China, Iran, Iraq, Pakistan, U.S.S.R. ; Europe: France, Greece, Italy, Romania, Yugoslavia; North America, Mexico, U.S.A. (cotton belt); Central America & West Indies: Guatemala, Nevis, Nicaragua, Puerto Rico, Salvador, St. Vincent; South America: Argentina, Brazil, Chile, Colombia, Peru, Venezuela. (C.M.I. Map 362). TRANSMISSION: Soil-borne, but may also be transmitted by water and seed. The pathogen has been recovered from delineated seed obtained from infected cotton plants in the Central African Republic, Congo, Tanganyika and Brazil (32: 186; 33: 143; 40: 754; 41: 389). The percentage infection ranged from 0.2 to 5.0.

Vascular Wilt of Common Naranjilla (Solanum quitoense) Caused by Fusarium oxysporum in Ecuador

2001 ◽  
Vol 2 (1) ◽  
pp. 15 ◽  
Author(s):  
J. B. Ochoa ◽  
B. Yangari ◽  
V. Galarza ◽  
J. Fiallos ◽  
M. A. Ellis
Keyword(s):  
Fusarium Oxysporum ◽  
Wilt Disease ◽  
Vascular Wilt ◽  
Major Constraint ◽  
Vascular Wilt Disease

Farmers have generally abandoned production of “common naranjilla” in many areas mainly due to uncontrollable epidemics of an apparent vascular wilt disease. Naranjilla vascular wilt (NVW) and is currently the major constraint to the production of naranjilla in Ecuador, where losses due to NVW may reach up to 80%. Accepted for publication 13 September 2001. Published 18 September 2001.

Fusarium oxysporum f. sp. ciceris. [Descriptions of Fungi and Bacteria].

1992 ◽  
Author(s):  
D. Brayford
Keyword(s):  
Fusarium Oxysporum ◽  
Cicer Arietinum ◽  
Geographical Distribution ◽  
Vascular Wilt

Abstract A description is provided for Fusarium oxysporum f. sp. ciceris. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Cicer arietinum (gram, chickpea). DISEASE: Vascular wilt. GEOGRAPHICAL DISTRIBUTION: First recorded from India; subsequently reported from Bangladesh, Burma, Chile, Ethiopia, Iran, Malawi, Mexico, Pakistan, Peru, Spain, Sudan, Syria, Tunisia and USA. TRANSMISSION: The fungus is soil borne. It may also be transmitted by seed (66, 3143).

Fusarium oxysporum f. sp. dianthi. [Descriptions of Fungi and Bacteria].

1996 ◽  
Author(s):  
D. Brayford
Keyword(s):  
Fusarium Oxysporum ◽  
Geographical Distribution ◽  
Water Flow ◽  
Contaminated Soil ◽  
Vascular Wilt ◽  
Planting Materials ◽  
Local Dispersal

Abstract A description is provided for Fusarium oxysporum f. sp. dianthi. Information is included on the disease caused by the organism, its transmission, geographical distribution, and hosts. HOSTS: Dianthus spp. (carnation, pinks, sweet williams), Lychnis chalcedoica L. (Caryophyllaceae) (Armstrong & Armstrong, 1954; Hood & Stewart, 1957). DISEASE: Vascular wilt. GEOGRAPHICAL DISTRIBUTION: Widespread where Dianthus is grown. TRANSMISSION: Via infected planting materials and contaminated soil. Local dispersal is by water flow and splash droplets containing slimy macro- or microconidia.

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