The relationship of pulse pressure and diastolic pressure to systolic pressure in healthy subjects, 20–94 years of age

1965 ◽  
Vol 70 (2) ◽  
pp. 163-171 ◽  
Author(s):  
Arthur M. Master ◽  
Richard P. Lasser
Keyword(s):  
Pulse Pressure ◽  
Healthy Subjects ◽  
Diastolic Pressure ◽  
Systolic Pressure ◽  
Relationship Of ◽  
The Relationship

Diastolic pressure, systolic pressure, or pulse pressure?

2000 ◽  
Vol 2 (3) ◽  
pp. 271-279 ◽  
Author(s):  
Charalambos Vlachopoulos ◽  
Michael O’Rourke
Keyword(s):  
Pulse Pressure ◽  
Diastolic Pressure ◽  
Systolic Pressure

scholarly journals Effect of Inflammation on the Relationship of Pulse Pressure and Mortality in Haemodialysis

Nephron Extra ◽  
2011 ◽  
Vol 1 (1) ◽  
pp. 292-299 ◽  
Author(s):  
Debasish Banerjee ◽  
Allan J. Collins ◽  
Charles A. Herzog
Keyword(s):  
Pulse Pressure ◽  
Relationship Of ◽  
The Relationship

Augmentor action of the sympathetic cardiac nerves in man

1960 ◽  
Vol 15 (4) ◽  
pp. 629-631 ◽  
Author(s):  
Walter C. Randall ◽  
Howard McNally
Keyword(s):  
Electrical Stimulation ◽  
Surgical Procedures ◽  
Pulse Pressure ◽  
Diastolic Pressure ◽  
Animal Experiments ◽  
Systolic Pressure ◽  
Myocardial Contraction ◽  
Inotropic Action ◽  
Upper Thoracic ◽  
Cardiac Nerves

During surgical procedures involving the upper thoracic sympathetic trunk in man, systolic and diastolic pressures were recorded during electrical stimulation. Significant and frequently profound elevation in systolic pressure, with little change in diastolic pressure, resulted in a marked increase in pulse pressure which has been designated as an augmentor response. Cardiac acceleration was an accompanying but less impressive response. The upper four or five thoracic segments mediate both the chronotropic and the inotropic action of the sympathetics but the T1 to T3 levels are the most significant. Corollary observations in animal experiments indicate the response is related to increased force of myocardial contraction with consequent increased systolic ejection. Submitted on February 1, 1960

Characterizing ventricular mechanics and energetics following repeated coronary microembolization

1997 ◽  
Vol 272 (1) ◽  
pp. H186-H194 ◽  
Author(s):  
K. Todaka ◽  
D. Leibowitz ◽  
S. Homma ◽  
P. E. Fisher ◽  
C. Derosa ◽  
...  
Keyword(s):  
Diastolic Pressure ◽  
Systolic Pressure ◽  
Control Group ◽  
Myocardial Mechanics ◽  
Ventricular Mechanics ◽  
Volume Relation ◽  
The Right ◽  
Important Characterization ◽  
The Relationship

Myocardial mechanics and energetics were investigated in an animal model of moderate chronic heart failure (CHF) created by repeated coronary microembolizations in six dogs. The final fractional area change was 34 +/- 4%. Hearts of these animals were isolated and cross-perfused, and balloons were placed in the left ventricle (LV). Chamber contractile state was markedly depressed in embolized hearts as assessed by the slope (Ees 2.74 +/- 0.49 vs. 4.00 +/- 1.18 mmHg/ml, P < 0.01) and volume axis intercept (V: 8.7 +/- 5.9 vs. 1.0 +/- 3.2 ml, P < 0.01) of end-systolic pressure-volume relation compared with a group of six normal dogs. The end-diastolic pressure-volume relation of embolized hearts was shifted to the right, indicating a dilation of the LV. However, systolic and diastolic stress strain relationships were similar in the two groups, suggesting that the average myocardial properties of the embolized hearts are similar to those of normal hearts. The relationship between oxygen consumption and pressure-volume area in embolized hearts had smaller intercept (2.98 +/- 0.44 vs 3.92 +/- 0.39 x 10(-2) ml O2.beat-1.100 g LV-1, P < 0.01) compared with the control group, with no change in the slope. These results contrast with previous findings in pacing CHF and serve as an important characterization of ventricular properties in this model of CHF from different etiology.

Relationship between Stress Levels and Eating with Hypertension in the the Baji Dakka Treatment Room at Labuang Baji Hospital in Makassar

2020 ◽  
Vol 1 (1) ◽  
pp. 1-11
Author(s):  
Ratna Ratna ◽  
H. Ismail ◽  
Sri Wahyuni
Keyword(s):  
Diastolic Pressure ◽  
Univariate Analysis ◽  
Systolic Pressure ◽  
The Body ◽  
Bivariate Analysis ◽  
Eating Patterns ◽  
Cross Sectional ◽  
Treatment Room ◽  
Stress Levels ◽  
The Relationship

Hypertension is established at systolic pressure of 140 mmHg / more when at rest, diastolic pressure of 90 mmHg / more when at rest or on the move. This study uses a cross sectional research method, the purpose of this study was to determine the relationship of stress levels and eating patterns with the incidence of hypertension in hospitalized patients in the Baji Dakka treatment room at Labuang Baji Hospital in Makassar. The method of sampling uses accidental sampling method, where data collection is done by interviewing, filling out questionnaires. Many factors can trigger hypertension, one of which is stress and diet. When a person experiences stress, the body will produce the hormone adrenaline which can increase blood pressure, and uncontrolled eating patterns lead to an increase in blood volume, so that the heart's work in pumping blood also increases and thickening of artery walls by fat or cholesterol (atherosclerosis) which including foods containing fat are organ meats, chicken eggs, coconut milk foods, and all foods processed with cooking oil. The number of samples 37 respondents. The collected data is processed and analyzed using a computer statistics program. Data analysis included univariate analysis by looking for frequency distribution, and bivariate analysis by chi-square test of significance α <= 0.05 to determine the relationship between stress and eating patterns on the incidence of hypertension. The results of the bivariate analysis showed a stress relationship p = 0,000 <α = 0.05 and dietary patterns p = 0,000 <α = 0.05.

scholarly journals Cardiovascular dynamics in healthy subjects with differing heart rate responses to tilt

2008 ◽  
Vol 105 (5) ◽  
pp. 1448-1453 ◽  
Author(s):  
Farah A. Ramirez-Marrero ◽  
Nisha Charkoudian ◽  
Emma C. Hart ◽  
Darrell Schroeder ◽  
Liu Zhong ◽  
...  
Keyword(s):  
Heart Rate ◽  
Pulse Pressure ◽  
Healthy Subjects ◽  
Systolic Pressure ◽  
Plasma Norepinephrine ◽  
Sex Distribution ◽  
Cardiovascular Dynamics ◽  
Fundamental Part ◽  
Wide Range ◽  
Head Up Tilt

Orthostatic stress such as head-up tilt (HUT) elicits a wide range of heart rate (HR) and arterial pressure (AP) responses among healthy individuals. In this study, we evaluated cardiovascular dynamics in healthy subjects with different HR responses to HUT, but without autonomic dysfunction. We measured AP (brachial artery) and HR (ECG) during 5 min of 60° HUT in 76 healthy normotensive individuals. We then chose individuals on the basis of the extremes of HR responses to HUT (high = ΔHR ≥ 20 beats/min, and low = ΔHR ≤ 10 beats/min; n = 15 per group). Peak HR during HUT was 87 ± 10 beats/min in the high and 69 ± 14 beats/min in the low group ( P < 0.05). High HR responders had lower systolic pressure at baseline (121 ± 9 vs. 129 ± 11 mmHg, P < 0.05) and during HUT (120 ± 10 vs. 131 ± 13 mmHg, P < 0.05), and higher plasma norepinephrine (NE) response to HUT (ΔNE: 156.9 ± 17.8 vs. 89.0 ± 17.2 pg/ml; P < 0.05). ΔNE during HUT was also significantly correlated with ΔHR when all 76 subjects were included in a regression analysis ( r = 0.39; P < 0.001). Pulse pressure was lower during HUT in high HR responders compared with low HR responders (45 ± 1 vs. 55 ± 2 mmHg, P < 0.05). High HR responders also had larger fluctuations in systolic and pulse pressure during HUT (coefficient of variation = 10.7 ± 0.7 vs. 5.7 ± 0.3%; 7.9 ± 0.5 vs. 4.1 ± 0.4%, respectively, P < 0.05). Sex distribution was different between groups (high: 5 women, 10 men; low: 10 women, 5 men). Higher HR with lower AP during HUT is consistent with normal baroreflex mechanisms of integration. Although interindividual variability appears to be a fundamental part of cardiovascular regulation, the mechanisms of these differences and the sex discrepancy requires further investigation.

The Effect of Age on the Relationship of Pulse Pressure and Left Ventricular Mass in Untreated Patients with Mild to Moderate Hypertension

2002 ◽  
Vol 11 (1) ◽  
pp. 13-17 ◽  
Author(s):  
Pedro Armario ◽  
Raquel Hernández del Rey ◽  
Montserrat Martín-Baranera ◽  
Noemí Andreu-Valls ◽  
Luis Miguel Ceresuela ◽  
...  
Keyword(s):  
Left Ventricular Mass ◽  
Pulse Pressure ◽  
Moderate Hypertension ◽  
Left Ventricular ◽  
Ventricular Mass ◽  
Relationship Of ◽  
The Relationship ◽  
Effect Of Age

Relationship between circadian blood pressure variation and circadian protein excretion in CKD

2007 ◽  
Vol 293 (3) ◽  
pp. F655-F659 ◽  
Author(s):  
Rajiv Agarwal
Keyword(s):  
Blood Pressure ◽  
Mean Arterial Pressure ◽  
Pulse Pressure ◽  
Excretion Rate ◽  
Sleep State ◽  
Circadian Blood Pressure ◽  
Protein Excretion ◽  
Pressure Changes ◽  
Relationship Of ◽  
The Relationship

Circadian blood pressure changes are blunted in patients with chronic kidney disease (CKD). Proteinuria is the most important correlate of hypertension in CKD. However, little is known about the influence of circadian blood pressure changes and variation in protein excretion rate. Furthermore, the impact of blood pressure components, e.g., mean arterial pressure and pulse pressure, on proteinuria has not been evaluated. To analyze the relationship of circadian changes in blood pressure on urinary protein excretion patterns, glomerular filtration rate was measured with iothalamate clearance and 24-h ambulatory blood pressure with SpaceLabs 90207 monitor in 22 patients with CKD. It was found that hourly protein excretion rates were 31% higher during the night. Excretion results of sodium, potassium, chloride, urea, and creatinine were also between 30 and 40% higher at night. Systolic, mean arterial, and pulse pressures but not diastolic pressure were related to daytime protein excretion rate. At night, the relationship of systolic, diastolic, and mean arterial pressures was significantly lower and essentially flat with respect to protein excretion rate, but the relationship of pulse pressure and proteinuria was not different from that seen during the day. Circadian variation in blood pressure did not impact circadian sodium excretion rate. In conclusion, these data suggest that patients with CKD have patterns of proteinuria that share different relationships with blood pressure components depending on the awake-sleep state. Pulse pressure is related to proteinuria independent of the awake-sleep state. Reducing mean arterial pressure during the day and pulse pressure during the day or night may be effective antiproteinuric strategies.

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