Persistent organic pollutants in human breast milk from Asian countries

2007 ◽  
Vol 146 (2) ◽  
pp. 400-413 ◽  
Author(s):  
Shinsuke Tanabe ◽  
Tatsuya Kunisue
Keyword(s):  
Breast Milk ◽  
Organic Pollutants ◽  
Persistent Organic Pollutants ◽  
Human Breast Milk ◽  
Asian Countries ◽  
Human Breast

Isolation of Lipophilic Persistent Organic Pollutants From Human Breast Milk

2012 ◽  
Vol 45 (11) ◽  
pp. 1412-1425 ◽  
Author(s):  
Christian Bjerregaard-Olesen ◽  
Philip Sebastian Hjelmborg ◽  
Eva Cecilie Bonefeld-Jørgensen
Keyword(s):  
Breast Milk ◽  
Organic Pollutants ◽  
Persistent Organic Pollutants ◽  
Human Breast Milk ◽  
Human Breast

Persistent Organic Pollutants in Human Breast Milk and Associations with Maternal Thyroid Hormone Homeostasis

2019 ◽  
Vol 54 (2) ◽  
pp. 1111-1119 ◽  
Author(s):  
Zhong-Min Li ◽  
Michael Albrecht ◽  
Hermann Fromme ◽  
Karl-Werner Schramm ◽  
Meri De Angelis
Keyword(s):  
Thyroid Hormone ◽  
Breast Milk ◽  
Organic Pollutants ◽  
Persistent Organic Pollutants ◽  
Human Breast Milk ◽  
Human Breast ◽  
Hormone Homeostasis ◽  
Maternal Thyroid

Occurrence of benzotriazole ultraviolet stabilizers (BUVSs) in human breast milk from three Asian countries

2019 ◽  
Vol 655 ◽  
pp. 1081-1088 ◽  
Author(s):  
Joon-Woo Kim ◽  
Kwang-Hyeon Chang ◽  
Maricar Prudente ◽  
Pham Hung Viet ◽  
Shin Takahashi ◽  
...  
Keyword(s):  
Breast Milk ◽  
Human Breast Milk ◽  
Asian Countries ◽  
Human Breast

Perfluorinated Compounds in Human Breast Milk from Several Asian Countries, and in Infant Formula and Dairy Milk from the United States

2008 ◽  
Vol 42 (22) ◽  
pp. 8597-8602 ◽  
Author(s):  
Lin Tao ◽  
Jing Ma ◽  
Tatsuya Kunisue ◽  
E. Laurence Libelo ◽  
Shinsuke Tanabe ◽  
...  
Keyword(s):  
United States ◽  
Breast Milk ◽  
Infant Formula ◽  
The United States ◽  
Perfluorinated Compounds ◽  
Human Breast Milk ◽  
Asian Countries ◽  
Human Breast ◽  
Dairy Milk

Organophosphorus flame retardants (PFRs) in human breast milk from several Asian countries

Chemosphere ◽  
2014 ◽  
Vol 116 ◽  
pp. 91-97 ◽  
Author(s):  
Joon-Woo Kim ◽  
Tomohiko Isobe ◽  
Mamoru Muto ◽  
Nguyen Minh Tue ◽  
Kana Katsura ◽  
...  
Keyword(s):  
Breast Milk ◽  
Flame Retardants ◽  
Human Breast Milk ◽  
Asian Countries ◽  
Human Breast ◽  
Organophosphorus Flame Retardants

Organohalogen compounds in human breast milk from Republic of Buryatia, Russia

2007 ◽  
Vol 146 (1) ◽  
pp. 225-232 ◽  
Author(s):  
Oyuna V. Tsydenova ◽  
Agus Sudaryanto ◽  
Natsuko Kajiwara ◽  
Tatsuya Kunisue ◽  
Valeriy B. Batoev ◽  
...  
Keyword(s):  
Breast Milk ◽  
Human Breast Milk ◽  
Human Breast ◽  
Organohalogen Compounds

Human breast milk suppresses the transcriptional regulation of IL-1β-induced NF-κB signaling in human intestinal cells

2004 ◽  
Vol 287 (5) ◽  
pp. C1404-C1411 ◽  
Author(s):  
Ryoko Minekawa ◽  
Takashi Takeda ◽  
Masahiro Sakata ◽  
Masami Hayashi ◽  
Aki Isobe ◽  
...  
Keyword(s):  
Breast Milk ◽  
Pathogenic Bacteria ◽  
Ischemia Reperfusion Injury ◽  
Ischemia Reperfusion ◽  
Nuclear Factor Κb ◽  
Protective Role ◽  
Intestinal Cells ◽  
Human Breast Milk ◽  
Human Breast ◽  
Activation Pathway

Neonatal necrotizing enterocolitis (NEC), which is a disease with a poor prognosis, is considered to be caused by the coincidence of intestinal ischemia-reperfusion injury and systemic inflammation due to the colonization of pathogenic bacteria. Interleukin (IL)-8, a proinflammatory cytokine, plays an important role in the pathophysiology of NEC. It was recently reported that IL-1β activates the IL-8 gene by regulating the transcriptional nuclear factor κB (NF-κB) signaling pathways in intestinal cells. The protective role of maternal milk in NEC pathogenesis has been reported in both human and animal studies. In this study, we show that human breast milk dramatically suppressed the IL-1β-induced activation of the IL-8 gene promoter by inhibiting the activation pathway of NF-κB. Moreover, we also show that human breast milk induced the production of IκBα. These results suggest that human breast milk could be protective and therapeutic in neonates with NEC by inhibiting the activation pathway of NF-κB.

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