Assessment of a prototype equipment for cuffless measurement of systolic and diastolic arterial blood pressure

2011 ◽  
Vol 44 (2) ◽  
pp. e57
Author(s):  
Giandomenico Nollo ◽  
Michela Masè ◽  
Walter Mattei ◽  
Roberta Cucino ◽  
Luca Faes
1992 ◽  
Vol 82 (2) ◽  
pp. 157-162 ◽  
Author(s):  
Margaret Ramsay ◽  
Fiona Broughton Pipkin ◽  
Peter Rubin

1. Twenty-eight healthy non-pregnant women and 28 women in the first or second trimester of pregnancy were studied. They were given an incremental intravenous infusion of either noradrenaline or angiotensin II. Pressor and heart rate responses were documented. 2. Dose-pressor response curves were constructed for the two agents in pregnant and non-pregnant women (n=14 in each group). The regression parameters of slope and intercept were calculated, and were used to derive the variables of dose required to elicit a 10 mmHg rise in systolic or diastolic blood pressure. 3. The pressor response to angiotensin II was diminished in pregnancy, with approximately twice the dose being required to raise the systolic or diastolic arterial blood pressure as in non-pregnant subjects. 4. The systolic pressor response to noradrenaline was slightly diminished in pregnancy, but the diastolic pressor response was unchanged. There were no significant differences between the doses of noradrenaline required to elicit a 10 mmHg rise in systolic or diastolic arterial blood pressure in pregnant or non-pregnant subjects. 5. There was a diminution in the bradycardia evoked in response to both hormones in pregnancy. 6. We conclude that the well-documented pressor insensitivity to angiotensin II during pregnancy is a specific phenomenon, not a manifestation of a generalized reduction in vascular reactivity.


Author(s):  
Ina Mežiņa-Mamajeva ◽  
Arvīds Grigāns ◽  
Pēteris Kučāns ◽  
Juris Kriķis ◽  
Jānis Raibarts ◽  
...  

Abstract Many studies have shown that systolic and diastolic arterial blood pressure changes due tohigher weight in both school-age children and in adolescents. Abnormalities of thyroid functionmight be no less important factor in relation to metabolic syndrome. The objective of the studywas to determine the relationship of arterial blood pressure, glucose, adiponectin and thyroidfunction parameters to major risk factors of metabolic syndrome. The risk factors tested werebody mass index (BMI) and waist circumference in female students aged 18 to 25 years. Westudied 105 RSU Red Cross college students aged 18 to 25 years. We calculated their BMI, lungvital capacity (ml), thyroid changes by ultrasound (USG) (27 students) and ECG (79 students). Inaddition, we measured their waist and chest circumferences, and systolic and diastolic bloodpressure in both arms. We determined concentration of glucose, adiponectin, thyroid stimulatinghormone (TSH) and free thyroxine (FT4) in blood. Statistical analyses were performed using theSPSS 15.01 package software. There was a statistically significant positive correlation betweenthe arterial blood pressure, BMI and increase of the waist circumference in students (average age20.7 years). The average arterial blood pressure in students was normal. The age at the onset ofmenarche had a significant positive effect on diastolic arterial blood pressure (P = 0.009-0,017).An increase in blood glucose concentration was closely associated with BMI (P = 0.03) and waistcircumference (P = 0.045). However, adiponectin concentration was correlated with systolic (P =0.007-0.048) and diastolic (P = 0.002-0.003) blood pressure. Significant ECG changes werefound in 10% of the subjects, indicating cardiovascular changes in these young women. Thehigher the FT4 and TSH concentrations, the more frequently were observed changes in the ECGST-segment (P = 0.01-0,008). A significant relationship between diastolic blood pressure and theage at onset of menarche (P = 0.009-0.017) was found. Increased arterial blood pressure wasassociated with an increase in BMI, waist circumference and adiponectin concentration, while anincrease in blood glucose concentration was associated with increased BMI and waist circumference,but not with the adiponectin level. Changes in ECG and increased diastolic arterial bloodpressure in relation to menarche indicate a possible role of the endocrine system and genetic factorsin regulation of the main parameters of metabolic syndrome


2008 ◽  
Vol 64 (5) ◽  
pp. 1188-1195 ◽  
Author(s):  
Samir Benchekroune ◽  
Peter C. J. Karpati ◽  
Christine Berton ◽  
Cédric Nathan ◽  
Joaquim Mateo ◽  
...  

2011 ◽  
Vol 44 (2) ◽  
pp. 201-207 ◽  
Author(s):  
Michela Masè ◽  
Walter Mattei ◽  
Roberta Cucino ◽  
Luca Faes ◽  
Giandomenico Nollo

2005 ◽  
Vol 133 (11-12) ◽  
pp. 505-509 ◽  
Author(s):  
Boris Jojic ◽  
Ljubica Leposavic

Introduction. Autogenic training is a widely recognised psychotherapy technique. The British School of Autogenic Training cites a large list of disorders, states, and changes, where autogenic training may prove to be of help. We wanted to explore the application of autogenic training as a therapy for adjustment disorder in adults. Our sample consisted of a homogeneous group of 35 individuals, with an average age of 39.3?1.6 years, who were diagnosed with adjustment disorder, F 43.2, in accordance with ICD 10 search criteria. Aim. The aim of our study was to research the effectiveness of autogenic training as a therapy for adjustment disorder in adults, by checking the influence of autogenic training on the biophysical and biochemical indicators of adjustment disorder. Method. We measured the indicators of adjustment disorder and their changes in three phases: before the beginning, immediately after the beginning, and six months after the completion, of a practical course in autogenic training. We measured systolic and diastolic arterial blood pressure, brachial pulse rate as well as the levels of cortisol in plasma, of cholesterol in blood, and of glucose. During that period, autogenic training functioned as the sole therapy. Results. The study confirmed our preliminary assumptions. The measurements we performed demonstrated that arterial blood pressure, pulse rate, concentration of cholesterol and cortisol, after the application of autogenic training among the subjects suffering from adjustment disorder, were lower in comparison to the initial values. These values remained lower even six months after the completion of the practical course in autogenic training. Conclusion. Autogenic training significantly decreases the values of physiological indicators of adjustment disorder, diminishes the effects of stress in an individual, and helps adults to cope with stress, facilitating their recuperation.


1956 ◽  
Vol 103 (4) ◽  
pp. 477-486 ◽  
Author(s):  
Sheldon E. Greisman

Further evidence has been obtained that L-norepinephrine infusions markedly decreases cutaneous capillary blood flow in the nailfold. Sustained ischemia of the capillary bed occurred prior to the attainment of hypertensive diastolic arterial blood pressure levels. Corroborative evidence of this nailfold capillary ischemia was obtained from the capillary reactive hyperemia that followed the abrupt discontinuance of the L-norepinephrine infusions. The infusion of angiotonin into persons with normal blood pressure did not induce sustained ischemia of the nailfold capillary vessels, even after definite hypertensive diastolic arterial blood pressure levels were attained. Corroboration of the adequacy of nailfold capillary blood flow was obtained by noting the absence of capillary reactive hyperemia following the abrupt cessation of the angiotonin infusions. The nailfold capillary bed in persons with angiotonin-induced hypertension could not be distinguished from that of persons with essential hypertension by direct microscopic examination. During the infusion of angiotonin into subjects with normal cardiovascular systems, the reactivity of the nailfold capillary bed to circulating L-norepinephrine was significantly increased, approaching the levels found in persons with essential hypertension. From the standpoint of its effects upon the nailfold capillary bed, angiotonin, unlike L-norepinephrine, is one substance which possesses the properties required of the hypothetical humoral pressor substance of essential hypertension.


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