Cardioversion of atrial fibrillation in a dog with structural heart disease using an esophageal-right atrial lead configuration

2014 ◽  
Vol 16 (4) ◽  
pp. 277-281 ◽  
Author(s):  
Robert A. Sanders ◽  
Alan G. Ralph ◽  
N. Bari Olivier
2011 ◽  
Vol 2011 ◽  
pp. 1-4 ◽  
Author(s):  
H. O. Savage ◽  
N. Ding ◽  
O. Eso ◽  
B. Sachdev ◽  
D. L. Lefroy

The formation of Intracardiac thrombi is rare in the absence of structural heart disease or atrial fibrillation. We describe a case of spontaneous right atrial thrombus formation that occurred in a patient with a hypercoagulable condition who had been sub optimally anticoagulated.


2020 ◽  
Vol 41 (Supplement_2) ◽  
Author(s):  
M Martinez-Selles ◽  
R Elosua ◽  
M Ibarrola ◽  
M De Andres ◽  
P Diez-Villanueva ◽  
...  

Abstract Background Advanced interatrial block (IAB), prolonged and bimodal P waves in surface ECG inferior leads, is an unrecognized surrogate of atrial dysfunction and a trigger of atrial dysrhythmias, mainly atrial fibrillation (AF). Our aim was to prospectively assess whether advanced IAB in sinus rhythm precedes AF and stroke in elderly outpatients with structural heart disease, a group not previously studied. Methods Prospective observational registry that included outpatients aged ≥70 years with structural heart disease and no previous diagnosis of AF. Patients were divided into three groups according to P-wave characteristics. Results Among 556 individuals, 223 had normal P-wave (40.1%), 196 partial IAB (35.3%), and 137 advanced IAB (24.6%). After a median follow-up of 694 days; 93 patients (16.7%) developed AF, 30 stroke (5.4%), and 34 died (6.1%). Advanced IAB was independently associated with AF (hazard ratio [HR] 2.9, 95% confidence interval [CI] 1.7–5.1, p<0.001), stroke (HR 3.8, 95% CI 1.4–10.7, p=0.010), and AF/stroke (HR 2.6, 95% CI 1.5–4.4, p=0.001). P-wave duration (ms) was independently associated with AF (HR 1.05, 95% CI 1.03–1.07, p<0.001), AF/stroke (HR 1.04, 95% CI 1.02–1.06, p<0.001), and mortality (HR 1.04, 95% CI 1.00–1.08, p=0.021). Conclusions The presence of advanced IAB in sinus rhythm is a risk factor for AF and stroke in an elderly population with structural heart disease and no previous diagnosis of AF. P-wave duration was also associated with all-cause mortality. Figure. Age- and sex-adjusted linear and non-linear association between P-wave duration (msec) and atrial fibrillation (A), stroke (B), and atrial fibrillation or stroke (C) risk. Results of a generalized additive model with spline smoothing functions and 4 degrees of freedom. Figure 1. Kaplan-Meyer curves of survival free of atrial fibrillation (A), stroke (B) and atrial fibrillation or stroke (C) in patients with normal P-wave, partial interatrial block (IAB) and advanced IAB. Funding Acknowledgement Type of funding source: None


2010 ◽  
Vol 3 (6) ◽  
pp. 606-615 ◽  
Author(s):  
Maurits A. Allessie ◽  
Natasja M.S. de Groot ◽  
Richard P.M. Houben ◽  
Ulrich Schotten ◽  
Eric Boersma ◽  
...  

2012 ◽  
Vol 44 (3) ◽  
pp. 211-219 ◽  
Author(s):  
Nicola Cooley ◽  
Mark J. Cowley ◽  
Ruby C. Y. Lin ◽  
Silvana Marasco ◽  
Chiew Wong ◽  
...  

Chronic atrial fibrillation (AF) is a complication associated with the dilated atria of patients with valvular heart disease and contributes to worsened pathology. We examined microRNA (miRNA) expression profiles in right and left atrial appendage tissue from valvular heart disease (VHD) patients. Right atrial (RA) appendage from patients undergoing coronary artery bypass grafting and left atrial (LA) appendage from healthy hearts, not used for transplant, were used as controls. There was no detectable effect of chronic AF on miRNA expression in LA tissue, but miRNA expression in RA was strongly influenced by AF, with 47 miRNAs (15 higher, 32 lower) showing differential expression between the AF and control sinus rhythm groups. VHD induced different changes in miRNA expression in LA compared with RA. Fifty-three (12 higher, 41 lower) miRNAs were altered by VHD in LA, compared with 5 (4 higher, 1 lower) in RA tissue. miRNA profiles also differed between VHD-LA and VHD-RA (13 higher, 26 lower). We conclude that VHD and AF influence miRNA expression patterns in LA and RA, but these are affected differently by disease progression and by the development of AF. These findings provide new insights into the progression of VHD.


ESC CardioMed ◽  
2018 ◽  
pp. 2208-2211
Author(s):  
Bhupesh Pathik ◽  
Jonathan M. Kalman

Atrial flutter refers to an electrocardiographic (ECG) appearance of continuously undulating flutter waves without an isoelectric baseline. It represents a heterogeneous group of atrial arrhythmias characterized by a macroreentrant mechanism. However, focal atrial tachycardia, especially if rapid and in the context of underlying structural heart disease or prior atrial surgery, may also cause a similar ECG appearance. A definition based on the underlying macroreentrant mechanism is therefore preferred particularly in the current era of three-dimensional electroanatomical mapping which allows detailed anatomical delineation of the circuit location. The clinical presentations of atrial macroreentry are variable and are influenced by ventricular response rate, presence of underlying structural heart disease, prior atrial surgery, or medications. The purpose of this chapter is to describe the different clinical presentations of this arrhythmia as well as its classification according to underlying mechanism. In addition, the clinical presentation of atrial macroreentry in special clinical situations is discussed. These include (1) the relationship between atrial fibrillation and cavotricuspid isthmus-dependent atrial macroreentry, (2) the organization of atrial fibrillation into atrial macroreentry with flecainide treatment, and (3) the association between atrial macroreentry and tachycardia-induced cardiomyopathy.


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