Selection for NaCl Tolerance in Cell Culture of Three Canary Island Tomato Land Races. I. Recovery of Tolerant Plantlets from NaCl-Tolerant Cell Strains

1988 ◽  
Vol 133 (1) ◽  
pp. 1-6 ◽  
Author(s):  
G. Garcia-Reina ◽  
V. Moreno ◽  
A. Luque
Nature ◽  
1964 ◽  
Vol 201 (4923) ◽  
pp. 1050-1051 ◽  
Author(s):  
ROBEBT M. EIBEN ◽  
STANLEY M. GARTLER

1989 ◽  
Vol 89 (4) ◽  
pp. 1299-1305
Author(s):  
Jochen Berlin ◽  
Robert B. van Huystee ◽  
Ludger Witte ◽  
Jürgen Bender ◽  
Hans J. Jäger ◽  
...  

1987 ◽  
Vol 131 (3-4) ◽  
pp. 225-236 ◽  
Author(s):  
Jochen Berlin ◽  
Christiane Mollenschott ◽  
Florenz Sasse ◽  
Ludger Witte ◽  
Gerd-Walter Piehl ◽  
...  

2014 ◽  
Vol 50 (6) ◽  
pp. 766-776 ◽  
Author(s):  
Ashok A. Nikam ◽  
Rachayya M. Devarumath ◽  
Mahadeo G. Shitole ◽  
Vikram S. Ghole ◽  
Prahlad N. Tawar ◽  
...  

1959 ◽  
Vol 110 (4) ◽  
pp. 629-642 ◽  
Author(s):  
William H. Murphy ◽  
Raymond Armstrong

By application of a variant of poliovirus, Type 2, MEF1 strain, as a selective agent it was possible to distinguish among stable parent strains of epithelial cells and their clonal derivatives by their differential morphologic response to infection. The variant of poliovirus grew in a number of cell strains without induction of observable cytopathogenic changes. Other strains of cells reacted to viral infection by manifesting partial or complete degeneration. Parent HeLa cells and virus underwent simultaneous serial propagation in the absence of homotypic antiserum to virus. The stability of the virus-cell relationship was established by results from replicate experiments conducted over a period of years. Some cell strains of common origin maintained in different laboratories did not react similarly to the cytopathogenic effect of virus. Representative experiments revealed that the morphologic response of HeLa cells to MEF virus infection was not influenced by the presence or absence of pleuropneumonia-like organisms. The differential morphologic response of cells to infection was confirmed by efficiency-of-plating experiments which revealed differences in the capacity of MEF virus to form plaques in the test cell strains. Serial passages of MEF virus in cell strains demonstrated differences in their selection for cytopathogenic "mutants" of virus.


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